6 research outputs found
MOESM4 of Granulin epithelin precursor promotes colorectal carcinogenesis by activating MARK/ERK pathway
Additional file 4. Univariable and multivariable Cox regression of prognostic parameters for overall survival in 190 patients with colorectal cancer
MOESM2 of Granulin epithelin precursor promotes colorectal carcinogenesis by activating MARK/ERK pathway
Additional file 2. Clinicopathological factors predicted prognosis for 190 CRC patients
MOESM1 of Granulin epithelin precursor promotes colorectal carcinogenesis by activating MARK/ERK pathway
Additional file 1. SuperArray SureSilencing shRNA plasmids mediated GEP knockdown
MOESM3 of Granulin epithelin precursor promotes colorectal carcinogenesis by activating MARK/ERK pathway
Additional file 3. Univariable and multivariable Cox regression of prognostic parameters for disease free survival in 190 patients with colorectal cancer
MOESM6 of Granulin epithelin precursor promotes colorectal carcinogenesis by activating MARK/ERK pathway
Additional file 6. Copy number change of GEP in ONCOMINE colon tissue datasets. (*P < 0.05; P < 0.01)
Additional file 2: of RASAL2 promotes tumor progression through LATS2/YAP1 axis of hippo signaling pathway in colorectal cancer
Figure S1. Hierarchical clustering of Illumina expression array in human CRC samples. Figure S2. RASAL2 expression of CRC in TCGA cohort. Figure S3. Western blot analysis for G1-S transition promoter CyclinD1 in cells with RASAL2 knockdown. Figure S4. shRASAL2 inhibited tumor growth in nude mice growth in vivo. Figure S5. RASAL2 RNA expression by siRNA knock down in CRCs. Figure S6. Kaplan Meier survival analysis of nuclear YAP1 expression in CRC patients. (PDF 551 kb