8 research outputs found
Π ΠΠΠΠ ΠΠ‘Π£ ΠΠ ΠΠ¨ΠΠΠΠ§ΠΠ‘ΠΠΠ ΠΠΠ‘Π€Π£ΠΠΠ¦ΠΠ ΠΠΠΠΠΠ ΠΠ
The authors of the review have analyzed papers published on the problem of ischemic myocardial dysfunction. They begin with a definition of the term βischemiaβ (derived from two Greek words:Β ischΕ, meaning to hold back, and haima, meaning blood) - a condition at which the arterial blood flow is insufficient to provide enough oxygen to prevent intracellular respiration from shifting from the aerobic to the anaerobic form. The poor rate of ATP generation from this process causes a decrease in cellular ATP, a concomitant rise in ADP, and ultimately, to depression inotropic (systolic) and lusitropic (diastolic) function of the affected segments of the myocardium. But with such simplicity of basic concepts, the consequences of ischemia so diverse. Influence of an ischemia on myocardial function so unequally at different patients, which is almost impossible to find two identical cases (as in the case of fingerprints). It depends on the infinite variety of lesions of coronary arteries, reperfusion (time and completeness of restoration of blood flow) and reactions of a myocardium which, apparently, has considerable flexibility in its response. Ischemic myocardial dysfunction includes a number of discrete states, such as acute left ventricular failure in angina, acute myocardial infarction, ischemic cardiomyopathy, stunning, hibernation, pre- and postconditioning. There are widely differing underlying pathophysiologic states. The possibility exists that several of these states can coexist.ΠΡΠΎΠ²Π΅Π΄Π΅Π½ Π°Π½Π°Π»ΠΈΠ· ΡΠ°Π±ΠΎΡ, ΠΎΠΏΡΠ±Π»ΠΈΠΊΠΎΠ²Π°Π½Π½ΡΡ
ΠΏΠΎ ΠΏΡΠΎΠ±Π»Π΅ΠΌΠ΅ ΠΈΡΠ΅ΠΌΠΈΡΠ΅ΡΠΊΠΎΠΉ Π΄ΠΈΡΡΡΠ½ΠΊΡΠΈΠΈ ΠΌΠΈΠΎΠΊΠ°ΡΠ΄Π°. ΠΡΠ΅ΠΌΠΈΡ (ΠΎΡ Π³ΡΠ΅Ρ. ischΕ β Π·Π°Π΄Π΅ΡΠΆΠΈΠ²Π°ΡΡ ΠΈ haima β ΠΊΡΠΎΠ²Ρ) -ΡΠΎΡΡΠΎΡΠ½ΠΈΠ΅, ΠΏΡΠΈ ΠΊΠΎΡΠΎΡΠΎΠΌ Π°ΡΡΠ΅ΡΠΈΠ°Π»ΡΠ½ΡΠΉ ΠΊΡΠΎΠ²ΠΎΡΠΎΠΊ ΡΡΠ°Π½ΠΎΠ²ΠΈΡΡΡ Π½Π΅Π΄ΠΎΡΡΠ°ΡΠΎΡΠ½ΡΠΌ, ΡΡΠΎΠ±Ρ ΠΎΠ±Π΅ΡΠΏΠ΅ΡΠΈΡΡ Π½Π΅ΠΎΠ±Ρ
ΠΎΠ΄ΠΈΠΌΠΎΠ΅ ΠΊΠΎΠ»ΠΈΡΠ΅ΡΡΠ²ΠΎ ΠΊΠΈΡΠ»ΠΎΡΠΎΠ΄Π° Π΄Π»Ρ ΠΏΡΠ΅Π΄ΠΎΡΠ²ΡΠ°ΡΠ΅Π½ΠΈΡ ΠΏΠ΅ΡΠ΅Ρ
ΠΎΠ΄Π° Π²Π½ΡΡΡΠΈΠΊΠ»Π΅ΡΠΎΡΠ½ΠΎΠ³ΠΎ Π΄ΡΡ
Π°Π½ΠΈΡ Ρ Π°ΡΡΠΎΠ±Π½ΠΎΠΉ ΡΠΎΡΠΌΡ Π½Π° Π°Π½Π°ΡΡΠΎΠ±Π½ΡΡ. ΠΠΈΠ·ΠΊΠ°Ρ ΡΠΊΠΎΡΠΎΡΡΡ Π³Π΅Π½Π΅ΡΠ°ΡΠΈΠΈ Π°Π΄Π΅Π½ΠΎΠ·ΠΈΠ½ΡΡΠΈΡΠΎΡΡΠ°ΡΠ° (ΠΠ’Π€) ΠΏΡΠΈ ΡΡΠΎΠΌ ΠΏΡΠΎΡΠ΅ΡΡΠ΅ ΠΏΡΠΈΠ²ΠΎΠ΄ΠΈΡ ΠΊ ΡΠ½ΠΈΠΆΠ΅Π½ΠΈΡ Π² ΠΊΠ»Π΅ΡΠΊΠ΅ ΠΠ’Π€, ΠΊΠΎΠ½ΠΊΠΎΠΌΠΈΠ½Π°Π½ΡΠ½ΠΎΠΌΡ ΠΏΠΎΠ²ΡΡΠ΅Π½ΠΈΡ Π°Π΄Π΅Π½ΠΎΠ·ΠΈΠ½Π΄ΠΈΡΠΎΡΡΠ°ΡΠ° ΠΈ Π² ΠΊΠΎΠ½Π΅ΡΠ½ΠΎΠΌ ΡΡΠ΅ΡΠ΅ ΠΊ Π΄Π΅ΠΏΡΠ΅ΡΡΠΈΠΈ ΠΈΠ½ΠΎΡΡΠΎΠΏΠ½ΠΎΠΉ (ΡΠΈΡΡΠΎΠ»ΠΈΡΠ΅ΡΠΊΠΎΠΉ) ΠΈ Π»ΡΡΠΈΡΡΠΎΠΏΠ½ΠΎΠΉ (Π΄ΠΈΠ°ΡΡΠΎΠ»ΠΈΡΠ΅ΡΠΊΠΎΠΉ) ΡΡΠ½ΠΊΡΠΈΠΉ ΠΏΠΎΡΠ°ΠΆΠ΅Π½Π½ΡΡ
ΡΠ΅Π³ΠΌΠ΅Π½ΡΠΎΠ² ΠΌΠΈΠΎΠΊΠ°ΡΠ΄Π°. ΠΠΎ ΠΏΡΠΈ Π²ΡΠ΅ΠΉ ΠΏΡΠΎΡΡΠΎΡΠ΅ ΡΡΠΈΡ
ΠΎΡΠ½ΠΎΠ²Π½ΡΡ
ΠΏΠΎΠ»ΠΎΠΆΠ΅Π½ΠΈΠΉ ΠΏΠΎΡΠ»Π΅Π΄ΡΡΠ²ΠΈΡ ΠΈΡΠ΅ΠΌΠΈΠΈ ΠΊΡΠ°ΠΉΠ½Π΅ ΡΠ°Π·Π½ΠΎΠΎΠ±ΡΠ°Π·Π½Ρ. ΠΠ»ΠΈΡΠ½ΠΈΠ΅ ΠΈΡΠ΅ΠΌΠΈΠΈ Π½Π° ΡΡΠ½ΠΊΡΠΈΡ ΠΌΠΈΠΎΠΊΠ°ΡΠ΄Π° Π½Π°ΡΡΠΎΠ»ΡΠΊΠΎ Π½Π΅ΠΎΠ΄ΠΈΠ½Π°ΠΊΠΎΠ²ΠΎ Ρ ΡΠ°Π·Π»ΠΈΡΠ½ΡΡ
ΠΏΠ°ΡΠΈΠ΅Π½ΡΠΎΠ², ΡΡΠΎ ΠΏΡΠ°ΠΊΡΠΈΡΠ΅ΡΠΊΠΈ Π½Π΅Π²ΠΎΠ·ΠΌΠΎΠΆΠ½ΠΎ Π²ΡΡΡΠ΅ΡΠΈΡΡ Π΄Π²Π° ΠΈΠ΄Π΅Π½ΡΠΈΡΠ½ΡΡ
ΡΠ»ΡΡΠ°Ρ (ΡΠ°ΠΊΠΆΠ΅ ΠΊΠ°ΠΊ ΠΎΠ΄ΠΈΠ½Π°ΠΊΠΎΠ²ΡΠ΅ ΠΎΡΠΏΠ΅ΡΠ°ΡΠΊΠΈ ΠΏΠ°Π»ΡΡΠ΅Π²). ΠΠ°Π²ΠΈΡΠΈΡ ΡΡΠΎ ΠΎΡ Π±Π΅ΡΠΊΠΎΠ½Π΅ΡΠ½ΠΎΠ³ΠΎ ΡΠ°Π·Π½ΠΎΠΎΠ±ΡΠ°Π·ΠΈΡ ΠΏΠΎΡΠ°ΠΆΠ΅Π½ΠΈΠΉ ΠΊΠΎΡΠΎΠ½Π°ΡΠ½ΡΡ
Π°ΡΡΠ΅ΡΠΈΠΉ, ΡΠ΅ΠΏΠ΅ΡΡΡΠ·ΠΈΠΈ (Π²ΡΠ΅ΠΌΡ ΠΈ ΠΏΠΎΠ»Π½ΠΎΡΠ° Π²ΠΎΡΡΡΠ°Π½ΠΎΠ²Π»Π΅Π½ΠΈΡ ΠΊΡΠΎΠ²ΠΎΡΠΎΠΊΠ°) ΠΈ ΡΠ΅Π°ΠΊΡΠΈΠΈ ΠΌΠΈΠΎΠΊΠ°ΡΠ΄Π°, ΠΊΠΎΡΠΎΡΡΠΉ, ΠΏΠΎ-Π²ΠΈΠ΄ΠΈΠΌΠΎΠΌΡ, ΠΈΠΌΠ΅Π΅Ρ Π·Π½Π°ΡΠΈΡΠ΅Π»ΡΠ½ΡΡ Π³ΠΈΠ±ΠΊΠΎΡΡΡ Π² ΡΡΠΎΠΌ ΠΎΡΠ²Π΅ΡΠ΅. ΠΡΠ΅ΠΌΠΈΡΠ΅ΡΠΊΠ°Ρ Π΄ΠΈΡΡΡΠ½ΠΊΡΠΈΡ ΠΌΠΈΠΎΠΊΠ°ΡΠ΄Π° Π²ΠΊΠ»ΡΡΠ°Π΅Ρ Π² ΡΠ΅Π±Ρ ΡΡΠ΄ Π΄ΠΈΡΠΊΡΠ΅ΡΠ½ΡΡ
ΡΠΎΡΡΠΎΡΠ½ΠΈΠΉ, ΡΠ°ΠΊΠΈΡ
ΠΊΠ°ΠΊ ΠΎΡΡΡΠ°Ρ Π»Π΅Π²ΠΎΠΆΠ΅Π»ΡΠ΄ΠΎΡΠΊΠΎΠ²Π°Ρ Π½Π΅Π΄ΠΎΡΡΠ°ΡΠΎΡΠ½ΠΎΡΡΡ ΠΏΡΠΈ ΡΡΠ΅Π½ΠΎΠΊΠ°ΡΠ΄ΠΈΠΈ, ΠΎΡΡΡΡΠΉ ΠΈΠ½ΡΠ°ΡΠΊΡ ΠΌΠΈΠΎΠΊΠ°ΡΠ΄Π°, ΠΈΡΠ΅ΠΌΠΈΡΠ΅ΡΠΊΠ°Ρ ΠΊΠ°ΡΠ΄ΠΈΠΎΠΌΠΈΠΎΠΏΠ°ΡΠΈΡ, ΠΎΠ³Π»ΡΡΠ΅Π½ΠΈΠ΅, Π³ΠΈΠ±Π΅ΡΠ½Π°ΡΠΈΡ ΠΈ ΠΏΡΠ΅- ΠΈ ΠΏΠΎΡΡΠΊΠΎΠ½Π΄ΠΈΡΠΈΠΎΠ½ΠΈΡΠΎΠ²Π°Π½ΠΈΠ΅. ΠΠ΅Ρ
Π°Π½ΠΈΠ·ΠΌΡ ΡΡΠΈΡ
ΡΠΎΡΡΠΎΡΠ½ΠΈΠΉ Π² Π·Π½Π°ΡΠΈΡΠ΅Π»ΡΠ½ΠΎΠΉ ΡΡΠ΅ΠΏΠ΅Π½ΠΈ ΡΠ°Π·Π»ΠΈΡΠ°ΡΡΡΡ, ΡΡΠΎ Π½Π΅ΠΎΠ±Ρ
ΠΎΠ΄ΠΈΠΌΠΎ ΡΡΠΈΡΡΠ²Π°ΡΡ ΠΏΡΠΈ ΡΠ°Π·ΡΠ°Π±ΠΎΡΠΊΠ΅ ΠΏΡΠΎΠ³ΡΠ°ΠΌΠΌ ΠΏΡΠΎΡΠΈΠ»Π°ΠΊΡΠΈΠΊΠΈ (ΠΏΠ΅ΡΠ²ΠΈΡΠ½ΠΎΠΉ ΠΈ Π²ΡΠΎΡΠΈΡΠ½ΠΎΠΉ). ΠΡΠΈ ΡΡΠΎΠΌ ΡΠ»Π΅Π΄ΡΠ΅Ρ ΠΏΡΠΈΠ½ΠΈΠΌΠ°ΡΡ Π²ΠΎ Π²Π½ΠΈΠΌΠ°Π½ΠΈΠ΅ Π²ΠΎΠ·ΠΌΠΎΠΆΠ½ΠΎΡΡΡ ΡΠΎΠ³ΠΎ, ΡΡΠΎ Π½Π΅ΠΊΠΎΡΠΎΡΡΠ΅ ΠΈΠ· ΡΡΠΈΡ
ΡΠΎΡΡΠΎΡΠ½ΠΈΠΉ ΠΌΠΎΠ³ΡΡ ΡΠΎΡΡΡΠ΅ΡΡΠ²ΠΎΠ²Π°ΡΡ
ΠΡΠ³Π°Π½ΠΎΠΏΡΠΎΡΠ΅ΠΊΡΠΈΠ²Π½Π°Ρ ΡΡΡΠ΅ΠΊΡΠΈΠ²Π½ΠΎΡΡΡ ΡΠΏΡΠΎΡΠ°ΡΡΠ°Π½Π° Ρ Π±ΠΎΠ»ΡΠ½ΡΡ Ρ ΡΠΎΠ½ΠΈΡΠ΅ΡΠΊΠΈΠΌ Π³Π»ΠΎΠΌΠ΅ΡΡΠ»ΠΎΠ½Π΅ΡΡΠΈΡΠΎΠΌ
The 12-week open uncontrollable study of eprosartan organoprotective efficacy was performed in 15 patients with chronic glomerulonephritis. Study results showed high organoprotective efficacy of eprosartan, prescribed in a dose of 600 mg daily, which is expressed as anti-proteinuric, anti-hematuric actions, positive dynamics of intraglomerular hemodynamics parameters, decreased left ventricular hypertrophy and stiffness and also normalized vasomotoric functions of brachial artery endothelium.ΠΡΠΎΠ²Π΅Π΄Π΅Π½ΠΎ 12-Π½Π΅Π΄Π΅Π»ΡΠ½ΠΎΠ΅ ΠΎΡΠΊΡΡΡΠΎΠ΅ Π½Π΅ΠΊΠΎΠ½ΡΡΠΎΠ»ΠΈΡΡΠ΅ΠΌΠΎΠ΅ ΠΈΡΡΠ»Π΅Π΄ΠΎΠ²Π°Π½ΠΈΠ΅ ΠΎΡΠ³Π°Π½ΠΎΠΏΡΠΎΡΠ΅ΠΊΡΠΈΠ²Π½ΠΎΠΉ ΡΡΡΠ΅ΠΊΡΠΈΠ²Π½ΠΎΡΡΠΈ ΡΠΏΡΠΎΡΠ°ΡΡΠ°Π½Π° Ρ 15 ΠΏΠ°ΡΠΈΠ΅Π½ΡΠΎΠ² Ρ Ρ
ΡΠΎΠ½ΠΈΡΠ΅ΡΠΊΠΈΠΌ Π³Π»ΠΎΠΌΠ΅ΡΡΠ»ΠΎΠ½Π΅ΡΡΠΈΡΠΎΠΌ. Π Π΅Π·ΡΠ»ΡΡΠ°ΡΡ ΠΈΡΡΠ»Π΅Π΄ΠΎΠ²Π°Π½ΠΈΡ ΡΠ²ΠΈΠ΄Π΅ΡΠ΅Π»ΡΡΡΠ²ΡΡΡ ΠΎ Π²ΡΡΠΎΠΊΠΎΠΉ ΠΎΡΠ³Π°Π½ΠΎΠΏΡΠΎΡΠ΅ΠΊΡΠΈΠ²Π½ΠΎΠΉ ΡΡΡΠ΅ΠΊΡΠΈΠ²Π½ΠΎΡΡΠΈ ΡΠΏΡΠΎΡΠ°ΡΡΠ°Π½Π°, Π½Π°Π·Π½Π°ΡΠ΅Π½Π½ΠΎΠ³ΠΎ Π² Π΄ΠΎΠ·Π΅ 600 ΠΌΠ³ Π² ΡΡΡΠΊΠΈ, ΠΊΠΎΡΠΎΡΠ°Ρ ΠΏΡΠΎΡΠ²Π»ΡΠ΅ΡΡΡ Π² Π²ΠΈΠ΄Π΅ Π°Π½ΡΠΈΠΏΡΠΎΡΠ΅ΠΈΠ½ΡΡΠΈΡΠ΅ΡΠΊΠΎΠ³ΠΎ, Π°Π½ΡΠΈΠ³Π΅ΠΌΠ°ΡΡΡΠΈΡΠ΅ΡΠΊΠΎΠ³ΠΎ Π΄Π΅ΠΉΡΡΠ²ΠΈΡ, ΠΏΠΎΠ»ΠΎΠΆΠΈΡΠ΅Π»ΡΠ½ΠΎΠΉ Π΄ΠΈΠ½Π°ΠΌΠΈΠΊΠΈ ΠΏΠ°ΡΠ°ΠΌΠ΅ΡΡΠΎΠ² Π²Π½ΡΡΡΠΈΠΊΠ»ΡΠ±ΠΎΡΠΊΠΎΠ²ΠΎΠΉ Π³Π΅ΠΌΠΎΡΠΈΡΠΊΡΠ»ΡΡΠΈΠΈ, ΡΠΌΠ΅Π½ΡΡΠ΅Π½ΠΈΡ Π³ΠΈΠΏΠ΅ΡΡΡΠΎΡΠΈΠΈ ΠΈ ΠΆΠ΅ΡΡΠΊΠΎΡΡΠΈ Π»Π΅Π²ΠΎΠ³ΠΎ ΠΆΠ΅Π»ΡΠ΄ΠΎΡΠΊΠ°, Π° ΡΠ°ΠΊΠΆΠ΅ Π½ΠΎΡΠΌΠ°Π»ΠΈΠ·Π°ΡΠΈΠΈ ΡΠΎΡΡΠ΄ΠΎΠ΄Π²ΠΈΠ³Π°ΡΠ΅Π»ΡΠ½ΠΎΠΉ ΡΡΠ½ΠΊΡΠΈΠΈ ΡΠ½Π΄ΠΎΡΠ΅Π»ΠΈΡ ΠΏΠ»Π΅ΡΠ΅Π²ΠΎΠΉ Π°ΡΡΠ΅ΡΠΈΠΈ
ΠΠΠ‘ΠΠΠ¦ΠΠ€ΠΠ§ΠΠ‘ΠΠΠ ΠΠΠ₯ΠΠΠΠΠΠ« ΠΠ ΠΠΠ ΠΠ‘Π‘ΠΠ ΠΠΠΠΠΠ― Π₯Π ΠΠΠΠ§ΠΠ‘ΠΠΠ ΠΠΠΠΠΠΠ ΠΠΠ§ΠΠ
The authors of the review have analyzed papers published on the problem of pathogenesis chronic kidney disease (CKD), which the experts of the K/DOQI (Kidney Disease Outcomes Quality Initiative) Advisory Board (National Kidney Foundation, USA) is defined as the presence of kidney damage or decreased level of kidney function for three months or more, irrespective of diagnosis. It is shown that the specific mechanisms which result directly from the nature of the disease, completely determine the course of CKD in its initial stage, whereas further reduction in the number of intact nephrons, cascade is initiated univer-sal for all of nephropathy pathological processes, culminating in the formation of nephrosclerosis, often even if the reason that caused the initial damage to the nephrons is eliminated: hyperfiltration, hypercoag-ulability, impaired renal transport protein, changes in the expression of mediators of cell damage, meta-bolic and endocrine mechanisms, polymorphism of genes controlling the expression of nephrotropic bio-logically active substances. Analyzed published data demonstrate that renal protection strategy (complex therapies aimed at the inhibition of the irreversible deterioration of kidney function and affect common to all of nephropathy mechanisms of progression) can slow the progression of CKD.Β ΠΠ²ΡΠΎΡΡ ΠΎΠ±Π·ΠΎΡΠ° Π°Π½Π°Π»ΠΈΠ·ΠΈΡΡΡΡ ΡΠ°Π±ΠΎΡΡ, ΠΎΠΏΡΠ±Π»ΠΈΠΊΠΎΠ²Π°Π½Π½ΡΠ΅ ΠΏΠΎ ΠΏΡΠΎΠ±Π»Π΅ΠΌΠ΅ ΠΏΠ°ΡΠΎΠ³Π΅Π½Π΅Π·Π° Ρ
ΡΠΎΠ½ΠΈΡΠ΅ΡΠΊΠΎΠΉ Π±ΠΎΠ»Π΅Π·-Π½ΠΈ ΠΏΠΎΡΠ΅ΠΊ (Π₯ΠΠ), ΠΊΠΎΡΠΎΡΡΡ ΡΠΊΡΠΏΠ΅ΡΡΡ ΠΠΎΠ½ΡΡΠ»ΡΡΠ°ΡΠΈΠ²Π½ΠΎΠ³ΠΎ ΡΠΎΠ²Π΅ΡΠ° ΠΈΠ½ΠΈΡΠΈΠ°ΡΠΈΠ²Ρ ΠΊΠ°ΡΠ΅ΡΡΠ²Π° Π»Π΅ΡΠ΅Π½ΠΈΡ Π·Π°Π±ΠΎ-Π»Π΅Π²Π°Π½ΠΈΡ ΠΏΠΎΡΠ΅ΠΊ ΠΠ°ΡΠΈΠΎΠ½Π°Π»ΡΠ½ΠΎΠ³ΠΎ ΠΏΠΎΡΠ΅ΡΠ½ΠΎΠ³ΠΎ ΡΠΎΠ½Π΄Π° Π‘Π¨Π ΠΎΠΏΡΠ΅Π΄Π΅Π»ΡΡΡ ΠΊΠ°ΠΊ Π½Π°Π»ΠΈΡΠΈΠ΅ ΠΏΠΎΡΠ΅ΡΠ½ΠΎΠ³ΠΎ ΠΏΠΎΠ²ΡΠ΅-ΠΆΠ΄Π΅Π½ΠΈΡ ΠΈΠ»ΠΈ ΡΠ½ΠΈΠΆΠ΅Π½Π½ΠΎΠ³ΠΎ ΡΡΠΎΠ²Π½Ρ ΡΡΠ½ΠΊΡΠΈΠΈ ΠΏΠΎΡΠ΅ΠΊ Π½Π° ΠΏΡΠΎΡΡΠΆΠ΅Π½ΠΈΠΈ Π½Π΅ ΠΌΠ΅Π½Π΅Π΅ 3 ΠΌΠ΅Ρ Π½Π΅Π·Π°Π²ΠΈΡΠΈΠΌΠΎ ΠΎΡ ΡΡΠΈΠΎ-Π»ΠΎΠ³ΠΈΠΈ. ΠΠΎΠΊΠ°Π·Π°Π½ΠΎ, ΡΡΠΎ ΡΠΏΠ΅ΡΠΈΡΠΈΡΠ΅ΡΠΊΠΈΠ΅ ΠΌΠ΅Ρ
Π°Π½ΠΈΠ·ΠΌΡ, ΠΎΠ±ΡΡΠ»ΠΎΠ²Π»Π΅Π½Π½ΡΠ΅ Π½Π΅ΠΏΠΎΡΡΠ΅Π΄ΡΡΠ²Π΅Π½Π½ΠΎ Ρ
Π°ΡΠ°ΠΊΡΠ΅ΡΠΎΠΌ Π·Π°Π±ΠΎΠ»Π΅Π²Π°Π½ΠΈΡ, ΡΠ΅Π»ΠΈΠΊΠΎΠΌ ΠΎΠΏΡΠ΅Π΄Π΅Π»ΡΡΡ ΡΠ΅ΡΠ΅Π½ΠΈΠ΅ Π₯ΠΠ Π»ΠΈΡΡ Π² Π½Π°ΡΠ°Π»ΡΠ½ΠΎΠΉ ΡΡΠ°Π΄ΠΈΠΈ, ΡΠΎΠ³Π΄Π° ΠΊΠ°ΠΊ ΠΏΡΠΈ ΡΠ΅Π΄ΡΠΊΡΠΈΠΈ ΡΠΈΡΠ»Π° ΠΈΠ½ΡΠ°ΠΊΡΠ½ΡΡ
Π½Π΅ΡΡΠΎΠ½ΠΎΠ² ΠΈΠ½ΠΈΡΠΈΠΈΡΡΠ΅ΡΡΡ ΠΊΠ°ΡΠΊΠ°Π΄ ΡΠ½ΠΈΠ²Π΅ΡΡΠ°Π»ΡΠ½ΡΡ
Π΄Π»Ρ Π²ΡΠ΅Ρ
Π½Π΅ΡΡΠΎΠΏΠ°ΡΠΈΠΉ ΠΏΠ°ΡΠΎΠ»ΠΎΠ³ΠΈΡΠ΅-ΡΠΊΠΈΡ
ΠΏΡΠΎΡΠ΅ΡΡΠΎΠ², Π·Π°Π²Π΅ΡΡΠ°ΡΡΠΈΡ
ΡΡ ΡΠΎΡΠΌΠΈΡΠΎΠ²Π°Π½ΠΈΠ΅ΠΌ Π½Π΅ΡΡΠΎΡΠΈΠ±ΡΠΎΠ·Π°, Π΄Π°ΠΆΠ΅ Π΅ΡΠ»ΠΈ ΠΏΡΠΈΡΠΈΠ½Π°, Π²ΡΠ·Π²Π°Π²ΡΠ°Ρ ΠΏΠ΅ΡΠ²ΠΎΠ½Π°ΡΠ°Π»ΡΠ½ΠΎΠ΅ ΠΏΠΎΠ²ΡΠ΅ΠΆΠ΄Π΅Π½ΠΈΠ΅ Π½Π΅ΡΡΠΎΠ½ΠΎΠ² ΡΡΡΡΠ°Π½Π΅Π½Π°: Π³ΠΈΠΏΠ΅ΡΡΠΈΠ»ΡΡΡΠ°ΡΠΈΡ, Π³ΠΈΠΏΠ΅ΡΠΊΠΎΠ°Π³ΡΠ»ΡΡΠΈΡ, Π½Π°ΡΡΡΠ΅-Π½ΠΈΠ΅ ΠΏΠΎΡΠ΅ΡΠ½ΠΎΠ³ΠΎ ΡΡΠ°Π½ΡΠΏΠΎΡΡΠ° Π±Π΅Π»ΠΊΠ°, ΠΈΠ·ΠΌΠ΅Π½Π΅Π½ΠΈΠ΅ ΡΠΊΡΠΏΡΠ΅ΡΡΠΈΠΈ ΠΌΠ΅Π΄ΠΈΠ°ΡΠΎΡΠΎΠ² ΠΏΠΎΠ²ΡΠ΅ΠΆΠ΄Π΅Π½ΠΈΡ ΠΊΠ»Π΅ΡΠΎΠΊ, ΠΌΠ΅ΡΠ°Π±ΠΎ-Π»ΠΈΡΠ΅ΡΠΊΠΈΠ΅ ΠΈ ΡΠ½Π΄ΠΎΠΊΡΠΈΠ½Π½ΡΠ΅ ΠΌΠ΅Ρ
Π°Π½ΠΈΠ·ΠΌΡ, ΠΏΠΎΠ»ΠΈΠΌΠΎΡΡΠΈΠ·ΠΌ Π³Π΅Π½ΠΎΠ², ΠΊΠΎΠ½ΡΡΠΎΠ»ΠΈΡΡΡΡΠΈΡ
ΡΠΊΡΠΏΡΠ΅ΡΡΠΈΡ Π½Π΅ΡΡΠΎ-ΡΡΠΎΠΏΠ½ΡΡ
Π±ΠΈΠΎΠ»ΠΎΠ³ΠΈΡΠ΅ΡΠΊΠΈ Π°ΠΊΡΠΈΠ²Π½ΡΡ
Π²Π΅ΡΠ΅ΡΡΠ². ΠΠΏΡΠ±Π»ΠΈΠΊΠΎΠ²Π°Π½Π½ΡΠ΅ Π΄Π°Π½Π½ΡΠ΅ Π΄Π΅ΠΌΠΎΠ½ΡΡΡΠΈΡΡΡΡ, ΡΡΠΎ Π½Π΅ΡΡΠΎ-ΠΏΡΠΎΡΠ΅ΠΊΡΠΈΠ²Π½Π°Ρ ΡΡΡΠ°ΡΠ΅Π³ΠΈΡ (ΠΊΠΎΠΌΠΏΠ»Π΅ΠΊΡ ΠΌΠ΅ΡΠΎΠ΄ΠΎΠ² Π»Π΅ΡΠ΅Π½ΠΈΡ, Π½Π°ΠΏΡΠ°Π²Π»Π΅Π½Π½ΡΡ
Π½Π° ΡΠΎΡΠΌΠΎΠΆΠ΅Π½ΠΈΠ΅ Π½Π΅ΠΎΠ±ΡΠ°ΡΠΈΠΌΠΎΠ³ΠΎ ΡΡ
ΡΠ΄ΡΠ΅Π½ΠΈΡ ΡΡΠ½ΠΊΡΠΈΠΈ ΠΏΠΎΡΠ΅ΠΊ ΠΈ Π²ΠΎΠ·Π΄Π΅ΠΉΡΡΠ²ΡΡΡΠΈΡ
Π½Π° ΠΎΠ±ΡΠΈΠ΅ Π΄Π»Ρ Π²ΡΠ΅Ρ
Π½Π΅ΡΡΠΎΠΏΠ°ΡΠΈΠΉ ΠΌΠ΅Ρ
Π°Π½ΠΈΠ·ΠΌΡ ΠΏΡΠΎΠ³ΡΠ΅Ρ-ΡΠΈΡΠΎΠ²Π°Π½ΠΈΡ) ΠΏΠΎΠ·Π²ΠΎΠ»ΡΠ΅Ρ Π·Π°ΠΌΠ΅Π΄Π»ΠΈΡΡ ΡΠ°Π·Π²ΠΈΡΠΈΠ΅ Π₯ΠΠ
Functionally-morphological kidneys condition in patients with rheumatoid arthritis
The aim of the investigation was to study the functional-morphological state of kidney in patients with rheumatoid arthritis (RΠ). Examination was performed on 75 patients with RA and 25 healthy subjects. In addition to complete clinical and instrumental examination accepted in specialised clinic, biopsy of kidney was conducted. At 62 patients changes in an urinary deposit are taped. According to 25 nephrobiopsies in 12 cases took place mesangioproliferative variant of a chronic glomerulonephritis, the amyloidosis was detected at 5 patients, in 8 cases - vascular changes, the weak and moderate phenomena of a productive vasculitis of arterioles, the capillaries, accompanied by a focal perivascular sclerosis. Nephroprotective effect of basic therapy of RΠ was found
Chronic kidney disease in patients with psoriasis, chronic opisthorchiasis and at their combination
The aim of the investigation was to study of features of Ρhronic kidney disease in patients with chronic opisthorchiasis (CO), psoriasis (Ps) and their combination. Examination was performed on 360 patients with CO, 100 patients with Ps, 50 patients with Ps combinated CO and 30 healthy subjects. In addition to complete clinical and instrumental examination accepted in specialised clinic, biopsy of kidney was conducted. The interrelation of intensity of sclerous processes in kidneys and renoprival syndrome with duration of CO and activity of Ps was found. Dehelmintization with biltricid promoted improvement of functional state of kidneys