Formation of Non-reciprocal Bands in Magnetized Diatomic Plasmonic Chains

We show that non-reciprocal bands can be formed in a magnetized periodic chain of spherical plasmonic particles with two particles per unit cell. Simplified form of symmetry operators in dipole approximations are used to demonstrate explicitly the relation between spectral non-reciprocity and broken spatial-temporal symmetries. Due to hybridization among plasmon modes and free photon modes, strong spectral non-reciprocity appears in region slightly below the lightline, where highly directed guiding of energy can be supported. The results may provide a clear guidance on the design of one-way waveguides

2,2′-Hexamethyl­enedi-1,3-benzothia­zole

The title compound, C20H20N2S2, was prepared by the reaction of suberic acid and 2-amino­thio­phenol under microwave irradiation. The mol­ecule lies on an inversion center

Membrane repair against H. pylori promotes cancer cell proliferation

Membrane repair is a universal response against physical and biological insults and enables cell survival. Helicobacter pylori is one of the most common human pathogens and the first formally recognized bacterial carcinogen associated with gastric cancer. However, little is known about host membrane repair in the context of H. pylori infection. Here we show that H. pylori disrupts the host plasma membrane and induces Ca2+ influx, which triggers the translocation of annexin family members A1 and A4 to the plasma membrane. This in turn activates a membrane repair response through the recruitment of lysosomal membranes and the induction of downstream signaling transduction pathways that promote cell survival and proliferation. Based on our data, we propose a new model by which H. pylori infection activates annexin A1 and A4 for membrane repair and how annexin A4 over-expression induced signaling promotes cell proliferation. Continual activation of this membrane repair response signaling cascade may cause abnormal cellular states leading to carcinogenesis. This study links H. pylori infection to membrane repair, providing insight into potential mechanisms of carcinogenesis resulting from membrane damage