2,433 research outputs found
Effects of benthic algae on release of soluble reactive phosphorus from sediments: a radioisotope tracing study
AbstractTo evaluate the effect of benthic algae on soluble reactive phosphorus (SRP) release from sediments in shallow lakes, experiments on SRP release with and without benthic algae in sediment cores and an experiment on SRP uptake by benthic algae were conducted using the radioisotope (32P) tracing method. The dissolved oxygen (DO) concentration in sediment cores was also investigated. The results show that benthic algae effectively reduce the release of SRP from sediments to overlying water. The uptake of SRP by benthic algae, which is the direct way in which benthic algae affect the SRP release from sediments, is low in filtered water and increases with the SRP concentration. However, in the experiment, the increased uptake rate lasted for a short time (in one hour), and after that it returned to a low rate. Benthic algae make the DO concentration and the oxic layer thickness increased, which can indirectly reduce the SRP release from sediments. These findings indicate that benthic algae can reduce the SRP release from sediments in both direct and indirect ways. It seems that the indirect way also plays an important role in reducing the SRP release from sediments
Tetrakis(ethyl pyridine-4-carboxylate-κN)bis(thiocyanato-κN)cobalt(II)
In the title complex, [Co(NCS)2(C8H9NO2)4], the CoII atom is six-coordinated by four N atoms from four ethyl pyridine-4-carboxylate ligands in the equatorial plane and two N atoms of thiocyanate ligands in the axial positions, showing a slightly distorted octahedral geometry. The structure exhibits disorder in one of the ethyl chains, which was refined using a two-site model with 0.70 (6):0.30 (6) occupancy
Top quark decays with flavor violation in the B-LSSM
The decays of top quark are extremely rare processes in the
standard model (SM). The predictions on the corresponding branching ratios in
the SM are too small to be detected in the future, hence any measurable signal
for the processes at the LHC is a smoking gun for new physics. In the extension
of minimal supersymmetric standard model with an additional local
gauge symmetry (B-LSSM), new gauge interaction and new flavor changing
interaction affect the theoretical evaluations on corresponding branching
ratios of those processes. In this work, we analyze those processes in the
B-LSSM, under a minimal flavor violating assumption for the soft breaking
terms. Considering the constraints from updated experimental data, the
numerical results imply ,
, and in our
chosen parameter space. Simultaneously, new gauge coupling constants
in the B-LSSM can also affect the numerical results of
.Comment: 20 pages, 4 figures, published in EPJC. arXiv admin note: substantial
text overlap with arXiv:1803.0990
Tetraaquabis(2-methyl-1H-benzimidazolium-1,3-diacetato-κO)cobalt(II) tetrahydrate
In the title compound, [Co(C12H11N2O4)2(H2O)4]·4H2O, the CoII atom lies on an inversion center and is octahedrally coordinated by six O atoms from four water molecules and two monodentate zwitterionic 2-methylbenzimidazolium-1,3-diacetate ligands. An intramolecular O—H⋯O hydrogen bond occurs. In the crystal, intermolecular O—H⋯O hydrogen bonds link the molecules into a three-dimensional network. π–π interactions between the imidazole and benzene rings [centroid–centroid distance = 3.9031 (17) Å] consolidate the crystal packing
2,2′-[(4,6-Dinitro-1,3-phenylene)dioxy]diacetic acid hemihydrate
The skeletons of both independent molecules of the carboxylic acid hemihydrate, C10H8N2O10·0.5H2O, are approximately planar [maximum deviations 0.642 (3) and 0.468 (1) Å]. The deviations arise from the twisting of the nitro groups with respect to the aromatic rings [dihedral angles = 3.24 (2) and 27.01 (1), and 7.87 (1) and 16.37 (2)° in the two molecules]. The crystal structure features intermolecular O—H⋯O hydrogen bonds, which the link the dicarboxylic acid and water molecules into a supramolecular layer network
Poly[diethylenetriammonium [aquadi-μ2-sulfato-sulfatocerium(III)]]
A new organically templated open-framework cerium sulfate, {(C4H16N3)[Ce(SO4)3(H2O)]}n, was hydrothermally synthesized. The CeIII cation is nine-coordinated by nine O atoms, including one water molecule. Two of the SO4 groups afford one monodentate and bidentate linkages as the bridge to connect adjacent CeIII cations, while the third SO4 group attaches the CeIII cation in a bidentate mode. The crystal structure consists of layers composed of eight-membered-ring networks formed by four CeO9 polyhedra and four SO4 tetrahedra. The triply protonated diethylenetriamine cations are located between adjacent layers and connect the layers via hydrogen bonds
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PACAP neuropeptide promotes Hepatocellular Protection via CREB-KLF4 dependent autophagy in mouse liver Ischemia Reperfusion Injury.
Organ ischemia reperfusion injury (IRI), associated with acute hepatocyte death, remains an unresolved problem in clinical orthotopic liver transplantation (OLT). Autophagy, an intracellular self-digesting progress, is responsible for cell reprograming required to regain post-stress homeostasis. Methods: Here, we analyzed the cytoprotective mechanism of pituitary adenylate cyclase-activating polypeptide (PACAP)-promoted hepatocellular autophagy in a clinically relevant mouse model of extended hepatic cold storage (4 °C UW solution for 20 h) followed by syngeneic OLT. Results: In contrast to 41.7% of liver graft failure by day 7 post-transplant in control group, PACAP treatment significantly improved graft survival (91.7% by day 14), and promoted autophagy-associated regeneration programs in OLT. In parallel in vitro studies, PACAP-enhanced autophagy ameliorated cellular damage (LDH/ALT levels), and diminished necrosis in H2O2-stressed primary hepatocytes. Interestingly, PACAP not only induced nuclear cAMP response element-binding protein (CREB), but also triggered reprogramming factor Kruppel-like factor 4 (KLF4) expression in IR-stressed OLT. Indeed, CREB inhibition attenuated hepatic autophagy and recreated hepatocellular injury in otherwise PACAP-protected livers. Furthermore, CREB inhibition suppressed PACAP-induced KLF4 expression, whereas KLF4 blockade abolished PACAP-promoted autophagy and neutralized PACAP-mediated hepatoprotection both in vivo and in vitro. Conclusion: Current study documents the essential neural regulation of PACAP-promoted autophagy in hepatocellular homeostasis in OLT, which provides the emerging therapeutic principle to combat hepatic IRI in OLT
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