学会抄録

<p><b>Observation of pulmonary artery sections</b> (200X, HE) The pulmonary artery wall thickness of disease (D) is noticeably increased. In the D sample, 1) the tunica adventicia was more compact and exhibited increased connective tissue; 2) the smooth muscle fiber was thicker; 3) there was excessive fiber production; and 4) the intima was more compact. The arrows indicate the pathological changes.</p

Basic summary of sequence and sequencing reads mapping to reference genome.

<p>Basic summary of sequence and sequencing reads mapping to reference genome.</p

Summary of AHI and blood routine in disease and normal groups.

<p>Summary of AHI and blood routine in disease and normal groups.</p

qRT-PCR and sequencing.

<p>qRT-PCR validation of differentially expressed genes in disease and normal samples of pulmonary artery for 12 genes.</p

KEGG enrichment analysis of DEGs in the pulmonary artery.

<p>The most 20 KEGG pathways were presented. The y-axis and x-axis indicate pathway name and rich factor, respectively. The size of circle dot means gene number.</p

Other putative significantly differential expressed genes regulating pulmonary artery remodeling and vascular contraction in this study.

<p>Other putative significantly differential expressed genes regulating pulmonary artery remodeling and vascular contraction in this study.</p

Putative significantly differential expressed molecules and genes enriched to Jak-STAT signaling pathway and NOD-like signaling pathway.

<p>Putative significantly differential expressed molecules and genes enriched to Jak-STAT signaling pathway and NOD-like signaling pathway.</p

GO enrichment analysis of DEGs in the pulmonary artery.

<p>The three main categories used for GO analysis. The y-axis and x-axis indicate subcategory name and gene number, respectively. * represents the significantly enriched GO terms.</p