11 research outputs found

    Renormalization of the Yang-Mills theory in the ambiguity-free gauge

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    The renormalization procedure for the Yang-Mills theory in the gauge free of the Gribov ambiguity is constructed. It is shown that all the ultraviolet infinities may be removed by renormalization of the parameters entering the classical Lagrangian and the local redefinition of the fields.Comment: 20 pages. Some explanations extended, one reference added. Final version published in the journa

    BRS "Symmetry", prehistory and history

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    Prehistory - Starting from 't Hooft's (1971) we have a short look at Taylor's and Slavnov's works (1971-72) and at the lectures given by Rouet and Stora in Lausanne-1973 which determine the transition from pre-history to history. History - We give a brief account of the main analyses and results of the BRS collaboration concerning the renormalized gauge theories, in particular the method of the regularization independent, algebraic renormalization, the algebraic proof of S-matrix unitarity and that of gauge choice independence of the renormalized physics. We conclude this report with a suggestion to the crucial question: what could remain of BRS invariance beyond perturbation theory.Comment: Talk given at: A Special day in honour of Raymond Stora, Annecy, July 8, 201

    Die Stoffwechselwirkungen der SchilddrĂĽsenhormone

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    The modulation of glucocorticoid receptor content by 3-O-methyl-D-glucose transport in human mononuclear leukocyte in obesity

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    Glucocorticoid receptors (GR) and 3-O-methyl-D glucose (3-O-MG) transport were determined in mononuclear leukocytes (MNL) from 11 abdominal obese subjects, 10 pituitary-dependent Cushing's syndrome (Cushing's disease) and 10 healthy controls. Using a whole-cell competitive binding assay and H-3-dexamethasone as tracer, MNL of abdominal obese subjects were found to have 4855+/-1389 sites/cell which was significantly lower (p0.05). These results indicated that, in abdominal obesity, the GR binding capacity in MNL is influenced by the changes in glucose transport. (J. Endocrinol. Invest. 21: 656-661, 1998) (C)1998, Editrice Kurtis
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