615 research outputs found

    Additional application of the NASCAP code. Volume 1: NASCAP extension

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    The NASCAP computer program comprehensively analyzes problems of spacecraft charging. Using a fully three dimensional approach, it can accurately predict spacecraft potentials under a variety of conditions. Several changes were made to NASCAP, and a new code, NASCAP/LEO, was developed. In addition, detailed studies of several spacecraft-environmental interactions and of the SCATHA spacecraft were performed. The NASCAP/LEO program handles situations of relatively short Debye length encountered by large space structures or by any satellite in low earth orbit (LEO)

    Additional application of the NASCAP code. Volume 2: SEPS, ion thruster neutralization and electrostatic antenna model

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    The interactions of spacecraft systems with the surrounding plasma environment were studied analytically for three cases of current interest: calculating the impact of spacecraft generated plasmas on the main power system of a baseline solar electric propulsion stage (SEPS), modeling the physics of the neutralization of an ion thruster beam by a plasma bridge, and examining the physical and electrical effects of orbital ambient plasmas on the operation of an electrostatically controlled membrane mirror. In order to perform these studies, the NASA charging analyzer program (NASCAP) was used as well as several other computer models and analytical estimates. The main result of the SEPS study was to show how charge exchange ion expansion can create a conducting channel between the thrusters and the solar arrays. A fluid-like model was able to predict plasma potentials and temperatures measured near the main beam of an ion thruster and in the vicinity of a hollow cathode neutralizer. Power losses due to plasma currents were shown to be substantial for several proposed electrostatic antenna designs

    Mechanical Work and Physiological Responses to Simulated Flat Water Slalom Kayaking

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    The purpose of this study was to assess the physical work demand in relation to metrics of force and subsequent physiological response to a simulated flatwater slalom competition. Eight New Zealand team members completed a standard incremental step-test to ascertain power:oxygen consumption relationship. This was followed by a simulated race run where breath-by-breath analysis along with force and power data logged at 50 Hz to determine stroke length, impulse, peak force, time to peak force, and rate of peak force per stroke. Physiological response to negotiating a flatwater slalom course was greater than straight-line paddling (36.89 ± 2.01 vs. 32.17 ± 1.97 ml⋅kg-1⋅min-1, p = 0.0065) at the same power output. Mean power output for the duration of the simulated race (91.63 ± 7.19 s) was 203.8 ± 45.0 W, incurring an oxygen deficit of 1.386 ± 0.541 L⋅min-1 translating to an overall anaerobic contribution of 32 ± 18% and aerobic contribution of 68 ± 18%. Moderate to strong relationships between time duration and stroke peak force (R2 = 0.354, R2 = 0.485) and rate of peak force development (R2 = 0.345, R2 = 0.426) but not for stroke length (R2 = 0.022, R2 = 0.012), impulse (R2 = 0.088, R2 = 0.097) or time to peak force (R2 = 0.001, R2 = 0.0001) for left and right strokes, respectively. The number of propulsive (<0.6 s) strokes outweighed turning/driving (>0.6 s) strokes with a ratio of 94:6%. Longer stroke duration was significantly correlated to greater impulse (R2 = 0.507, p < 0.0001) and time to peak force (R2 = 0.851, p < 0.0001), but a lower rate of force development (R2 = 0.107, p < 0.0001). The results show that a flatwater slalom under simulated race conditions entails initial supra-maximal (anaerobic) work rate with a subsequent transition to one associated with maximal aerobic capacity. Inability to sustain work done and the subsequent decline in peak force and force profile per stroke requires further research regarding strategies to enhance performance

    Nonequilibrium dynamics of fully frustrated Ising models at T=0

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    We consider two fully frustrated Ising models: the antiferromagnetic triangular model in a field of strength, h=HTkBh=H T k_B, as well as the Villain model on the square lattice. After a quench from a disordered initial state to T=0 we study the nonequilibrium dynamics of both models by Monte Carlo simulations. In a finite system of linear size, LL, we define and measure sample dependent "first passage time", trt_r, which is the number of Monte Carlo steps until the energy is relaxed to the ground-state value. The distribution of trt_r, in particular its mean value, , is shown to obey the scaling relation, L2ln(L/L0) \sim L^2 \ln(L/L_0), for both models. Scaling of the autocorrelation function of the antiferromagnetic triangular model is shown to involve logarithmic corrections, both at H=0 and at the field-induced Kosterlitz-Thouless transition, however the autocorrelation exponent is found to be HH dependent.Comment: 7 pages, 8 figure

    The dose of hemodialysis and patient mortality

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    The dose of hemodialysis and patient mortality. The relationship between the delivered dose of hemodialysis and patient mortality remains somewhat controversial. Several observational studies have shown improved patient survival with higher levels of delivered dialysis dose. However, several other unmeasured variables, changes in patient mix or medical management may have impacted on this reported difference in mortality. The current study of a U.S. national sample of 2,311 patients from 347 dialysis units estimates the relationship of delivered hemodialysis dose to mortality, with a statistical adjustment for an extensive list of comorbidity/risk factors. Additionally this study investigated the existence of a dose beyond which more dialysis does not appear to lower mortality. We estimated patient survival using proportional hazards regression techniques, adjusting for 21 patient comorbidity/risk factors with stratification for nine Census regions. The patient sample was 2,311 Medicare hemodialysis patients treated with bicarbonate dialysate as of 12/31/90 who had end-stage renal disease for at least one year. Patient follow-up ranged between 1.5 and 2.4 years. The measurement of delivered therapy was based on two alternative measures of intradialytic urea reduction, the urea reduction ratio (URR) and Kt/V (with adjustment for urea generation and ultrafiltration). Hemodialysis patient mortality showed a strong and robust inverse correlation with delivered hemodialysis dose whether measured by Kt/V or by URR. Mortality risk was lower by 7% (P = 0.001) with each 0.1 higher level of delivered Kt/V. (Expressed in terms of URR, mortality was lower by 11% with each 5 percentage point higher URR; P = 0.001). Above a URR of 70% or a Kt/V of 1.3 these data did not provide statistical evidence of further reductions in mortality. In conclusion, the delivered dose of hemodialysis therapy is an important predictor of patient mortality. In a population of dialysis patients with a very high mortality rate, it appears that increasing the level of delivered therapy offers a practical and efficient means of lowering the mortality rate. The level of hemodialysis dose measured by URR or Kt/V beyond which the mortality rate does not continue to decrease, though not well defined with this study, appears to be above current levels of typical treatment of hemodialysis patients in the U.S
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