1,698 research outputs found
Quantifying immediate price impact of trades based on the -shell decomposition of stock trading networks
Traders in a stock market exchange stock shares and form a stock trading
network. Trades at different positions of the stock trading network may contain
different information. We construct stock trading networks based on the limit
order book data and classify traders into classes using the -shell
decomposition method. We investigate the influences of trading behaviors on the
price impact by comparing a closed national market (A-shares) with an
international market (B-shares), individuals and institutions, partially filled
and filled trades, buyer-initiated and seller-initiated trades, and trades at
different positions of a trading network. Institutional traders professionally
use some trading strategies to reduce the price impact and individuals at the
same positions in the trading network have a higher price impact than
institutions. We also find that trades in the core have higher price impacts
than those in the peripheral shell.Comment: 6 pages including 3 figures and 1 tabl
(E)-2-{3-[4-(DiphenylÂamino)styrÂyl]-5,5-dimethylÂcycloÂhex-2-enylÂidene}ÂmalonoÂnitrile
In the title compound, C31H27N3, the cycloÂhexene ring has an envelope configuration. In the crystal structure, there is an 34 Å3 void around the inversion center, but the low electron density (0.13 e Å−3) in the difference Fourier map suggests no solvent molÂecule occupying this void. No hydrogen bonding is found in the crystal structure
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Calcineurin B-Like Proteins CBL4 and CBL10 Mediate Two Independent Salt Tolerance Pathways in Arabidopsis.
In Arabidopsis, the salt overly sensitive (SOS) pathway, consisting of calcineurin B-like protein 4 (CBL4/SOS3), CBL-interacting protein kinase 24 (CIPK24/SOS2) and SOS1, has been well defined as a crucial mechanism to control cellular ion homoeostasis by extruding Na+ to the extracellular space, thus conferring salt tolerance in plants. CBL10 also plays a critical role in salt tolerance possibly by the activation of Na+ compartmentation into the vacuole. However, the functional relationship of the SOS and CBL10-regulated processes remains unclear. Here, we analyzed the genetic interaction between CBL4 and CBL10 and found that the cbl4 cbl10 double mutant was dramatically more sensitive to salt as compared to the cbl4 and cbl10 single mutants, suggesting that CBL4 and CBL10 each directs a different salt-tolerance pathway. Furthermore, the cbl4 cbl10 and cipk24 cbl10 double mutants were more sensitive than the cipk24 single mutant, suggesting that CBL10 directs a process involving CIPK24 and other partners different from the SOS pathway. Although the cbl4 cbl10, cipk24 cbl10, and sos1 cbl10 double mutants showed comparable salt-sensitive phenotype to sos1 at the whole plant level, they all accumulated much lower Na+ as compared to sos1 under high salt conditions, suggesting that CBL10 regulates additional unknown transport processes that play distinct roles from the SOS1 in Na+ homeostasis
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