Telomere length and risk of lung cancer: results for overall study and stratifying by years from enrollment to case diagnosis.

†<p>Telomere length categorized using tertiles in controls as cut-points.</p>*<p>N_Co indicates number of controls; and N_Ca, number of cases.</p>£<p>Odds ratios computed using conditional logistic regression adjusted for age and ever smoking.</p>‡<p>P trend calculated by using log transformed telomere length as continuous variable, adjusted for age and ever smoking.</p

Mean telomere length in association with rs2736100 (<i>CLPTM1L-TERT</i>), by case-control status in the Shanghai Women’s Health Study^*(All cases and controls).

*<p>log transformed telomere length as continuous variable was used.</p>‡<p>mean (SD).</p>†<p>P for trend calculated by using linear regression and rs2736100 by assigning the ordinal values 1, 2, and 3 for TT, GT, and GG respectively, adjusted for age and ever smoking.</p>£<p>P value from spearman correlation test.</p

Selected characteristic of lung cancer cases and individually matched controls selected from the Shanghai Women’s Health Study (recruited between 1997–2000).

$<p>Spearman correlation (r) with telomere length in controls is −0.41(P<0.0001).</p>£<p>P value of spearman r with telomere length in controls >0.05.</p>‡<p>Family history of lung cancer in first degree relatives.</p>*<p>NOS indicates not otherwise specified.</p>†<p>NA indicates not available.</p>††<p>NA indicates not available, as only never-smoking subjects were genotyped.</p