1,717 research outputs found

    Sharp Morrey regularity for an even order elliptic system

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    In this short note, we establish a sharp Morrey regularity theory for an even order elliptic system of Rivi\`ere type: \begin{equation*} \Delta^{m}u=\sum_{l=0}^{m-1}\Delta^{l}\left\langle V_{l},du\right\rangle +\sum_{l=0}^{m-2}\Delta^{l}\delta\left(w_{l}du\right)+f\qquad \text{in} B^{2m} \end{equation*} under minimal regularity assumptions on the coefficients functions V^l, w^l and that f belongs to certain Morrey space. This can be regarded as a further extension of the recent L^p-regularity theory obtained by Guo-Xiang-Zheng [15], and generalizes [7, 27] for second and fourth order elliptic systems.Comment: 12 page

    Testing the light scalar meson as a non-qqˉq\bar q state in semileptonic DD decays

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    To distinguish between the normal qqˉq\bar q and exotic diquark-antidiqark (q2qˉ2q^2\bar q^2) contents of the lowest-lying scalar meson (S0S_0), we investigate the semileptonic DS0e+νe,S0M1M2D\to S_0 e^+\nu_e, S_0\to M_1 M_2 decays, where M1(2)M_{1(2)} represents a pseudoscalar meson. With the form factors extracted from the current data, we calculate B(Ds+σ0e+νe,σ0π0π0)=(12.94.9+6.3)×104{\cal B}(D_s^+\to \sigma_0 e^+\nu_e,\sigma_0\to\pi^0\pi^0) =(12.9^{+6.3}_{-4.9})\times 10^{-4} and (0.80.7+1.2)×104(0.8^{+1.2}_{-0.7})\times 10^{-4} for the qqˉq\bar q and q2qˉ2q^2\bar q^2 quark structures, respectively, and compare them to the experimental upper limit: 6.4×1046.4\times 10^{-4}. It is clearly seen that S0S_0 prefers to be the q2qˉ2q^2\bar q^2 bound state. Particularly, Bqqˉ(Ds+σ0e+νe,σ0π+π)=(25.8  9.8+12.5)×104{\cal B}_{q\bar q}(D_s^+\to \sigma_0 e^+\nu_e,\sigma_0\to\pi^+\pi^-) =(25.8^{+12.5}_{-\;\,9.8})\times 10^{-4} and Bq2qˉ2(Ds+σ0e+νe,σ0π+π)=(1.51.3+2.4)×104{\cal B}_{q^2\bar q^2}(D_s^+\to \sigma_0 e^+\nu_e,\sigma_0\to\pi^+\pi^-) =(1.5^{+2.4}_{-1.3})\times 10^{-4} are predicted to deviate far from each other, useful for a clear experimental investigation.Comment: 10 pages, 1 figure, 1 tabl

    Probe of Spin Dynamics in Superconducting NbN Thin Films via Spin Pumping

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    The emerging field of superconductor (SC) spintronics has attracted intensive attentions recently. Many fantastic spin dependent properties in SC have been discovered, including the observation of large magnetoresistance, long spin lifetimes and the giant spin Hall effect in SC, as well as spin supercurrent in Josephson junctions, etc. Regarding the spin dynamic in SC films, few studies has been reported yet. Here, we report the investigation of the spin dynamics in an s-wave superconducting NbN film via spin pumping from an adjacent insulating ferromagnet GdN layer. A profound coherence peak of the Gilbert damping is observed slightly below the superconducting critical temperature of the NbN layer, which is consistent with recent theoretical studies. Our results further indicate that spin pumping could be a powerful tool for investigating the spin dynamics in 2D crystalline superconductors.Comment: 11 pages, 4 figures, and S

    LIF Upregulates Expression of NK-1R in NHBE Cells

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    Leukemia inhibitory factor (LIF), a cytokine at the interface between neurobiology and immunology, is mainly mediated through JAK/STAT pathway and MAPK/ERK pathway. Evidence suggested LIF is related to the higher expression of neurokinin-1 receptor (NK-1R) in asthma. In this study, the immunohistochemistry stain showed the expressions of NK-1R, LIF, p-STAT3, and p-ERK1/2 in the lung tissues of allergic rats were increased compared with the controls, and the main positive cell type was airway epithelial cell. Normal human bronchial epithelial cells were treated with LIF in the presence or absence of AG490 (JAK2 inhibitor), PD98059 (MEK inhibitor), and the siRNA against STAT3. Western blot and RT-PCR indicated that LIF induced the expression of NK-1R, which was inhibited by the inhibitors mentioned above. No significant interaction was found between JAK/STAT pathway and MAPK/ERK pathway. In summary, bronchial epithelial cell changes in asthma are induced by LIF which promotes the expression of NK-1R, and JAK/STAT pathway and MAPK/ERK pathway may participate in this process

    Apocynum Tablet Protects against Cardiac Hypertrophy via Inhibiting AKT and ERK1/2 Phosphorylation after Pressure Overload

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    Background. Cardiac hypertrophy occurs in many cardiovascular diseases. Apocynum tablet (AT), a traditional Chinese medicine, has been widely used in China to treat patients with hypertension. However, the underlying molecular mechanisms of AT on the hypertension-induced cardiac hypertrophy remain elusive. The current study evaluated the effect and mechanisms of AT on cardiac hypertrophy. Methods. We created a mouse model of cardiac hypertrophy by inducing pressure overload with surgery of transverse aortic constriction (TAC) and then explored the effect of AT on the development of cardiac hypertrophy using 46 mice in 4 study groups (combinations of AT and TAC). In addition, we evaluated the signaling pathway of phosphorylation of ERK1/2, AKT, and protein expression of GATA4 in the cardioprotective effects of AT using Western blot. Results. AT inhibited the phosphorylation of Thr202/Tyr204 sites of ERK1/2, Ser473 site of AKT, and protein expression of GATA4 and significantly inhibited cardiac hypertrophy and cardiac fibrosis at 2 weeks after TAC surgery (P<0.05). Conclusions. We experimentally demonstrated that AT inhibits cardiac hypertrophy via suppressing phosphorylation of ERK1/2 and AKT

    {1,8-Bis[2-(2-oxidobenzyl­idene­amino)phen­oxy]-3,6-dioxaocta­ne}nitrato­praseodymium(III) trichloro­methane solvate

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    In the title compound, [Pr(C32H30N2O6)(NO3)]·CHCl3, the PrIII ion is ten-coordinated by eight O atoms and two N atoms from the acyclic crown-type Schiff base ligand and the bidentate nitrate group. The coordination polyhedron around PrIII is a distorted bicapped square anti­prism. The chloro­form solvent mol­ecule is not involved either in coordination to the PrIII center or in hydrogen bonding to the complex. The Pr—O(phenolate) bonds are significantly shorter than the Pr—O(ether) and Pr—O(nitrate) bonds, which suggests that the Pr—O(phenolate) bond is stronger than these other bonds. In the crystal structure, the acyclic crown-type Schiff base ligand wraps around the PrIII centre, forming a pseudo-ring
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