Robust consensus control of uncertain multi-agent systems with input delay: a model reduction method

This paper addresses the robust consensus control design for input-delayed multi-agent systems subject to parametric uncertainties. To deal with the input delay, the Artstein model reduction method is employed by a state transformation. The input-dependent integral term that remains in the transformed system, due to the model uncertainties, is judiciously analysed. By carefully exploring certain features of the Laplacian matrix, sufficient conditions for the global consensus under directed communication topology are identified using Lyapunov-Krasovskii functionals in the time domain. The proposed control only relies on relative state information of the subsystems via network connections. The effectiveness and robustness of the proposed control design is demonstrated through a numerical simulation example

Consensus disturbance rejection for Lipschitz nonlinear multi-agent systems with input delay: a DOBC approach

In this paper, a new predictor-based consensus disturbance rejection method is proposed for high-order multi agent systems with Lipschitz nonlinearity and input delay. First, a distributed disturbance observer for consensus control is developed for each agent to estimate the disturbance under the delay constraint. Based on the conventional predictor feedback approach, a non-ideal predictor based control scheme is constructed for each agent by utilizing the estimate of the disturbance and the prediction of the relative state information. Then, rigorous analysis is carried out to ensure that the extra terms associated with disturbances and nonlinear functions are properly considered. Sufficient conditions for the consensus of the multi-agent systems with disturbance rejection are derived based on the analysis in the framework of Lyapunov-Krasovskii functionals. A simulation example is included to demonstrate the performance of the proposed control scheme. (C) 2016 The Franklin Institute. Published by Elsevier Ltd. All rights reserved.National Natural Science Foundation of China [61673034]SCI(E)ARTICLE1,SI298-31535

An analytic model of typhoon wind field and simulation of storm tides

Storm tides have intensified due to global climate warming, with limited attention given to storm current velocity (SCV) due to data scarcity during hurricanes/typhoons and limitations in existing wind models’ accuracy. We propose an analytic model incorporating sea-surface resistance into the gradient wind equation, offering a theoretically robust approach. Through rigorous verification against measured data, our model demonstrates significant accuracy improvement compared to established models. Simulating storm tides during Typhoon Rammasun using our approach reveals strong agreement between calculated SCVs and measured data, surpassing the performance of the Holland model. Notably, typhoon storm surges primarily respond to pressure, while SCVs are predominantly governed by wind speed in open sea. The highest water level aligns with the lowest pressure, with maximum SCVs trailing the maximum wind radius. SCVs significantly exceed astronomical tidal current velocities (ACVs) in the open sea, reaching a maximum of 3.57 m/s. Areas where the SCV-to-ACV ratio exceeds 3 constitute 21.4% of the study area. Combining our wind model with Typhoon SCV simulations provides valuable insights into storm tide dynamics, advancing our understanding of storm tide mechanisms and informing mitigation strategies

Preparation of ZnO hybrid nanoparticles by ATRP

Zinc oxide (ZnO) is a wide bandgap semiconductor material that has attracted widespread interest as particle filler in polymer nanocomposite materials. However, its applications have been hindered by the limited dispersibility and surface-modification techniques. Herein, three distinct approaches for the synthesis of polymer-tethered ZnO hybrid materials are compared in terms of uniformity and yield of the particle-brush product: “grafting-from”, “grafting-onto”, and “grafted-copolymer template” methods. In the “grafting-from” method, pristine ZnO nanoparticles (NP) were first functionalized with atom transfer radical polymerization (ATRP) initiators followed by grafting-from process to form poly(methyl methacrylate) (PMMA) or poly(styrene-co-acrylonitrile) (PSAN) tethered polymer chains. In the “grafting-onto” method, PMMA-b-PAA (poly[acrylic acid]) and PSAN-b-PAA diblock copolymers were prepared and attached onto the surface of ZnO NPs using sonication bath. For the “grafted-copolymer template” method, PSAN-b-PtBA-Br (poly[tert-butyl acrylate]-Br) macroinitiators were crosslinked with divinylbenzene (DVB) to form PSAN-b-PtBA-PDVB core-shell star polymers. After hydrolysis to form PSAN-b-PAA-PDVB star polymers, the functional stars were used as polymer templates for the synthesis of ZnO NPs within the PAA-core of the stars. Core-shell molecular bottlebrushes with PAA-b-PS block-copolymer side chains were also used as anisotropic analogues of star template to prepared worm-like ZnO particles. Several ZnO precursors, zinc nitrite, zinc 2-ethylhexanoate, and zinc acetate were evaluated as precursors of ZnO. Conditions were identified that enable the synthesis of polymer-tethered ZnO with excellent size uniformity and dispersion characteristics using the star-template method

Sp1 Is Essential for p16(INK4a) Expression in Human Diploid Fibroblasts during Senescence

BACKGROUND: p16 (INK4a) tumor suppressor protein has been widely proposed to mediate entrance of the cells into the senescent stage. Promoter of p16 (INK4a) gene contains at least five putative GC boxes, named GC-I to V, respectively. Our previous data showed that a potential Sp1 binding site, within the promoter region from −466 to −451, acts as a positive transcription regulatory element. These results led us to examine how Sp1 and/or Sp3 act on these GC boxes during aging in cultured human diploid fibroblasts. METHODOLOGY/PRINCIPAL FINDINGS: Mutagenesis studies revealed that GC-I, II and IV, especially GC-II, are essential for p16 (INK4a) gene expression in senescent cells. Electrophoretic mobility shift assays (EMSA) and ChIP assays demonstrated that both Sp1 and Sp3 bind to these elements and the binding activity is enhanced in senescent cells. Ectopic overexpression of Sp1, but not Sp3, induced the transcription of p16 (INK4a). Both Sp1 RNAi and Mithramycin, a DNA intercalating agent that interferes with Sp1 and Sp3 binding activities, reduced p16 (INK4a) gene expression. In addition, the enhanced binding of Sp1 to p16 (INK4a) promoter during cellular senescence appeared to be the result of increased Sp1 binding affinity, not an alteration in Sp1 protein level. CONCLUSIONS/SIGNIFICANCE: All these results suggest that GC- II is the key site for Sp1 binding and increase of Sp1 binding activity rather than protein levels contributes to the induction of p16 (INK4a) expression during cell aging