Enhanced Intralayer Ferromagnetism in CrI₃ by Interfacial Super-Superexchange Interaction

Manipulating the interlayer exchange interaction in two-dimensional (2D) layered materials is crucial for achieving intrinsic long-range magnetic ordering for high-performance spintronic devices. In this work, we propose a general and experimentally feasible approach to enhance the ferromagnetism of a monolayer material in van der Waals (vdW) heterostructures by taking advantage of the interfacial super-superexchange interaction. As a proof of concept, we consider the CrI3/Fe3GeTe2 heterostructure with a strong Cr–I···Te–Fe super-superexchange interaction. Our first-principles calculations show that the interlayer distance and electronic coupling between CrI3 and Fe3GeTe2 sheets highly depend on their stacking geometry, exhibiting distinct weak and strong coupling regions. Specifically, a Cr–I–Te angle of ∼103° leads to the strongest interfacial coupling, robust ferromagnetism for the interlayer spin configuration, and enhanced Curie temperature of the CrI3 monolayer by nearly two fold

Genomic Profiling of MicroRNAs and Proteomics Reveals an Early Molecular Alteration Associated with Tumorigenesis Induced by MC-LR in Mice

Studies have demonstrated that microcystins (MCs) can act as potential carcinogens and have caused serious risk to public environmental health. The molecular mechanisms of MC-induced susceptibility to carcinogenesis are largely unknown. In this study, we performed for the first time a comprehensive analysis of changes in microRNAs (miRNAs) and proteins expression in livers of mice treated with MC-LR. Utilizing microarray and two-dimensional gel electrophoresis (2-DE) analysis, we identified 37 miRNAs and 42 proteins significantly altered. Many aberrantly expressed miRNAs were related to various cancers (e.g., miR-125b, hepatocellular carcinoma; miR-21, leukemia; miR-16, chronic lymphocytic leukemia; miR-192, pituitary adenomas; miR-199a-3p, ovarian cancer; miR-34a, pancreatic cancer). Several miRNAs (e.g., miR-34a, miR-21) and proteins (e.g., TGM2, NDRG2) that play crucial roles in liver tumorigenesis were first found to be affected by MC-LR in mouse liver. MC-LR also altered the expression of a number of miRNAs and proteins involved in several pathways related to tumorigenesis, such as glutathione metabolism, VEGF signaling, and MAPK signaling pathway. Integration of post-transcriptomics, proteomics, and transcriptomics reveals that the networks miRNAs and their potential target genes and proteins involved in had a close association with carcinogenesis. These results provide an early molecular mechanism for liver tumorigenesis induced by MCs

Corpora statistics from the digital camera and mobile phone domains.

<p>Corpora statistics from the digital camera and mobile phone domains.</p

Comparison of the results of product aspect clustering when background knowledge is captured from the Web for both domains.

<p>Comparison of the results of product aspect clustering when background knowledge is captured from the Web for both domains.</p

Statistics of the corpus.

<p>Statistics of the corpus.</p

Comparisons of the results obtained using different similarity computation measures in the product aspect clustering task for the camera and mobile phone domains.

<p>Comparisons of the results obtained using different similarity computation measures in the product aspect clustering task for the camera and mobile phone domains.</p

Example of the three types of sentences.

<p>Example of the three types of sentences.</p

Comparison of the results of our method and two baselines.

<p>Comparison of the results of our method and two baselines.</p

Algorithm for the cascading rule-based approach (Baseline1).

<p>Algorithm for the cascading rule-based approach (Baseline1).</p

Additional file 1: Figure S1. of Isolation and characterization of chromosomal markers in Poa pratensis

PpCR-1 monomer hunting from the amplified sequence by designated primers of clone 6. Figure S2. PpTR-1 monomer hunting from the amplified sequence by designated primers of clone 1. Figure S3. PpTR-2 monomer hunting from the amplified sequence by designated primers of clone 23. Figure S4. PpTR-3 monomer hunting from the amplified sequence by designated primers of clone 94. (DOC 44 kb