Zc(3900)Z_c(3900) as a DDˉ∗D\bar{D}^* molecule from the pole counting rule

A comprehensive study on the nature of the $Z_c(3900)$ resonant structure is carried out in this work. By constructing the pertinent effective Lagrangians and considering the important final-state-interaction effects, we first give a unified description to all the relevant experimental data available, including the $J/\psi\pi$ and $\pi\pi$ invariant mass distributions from the $e^+e^-\to J/\psi\pi\pi$ process, the $h_c\pi$ distribution from $e^+e^-\to h_c\pi\pi$ and also the $D\bar D^{*}$ spectrum in the $e^+e^-\to D\bar D^{*}\pi$ process. After fitting the unknown parameters to the previous data, we search the pole in the complex energy plane and find only one pole in the nearby energy region in different Riemann sheets. Therefore we conclude that $Z_c(3900)$ is of $D\bar D^*$ molecular nature, according to the pole counting rule method~[Nucl.~Phys.~A543, 632 (1992); Phys.~Rev.~D 35,~1633 (1987)]. We emphasize that the conclusion based upon the pole counting method is not trivial, since both the $D\bar D^{*}$ contact interactions and the explicit $Z_c$ exchanges are introduced in our analyses and they lead to the same conclusion.Comment: 21 pages, 9 figures. To match the published version in PRD. Additional discussion on the spectral density function is include

Non-Abelian Chiral Spin Liquid on the Kagome Lattice

We study $S=1$ spin liquid states on the kagome lattice constructed by Gutzwiller-projected $p_x+ip_y$ superconductors. We show that the obtained spin liquids are either non-Abelian or Abelian topological phases, depending on the topology of the fermionic mean-field state. By calculating the modular matrices $S$ and $T$, we confirm that projected topological superconductors are non-Abelian chiral spin liquid (NACSL). The chiral central charge and the spin Hall conductance we obtained agree very well with the $SO(3)_1$ (or, equivalently, $SU(2)_2$) field theory predictions. We propose a local Hamiltonian which may stabilize the NACSL. From a variational study we observe a topological phase transition from the NACSL to the $Z_2$ Abelian spin liquid.Comment: 12 pages, 7 figures, 1 tabl

The protective effect of resveratrol on human lens epithelial cells against ultraviolet-induced apoptosis

AIM: To investigate the protective effect of resveratrol on human lens epithelial cells against ultraviolet-induced apoptosis. METHODS:Subcultured human lens epithelial cell line, ultraviolet induced cell apoptosis, 20μmol/L resveratrol pretreated cell, the indicators change was observed: rate of apoptosis was detected by flow cytometry and apoptosis-related factors of caspses-3 and caspase-9 were detected by colorimetric detection, ultrastructure changes were observed under transmission electron microscope. RESULTS: Flow cytometry instrument testing found that resveratrol can suppress the apoptosis induced by ultraviolet irradiation, caspses-3 and caspase-9 content in positive control group were significantly higher than that of the negative control group at the same time period, the difference was statistically significant(P<0.05); caspses-3 and caspase-9 content in experimental group were lower than that in the positive control group at the same time, the difference was statistically significant(P<0.05). In addition, the damage of human lens epithelial cells was alleviated with the incubation time of resveratrol elongated. CONCLUSION:Resveratrol may inhibit ultraviolet-induced apoptosis of human lens epithelial cells, it has preventive function against radioactive cataract, and it can provide reliable evidence for pursuing effective medicine to prevent and treat cataract

C. elegans fatty acid two-hydroxylase regulates intestinal homeostasis by affecting heptadecenoic acid production

Background/Aims: The hydroxylation of fatty acids at the C-2 position is the first step of fatty acid α-oxidation and generates sphingolipids containing 2-hydroxy fatty acyl moieties. Fatty acid 2-hydroxylation is catalyzed by Fatty acid 2-hydroxylase (FA2H) enzyme. However, the precise roles of FA2H and fatty acid 2-hydroxylation in whole cell homeostasis still remain unclear. Methods: Here we utilize Caenorhabditis elegans as the model and systemically investigate the physiological functions of FATH-1/C25A1.5, the highly conserved worm homolog for mammalian FA2H enzyme. Immunostaining, dye-staining and translational fusion reporters were used to visualize FATH-1 protein and a variety of subcellular structures. The “click chemistry” method was employed to label 2-OH fatty acid in vivo. Global and tissue-specific RNAi knockdown experiments were performed to inactivate FATH-1 function. Lipid analysis of the fath-1 deficient mutants was achieved by mass spectrometry. Results: C. elegans FATH-1 is expressed at most developmental stages and in most tissues. Loss of fath-1 expression results in severe growth retardation and shortened lifespan. FATH-1 function is crucially required in the intestine but not the epidermis with stereospecificity. The “click chemistry” labeling technique showed that the FATH-1 metabolites are mainly enriched in membrane structures preferable to the apical side of the intestinal cells. At the subcellular level, we found that loss of fath-1 expression inhibits lipid droplets formation, as well as selectively disrupts peroxisomes and apical endosomes. Lipid analysis of the fath-1 deficient animals revealed a significant reduction in the content of heptadecenoic acid, while other major FAs remain unaffected. Feeding of exogenous heptadecenoic acid (C17: 1), but not oleic acid (C18: 1), rescues the global and subcellular defects of fath-1 knockdown worms. Conclusion: Our study revealed that FATH-1 and its catalytic products are highly specific in the context of chirality, C-chain length, spatial distribution, as well as the types of cellular organelles they affect. Such an unexpected degree of specificity for the synthesis and functions of hydroxylated FAs helps to regulate protein transport and fat metabolism, therefore maintaining the cellular homeostasis of the intestinal cells. These findings may help our understanding of FA2H functions across species, and offer potential therapeutical targets for treating FA2H-related diseases

On leptonic width of X(4260)X(4260)

New measurements on cross sections in $e^+e^-\to J/\psi\pi^+\pi^-$, $h_c\pi^+\pi^-$, $D^0D^{*-}\pi^++c.c.$, $\psi(2S)\pi^+\pi^-$, $\omega\chi_{c0}$ and $J/\psi\eta$ channels have been carried out by BESIII, Belle and BABAR collaborations, and also in the $D_s^*\bar D_s^*$ channel. We perform extensive numerical analyses by combining all these data available, together with those in $D\bar D^*$ and $D^*\bar D^*$ channels. Though the latter show no evident peak around $\sqrt{s}=4.230$ GeV, the missing $X(4260)$ is explained as that it is concealed by the interference effects of the well established charmonia $\psi(4040)$, $\psi(4160)$ and $\psi(4415)$. Our analyses reveal that the leptonic decay width of $X(4260)$ ranges from $O(10^2)$ eV to $O(1)$ keV, and hence may be explained in the conventional quark model picture. That is, the $X(4260)$ may well be interpreted as a mixture of $4^3S_1$ and $3^3D_1$ states.Comment: two small mistakes are fixed, figures redrawn, major physical outputs remain unchanged. Version published in EPJ