74 research outputs found
Charge gap in the one--dimensional dimerized Hubbard model at quarter-filling
We propose a quantitative estimate of the charge gap that opens in the
one-dimensional dimerized Hubbard model at quarter-filling due to dimerization,
which makes the system effectively half--filled, and to repulsion, which
induces umklapp scattering processes. Our estimate is expected to be valid for
any value of the repulsion and of the parameter describing the dimerization. It
is based on analytical results obtained in various limits (weak coupling,
strong coupling, large dimerization) and on numerical results obtained by exact
diagonalization of small clusters. We consider two models of dimerization:
alternating hopping integrals and alternating on--site energies. The former
should be appropriate for the Bechgaard salts, the latter for compounds where
the stacks are made of alternating and molecules. % and ( denotes , , ...).Comment: 33 pages, RevTeX 3.0, figures on reques
Comparative proteomic analysis of the effects of high-concentrate diet on the hepatic metabolism and inflammatory response in lactating dairy goats
Intrathecal Injection of Spironolactone Attenuates Radicular Pain by Inhibition of Spinal Microglia Activation in a Rat Model
Microglia might play an important role in nociceptive processing and hyperalgesia by neuroinflammatory process. Mineralocorticoid receptor (MR) expressed on microglia might play a central role in the modulation of microglia activity. However the roles of microglia and MR in radicular pain were not well understood. This study sought to investigate whether selective MR antagonist spironolactone develop antinociceptive effects on radicular pain by inhibition neuroinflammation induced by spinal microglia activation.Radicular pain was produced by chronic compression of the dorsal root ganglia with SURGIFLO™. The expression of microglia, interleukin beta (IL-1β), interleukin 6 (IL-6), tumor necrosis factor alpha (TNF-α), NR1 subunit of the NMDA receptor (t-NR1), and NR1 subunit phosphorylated at Ser896 (p-NR1) were also markedly up-regulated. Intrathecal injection of spironolactone significantly attenuated pain behaviors as well as the expression of microglia, IL-1β, TNF-α, t-NR1, and p-NR1, whereas the production of IL-6 wasn't affected.These results suggest that intrathecal delivery spironolactone has therapeutic effects on radicular pain in rats. Decreasing the activation of glial cells, the production of proinflammatory cytokines and down-regulating the expression and phosphorylation of NMDA receptors in the spinal dorsal horn and dorsal root ganglia are the main mechanisms contributing to its beneficial effects
Aryl-1,3,5-triazine ligands of histamine H4 receptor attenuate inflammatory and nociceptive response to carrageen, zymosan and lipopolysaccharide
Inhibition of spontaneous development of hyperplastic alveolar nodules and mammary tumors in c3h mice fed phenylalanine-deficient diets.
Influence of dietary phenylalanine deficiency on the mammary tumor virus activity in c3h mice.
The developmental, noduligenic, and tumorigenic potentials of trans- planted mammary glands and primary ducts from c3h mice previously fed a phenylalanine-deficient diet.
Inhibition of transformation of mammary preneoplastic nodules to tumor in c3h mice fed a phenylalanine-deficient diet.
Inhibition of pituitary-induced nodular hyperplasia in mammary glands of c3h mice fed a phenylalanine-deficient diet.
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