9 research outputs found
Histological analysis of myocardial infarction area.
<p>Infarcted area analysis showing no differences among groups. Data are expressed as mean ± SEM (n = 7). *P<0.05.</p
Interstitial collagen deposition evaluation in rat hearts.
<p>Representative images of histological sections stained with Picrosirius of Control (A), OVX+SHAM<sub>SED</sub> (B), OVX+SHAM<sub>ET</sub> (C), OVX+MI<sub>SED</sub> (D) and OVX+MI<sub>ET</sub> (E) groups. Data are expressed as mean ± SEM (n = 7). *P<0.05. Magnifier 400x. Bar: 200 µm.</p
Cardiac function measurements.
<p>(A) Left ventricular systolic pressure (LVSP) demonstrating no differences among groups. (B) Exercise training reduced left ventricular end diastolic pressure (LVEDP) in OVX+MI<sub>ET</sub> group compared to MI which occurs in parallel with an increased +dP/dt (C). There were a decrease in -dP/dt in the MI group compared with other groups which was not restored by ET (D). Data are expressed as mean ± SEM (n  =  12). *P<0.05.</p
Morphometric parameters eight weeks after myocardial infarction and exercise training in ovariectomized rats.
<p>Data are expressed as mean±SEM. BW, body weight; UW, uterine weight; RV, right ventricle; CS, Citrate Synthase.</p>#<p> <i>P</i><0.05 vs Control;</p>†<p> <i>P</i><0.05 vs OVX+SHAM<sub>ET</sub>;</p>‡<p> <i>P</i><0.05 vs OVX+SHAM<sub>SED</sub>;</p>$<p> <i>P</i><0.05 vs OVX+MI<sub>SED</sub>.</p><p>Morphometric parameters eight weeks after myocardial infarction and exercise training in ovariectomized rats.</p
Hemodynamic parameters eight weeks after myocardial infarction and exercise training in ovariectomized rats.
<p>Data are reposted as mean±SEM. HR, heart rate, SBP, systolic blood pressure, DBP, diastolic blood pressure and MAP, mean arterial pressure.</p>#<p> <i>P</i><0.05 vs Control;</p>†<p> <i>P</i><0.05 vs OVX+SHAM<sub>ET</sub>;</p>‡<p> <i>P</i><0.05 vs OVX+SHAM<sub>SED</sub>;</p>$<p> <i>P</i><0.05 vs OVX+MI<sub>SED</sub>.</p><p>Hemodynamic parameters eight weeks after myocardial infarction and exercise training in ovariectomized rats.</p
Analysis of oxidative stress in cardiac tissue.
<p>Analysis of superoxide formation in sections of cardiac tissue by the dihydroethidium fluorescence. Representative images of Control (A), OVX+SHAM<sub>SED</sub> (B), OVX+SHAM<sub>ET</sub> (C), OVX+MI<sub>SED</sub> (D) and OVX+MI<sub>ET</sub> (E) groups. Data are expressed as mean ± SEM (n = 4). *P<0.05. Bar: 200 µm.</p
Myocyte cross sectional area evaluation.
<p>Representative images of histological sections stained with hematoxylin and eosin of Control (A), OVX+SHAM<sub>SED</sub> (B), OVX+SHAM<sub>ET</sub> (C), OVX+MI<sub>SED</sub> (D) and OVX+MI<sub>ET</sub> (E) groups. Data are expressed as mean ± SEM (n = 7). *P<0.05. Magnifier 400x. Bar: 50 µm.</p
Histological analysis of myocardial infarction area.
<p>Infarcted area analysis showing no differences among groups. Data are expressed as mean ± SEM (n = 7). *P<0.05.</p
Oxidant and antioxidants proteins expression.
<p>Expression of AT1 receptor (A), Gp91phox (B) and the antioxidant enzymes SOD-2 (C) and catalase (D). The densities were normalized to the control protein GAPDH. Data are expressed as mean ± SEM (n = 5). *P<0.05.</p