103 research outputs found

    Energy averages and fluctuations in the decay out of superdeformed bands

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    We derive analytic formulae for the energy average (including the energy average of the fluctuation contribution) and variance of the intraband decay intensity of a superdeformed band. Our results may be expressed in terms of three dimensionless variables: Γ↓/ΓS\Gamma^{\downarrow}/\Gamma_S, ΓN/d\Gamma_N/d, and ΓN/(ΓS+Γ↓)\Gamma_N/(\Gamma_S+\Gamma^{\downarrow}). Here Γ↓\Gamma^{\downarrow} is the spreading width for the mixing of a superdeformed (SD) state ∣0>|0> with the normally deformed (ND) states ∣Q>|Q> whose spin is the same as ∣0>|0>'s. The ∣Q>|Q> have mean level spacing dd and mean electromagnetic decay width ΓN\Gamma_N whilst ∣0>|0> has electromagnetic decay width ΓS\Gamma_S. The average decay intensity may be expressed solely in terms of the variables Γ↓/ΓS\Gamma^{\downarrow}/\Gamma_S and ΓN/d\Gamma_N/d or, analogously to statistical nuclear reaction theory, in terms of the transmission coefficients T0(E)T_0(E) and TNT_N describing transmission from the ∣Q>|Q> to the SD band via ∣0∠|0\angle and to lower ND states. The variance of the decay intensity, in analogy with Ericson's theory of cross section fluctuations depends on an additional variable, the correlation length \Gamma_N/(\Gamma_S+\Gamma^{\downarrow})=\frac{d}{2\pi}T_N/(\Gamma_S+\Gamma^{\d ownarrow}). This suggests that analysis of an experimentally obtained variance could yield the mean level spacing dd as does analysis of the cross section autocorrelation function in compound nuclear reactions. We compare our results with those of Gu and Weidenm\"uller.Comment: revtex4, 14 pages, 4 figures, to appear in Physical Review

    Zn-Neighbor Cu NQR in Zn-Substituted YBa2Cu3O7-d and YBa2Cu4O8

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    We studied local electronic states near Zn in optimally doped YBa2_2(Cu1−x_{1-x}Zn_x)3_3O7−δ_{7-\delta} and underdoped YBa2_2(Cu1−x_{1-x}Zn_x)4_4O8_8 via satellite signals of plane-site Cu(2) nuclear quadrupole resonance (NQR) spectra. From the relative intensity of Cu NQR spectra, the satellite signals are assigned to Zn-neighbor Cu NQR lines. The Cu nuclear spin-lattice relaxation time of the satellite signal is shorter than that of the main signal, which indicates that the magnetic correlation is locally enhanced near Zn both for the underdoped and the optimally doped systems. The pure YBa2_2Cu4_4O8_8 is a stoichiometric, homogenous, underdoped electronic system; nevertheless, the Zn-induced inhomogeneous magnetic response in the CuO2_2 plane is more marked than that of the optimally doped YBa2_2Cu3_3O7−δ_{7-\delta}.Comment: 9 pages including 8 figures, to be published in Phys. Rev.

    Time course of airway remodelling after an acute chlorine gas exposure in mice

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    Accidental chlorine (Cl2) gas inhalation is a common cause of acute airway injury. However, little is known about the kinetics of airway injury and repair after Cl2 exposure. We investigated the time course of airway epithelial damage and repair in mice after a single exposure to a high concentration of Cl2 gas. Mice were exposed to 800 ppm Cl2 gas for 5 minutes and studied from 12 hrs to 10 days post-exposure. The acute injury phase after Cl2 exposure (≤ 24 hrs post-exposure) was characterized by airway epithelial cell apoptosis (increased TUNEL staining) and sloughing, elevated protein in bronchoalveolar lavage fluid, and a modest increase in airway responses to methacholine. The repair phase after Cl2 exposure was characterized by increased airway epithelial cell proliferation, measured by immunoreactive proliferating cell nuclear antigen (PCNA), with maximal proliferation occurring 5 days after Cl2 exposure. At 10 days after Cl2 exposure the airway smooth muscle mass was increased relative to controls, suggestive of airway smooth muscle hyperplasia and there was evidence of airway fibrosis. No increase in goblet cells occurred at any time point. We conclude that a single exposure of mice to Cl2 gas causes acute changes in lung function, including pulmonary responsiveness to methacholine challenge, associated with airway damage, followed by subsequent repair and airway remodelling

    Benefit Claims of Functional Food

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    Regulation of Functional Foods and Ingredients

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