Gauge invariance, massless modes and topology of gauge fields in multi-band superconductors

Multi-phase physics is a new physics of multi-gap superconductors. Multi-band superconductors exhibit many interesting and novel properties. We investigate the dynamics of the phase-difference mode and show that this mode yields a new excitation mode. The phase-difference mode is represented as an abelian vector field. There are massless modes when the number of gaps is greater than three and the Josephson term is frustrated. The fluctuation of phase-difference modes with non-trivial topology leads to the existence of a fractional-quantum flux vortex in a magnetic field. A superconductor with a fractional-quantum flux vortex is regarded as a topological superconductor with the integer Chern number.Comment: Proceedings of the 12th Asia and Pacific Physics Conference (2013

Reply to "Comment on 'Isotope effect in multi-band and multi-channel attractive systems and inverse isotope effect in iron-based superconductors'"

The Comment insists on the following: in our model it is assumed that the effective interactions have specific energy ranges within the single band with a cutoff at \omega_1 for the phononic part and a range from \omega_1 to \omega_2 in the AF channel. Our reply is that we assume that V_i(k,k')\neq 0 if |\xi_k|<\omega_i and |\xi_{k'}|<\omega_i, and otherwise V_i(k,k')= 0 (i=1,2), as stated in our paper. This is the model of BCS type with two attractive interactions, and this assumption is the characteristic of the BCS approximation. The claim "the integration limits have been modified such that the AF channel mediated pairing sets in where the ph-channel pairing terminates and is limited at an energy given by \omega_j=\omega_{AF}" in the Comment is wrong. We describe the model and the method to solve the gap equation in more detail

Synthesis of ErFeAsO-based superconductors by hydrogen doping method

Here we demonstrate the technique to stabilize the ErFeAsO-based superconductor with the smallest lattice constants in LnFeAsO1-y (Ln = lanthanide) series using hydrogen doping method. Polycrystalline samples were synthesized by heating pellets with nominal compositions of ErFeAsO1-y (1-y = 0.75 ~ 0.95) sandwiched between pellets of LaFeAsO0.8H0.8 compositions at 1100 {\deg}C under a pressure of 5.0 - 5.5 GP. The sample with lattice constants of a = 3.8219 {\AA} and c = 8.2807 {\AA} shows the highest superconducting critical temperatures (Tc) of 44.5 K and 41.0 K determined by onset transitions of resistivity and susceptibility, respectively. We discuss phase diagram of Ln dependence of Tc in LnFeAsO-based superconductors.Comment: 12 Pages, 5 Figures, Accepted for publication in EP

Inverse Iron Isotope Effect on the transition temperature of the (Ba,K)Fe2As2 superconductor

We report that (Ba,K)Fe2As2 superconductor (a transition temperature, Tc = 38 K) shows inverse Iron isotope effect (-0.18) (the sample including the larger atomic weight of Fe depicts higher Tc). Measurements of both temperature dependent magnetization and resistivity reveal a clear inverse shift by systematic studies on Tc using three types of Fe-isotopes (Fe-54, natural Fe and Fe-57). This indicates the first evidence of the inverse isotope effect in high-Tc superconductors. This atomic mass dependence on Tc implies the exotic coupling mechanism.Comment: 12 pages, 6 figure

Cryostat to provide a solid deuterium layer in a plastic shell for the Gekko XII glass laser system

Copyright 1992 American Institute of Physics. This article may be downloaded for personal use only. Any other use requires prior permission of the author and the American Institute of Physics. The following article appeared in Review of Scientific Instruments, 63(6), 3378-3383, 1992 and may be found at http://dx.doi.org/10.1063/1.114255

CYP1B1 promotes tumorigenesis via altered expression of CDC20 and DAPK1 genes in renal cell carcinoma.

BackgroundCytochrome P450 1B1 (CYP1B1) has been shown to be up-regulated in many types of cancer including renal cell carcinoma (RCC). Several reports have shown that CYP1B1 can influence the regulation of tumor development; however, its role in RCC has not been well investigated. The aim of the present study was to determine the functional effects of CYP1B1 gene on tumorigenesis in RCC.MethodsExpression of CYP1B1 was determined in RCC cell lines, and tissue microarrays of 96 RCC and 25 normal tissues. To determine the biological significance of CYP1B1 in RCC progression, we silenced the gene in Caki-1 and 769-P cells by RNA interference and performed various functional analyses.ResultsFirst, we confirmed that CYP1B1 protein expression was significantly higher in RCC cell lines compared to normal kidney tissue. This trend was also observed in RCC samples (p &lt; 0.01). Interestingly, CYP1B1 expression was associated with tumor grade and stage. Next, we silenced the gene in Caki-1 and 769-P cells by RNA interference and performed various functional analyses to determine the biological significance of CYP1B1 in RCC progression. Inhibition of CYP1B1 expression resulted in decreased cell proliferation, migration and invasion of RCC cells. In addition, reduction of CYP1B1 induced cellular apoptosis in Caki-1. We also found that these anti-tumor effects on RCC cells caused by CYP1B1 depletion may be due to alteration of CDC20 and DAPK1 expression based on gene microarray and confirmed by real-time PCR. Interestingly, CYP1B1 expression was associated with CDC20 and DAPK1 expression in clinical samples.ConclusionsCYP1B1 may promote RCC development by inducing CDC20 expression and inhibiting apoptosis through the down-regulation of DAPK1. Our results demonstrate that CYP1B1 can be a potential tumor biomarker and a target for anticancer therapy in RCC