Redes digitais e processos colaborativos em dança: por uma ecologia do corpo, corpomídia

Este artigo é um recorte extraído da tese de doutorado “Deslocar para permanecer: implicações políticas das redes digitais nos processos criativos colaborativos”, defendida em 2016 na PUC - SP. O problema que instiga esta pesquisa é o paradoxo produzido pela promessa de democratização que as redes trouxeram, reconfigurando o próprio conceito de democracia, de participação e de compartilhamento, mas que não impediu a mitificação da horizontalidade de comunicação como sinônimo do ideal iluminista de liberdade, igualdade e fraternidade (KATZ, 2014). A pesquisa se apoia na Teoria Corpomídia (KATZ & GREINER) para investigar o papel do corpo como uma ecologia de possibilidades alternativas de existênci

Pharyngeal-cervical-brachial variant of Guillain-Barre syndrome.

The pharyngeal-cervical-brachial (PCB) variant of Guillain-Barré syndrome is defined by rapidly progressive oropharyngeal and cervicobrachial weakness associated with areflexia in the upper limbs. Serial nerve conduction studies suggest that PCB represents a localised subtype of Guillain-Barré syndrome characterised by axonal rather than demyelinating neuropathy. Many neurologists are unfamiliar with PCB, which is often misdiagnosed as brainstem stroke, myasthenia gravis or botulism. The presence of additional ophthalmoplegia and ataxia indicates overlap with Fisher syndrome. Half of patients with PCB carry IgG anti-GT1a antibodies which often cross-react with GQ1b, whereas most patients with Fisher syndrome carry IgG anti-GQ1b antibodies which always cross-react with GT1a. Significant overlap between the clinical and serological profiles of these patients supports the view that PCB and Fisher syndrome form a continuous spectrum. In this review, we highlight the clinical features of PCB and outline new diagnostic criteria

Atypical Bickerstaff brainstem encephalitis: pitfalls of diagnosis.

10.1016/j.pediatrneurol.2013.03.023Pediatric Neurology494e9-PNEU

Surgery itself does not trigger Guillain-Barre syndrome.

10.1111/ene.12065European Journal of Neurology203e40-e40EJNE

Mimics and chameleons in Guillain-Barre and Miller Fisher syndromes.

Guillain-Barré syndrome (GBS) and its variant, Miller Fisher syndrome (MFS) have several subtypes, together forming a continuous spectrum of discrete and overlapping syndromes. Such is the heterogeneity within this spectrum that many physicians may be surprised to learn that these disorders are related pathophysiologically, and therefore share certain clinical features. These include history of antecedent infection, monophasic disease course and symmetrical cranial or limb weakness. The presence of cerebrospinal fluid albuminocytological dissociation (raised protein, normal cell count), antiganglioside antibodies and neurophysiological evidence of axonal or demyelinating neuropathy also support a diagnosis in many cases, but should not be relied upon. Mimics of GBS and MFS can broadly be divided into those presenting with symmetrical limb weakness and those presenting with brainstem signs. MFS and the pharyngeal-cervical-brachial variant of GBS are frequently mistaken for brainstem stroke, botulism or myasthenia gravis, whereas Bickerstaff's brainstem encephalitis is often diagnosed as Wernicke's encephalopathy. Chameleons or atypical presentations of GBS-related disorders include: paraparetic GBS, bifacial weakness with paraesthesias, acute ataxic neuropathy, acute ophthalmoparesis, acute ptosis and acute mydriasis. Many neurologists may also not be aware that deep tendon reflexes remain present or may even appear brisk in up to 10% of patients with GBS. Correct diagnosis of GBS-related disorders helps to avoid unnecessary investigations and allows early immunotherapy if appropriate