20 research outputs found

    Non-random topology of stock markets

    No full text
    We have analysed the cross correlations of daily fluctuations for [iopmath latex="N=6358N=6358"] N = 6358 [/iopmath] US stock prices during the year 1999. From those [iopmath latex="N(N−1)/2N(N-1)/2"] N(N-1)/2 [/iopmath] correlation coefficients, the minimum spanning tree (MST) has been built. We have investigated the topology exhibited by the MST. Even though the average coordination number of stocks is [iopmath latex="langlenrangleapprox2langle n rangleapprox 2"] n2 [/iopmath], the variance [iopmath latex="sigmasigma"] [/iopmath] of the topological distribution [iopmath latex="f(n)f(n)"] f(n) [/iopmath] diverges! More precisely, we have found that [iopmath latex="f(n)simn−2.2f(n) sim n^{-2.2}"] f(n)~n-2.2 [/iopmath] holds over two decades. We have studied the topological correlations for neighbouring nodes: an extremely broad set of local configurations exists, confirming the divergence of [iopmath latex="sigmasigma"] [/iopmath].

    Intercombination transition rates in Al-like Au

    No full text
    N=3, Δn=0\Delta n=0 intercombination transitions in Al-like ions of Au have been studied by time-resolved EUV spectroscopy of foil-excited ion beams. Wavelengths and lifetimes are compared to the available relativistic calculations. The theoretical data are found to be useful for guidance, but of clearly insufficient precision, in particular for the transition probabilities

    Experimental INvestigation of radiative decay rates of metastable levels in Eu II

    No full text
    The radiative lifetimes of the metastable a 9DJ (J = 2-5) levels in Eu II have been measured by laser probing of a stored ion beam. The lifetimes of all four J levels are slightly above 1 s. A deviation from the expected energy dependence of the decay rates for electric quadrupole transitions is observed and discussed

    The callipyge mutation enhances bidirectional long-range DLK1-GTL2 intergenic transcription in cis

    No full text
    The callipyge mutation (CLPG) is an A to G transition that affects a muscle-specific long-range control element located in the middle of the 90-kb DLK1-GTL2 intergenic (IG) region. It causes ectopic expression of a 327-kb cluster of imprinted genes in skeletal muscle, resulting in the callipyge muscular hypertrophy and its non-Mendelian inheritance pattern known as polar overdominance. We herein demonstrate that the CLPG mutation alters the muscular epigenotype of the DLK1-GTL2 IG region in cis, including hypomethylation, acquisition of novel DNase-I hypersentivite sites, and, most strikingly, strongly enhanced bidirectional, long-range IG transcription. The callipyge phenotype thus emerges as a unique model to study the functional significance of IG transcription, which recently has proven to be a widespread, yet elusive, feature of the mammalian genome
    corecore