728 research outputs found
Triangle singularity in the decays
We study the reaction and
find that the mechanism to produce this decay develops a triangle singularity
around ~MeV. The differential width
shows a rapid growth around the
invariant mass being 1515~MeV as a consequence of the triangle singularity of
this mechanism, which is directly tied to the nature of the and
as dynamically generated resonances from the interaction of
pseudoscalar mesons. The branching ratios obtained for the decays are of the order of , accessible in
present facilities, and we argue that their observation should provide relevant
information concerning the nature of the low-lying scalar mesons.Comment: 12 pages, 8 figures, published in EPJ
Revisiting the as a hadronic molecule and its strong decays
Recently, the Belle collaboration measured the ratios of the branching
fractions of the newly observed excited state. They did not
observe significant signals for the decay, and reported an upper limit for the ratio of the three
body decay to the two body decay mode of . In
this work, we revisit the newly observed from the molecular
perspective where this resonance appears to be a dynamically generated state
with spin-parity from the coupled channels interactions of the and in -wave and in -wave. With
the model parameters for the -wave interaction, we show that the ratio of
these decay fractions reported recently by the Belle collaboration can be
easily accommodated.Comment: Published version. Published in Eur.\ Phys.\ J.\ C {\bf 80}, 361
(2020
Hadronic molecular states with the quark contents , , and
We study the hadronic molecular states with the quark content
by investigating the interactions of the ,
, , , , , , and systems. By solving the
Bethe-Salpeter equation within the extended local hidden gauge formalism, we
find altogether six poles qualifying as possible hadronic molecular states: one
pole of below the - threshold, one pole of
below the - threshold, one pole of
below the - threshold, and three poles
of below the - threshold.
Their binding energies are calculated to be about 10-20 MeV with the cut-off
momentum . Similarly, we study the hadronic
molecular states with by investigating the interactions of
the , , ,
, , ,
, systems, and the states with
by investigating the interactions of the ,
, , , ,
, , systems. However, no
deeply-bound poles are found in these systems.Comment: 12 pages, 9 figures, revised version to be published in PR
A case of ‘fat-free’ pleomorphic lipoma occurring in the upper back and axilla simultaneously
Pleomorphic lipoma is a rare neoplasm that predominantly occurs in the dermis or subcutis of the posterior neck, upper back, and shoulders. Although pleomorphic lipoma is a benign tumor, it may contain atypical cells. As a variant of spindle cell lipoma, pleomorphic lipoma clinically presents as a slow-growing and well-circumscribed subcutaneous mass. Rarely, some patients have multiple lesions. Histologically, pleomorphic lipoma is composed of mature fat, bland spindle-shaped mesenchymal cells, and coarse ‘rope-like’ collagen bands. In addition, lipoma contains multinucleated floret-like giant cells. Although spindle cell lipoma/pleomorphic lipoma with little fat was seen in the original series described by Enzinger and Harvey, cases with little to no fat remain diagnostically challenging. Herein, we report a case of ‘fat-free’ pleomorphic lipoma occurring in the upper back and axilla simultaneously. Although the lipoma was typically composed of bland spindle-shaped cells, rope-like collagen, scattered floret-like giant cells, and striking stromal myxoid change in the background, mature fat was absent. Immunohistochemical analyses showed positive staining for CD34, vimentin, and Bcl-2, and negative staining for S100, confirming the diagnosis of pleomorphic lipoma
Impaired ubiquitin–proteasome system activity in the synapses of Huntington's disease mice
Huntington's disease (HD) is caused by the expansion of a polyglutamine tract in the N-terminal region of huntingtin (htt) and is characterized by selective neurodegeneration. In addition to forming nuclear aggregates, mutant htt accumulates in neuronal processes as well as synapses and affects synaptic function. However, the mechanism for the synaptic toxicity of mutant htt remains to be investigated. We targeted fluorescent reporters for the ubiquitin–proteasome system (UPS) to presynaptic or postsynaptic terminals of neurons. Using these reporters and biochemical assays of isolated synaptosomes, we found that mutant htt decreases synaptic UPS activity in cultured neurons and in HD mouse brains that express N-terminal or full-length mutant htt. Given that the UPS is a key regulator of synaptic plasticity and function, our findings offer insight into the selective neuronal dysfunction seen in HD and also establish a method to measure synaptic UPS activity in other neurological disease models
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