A numerical study of Richtmyer–Meshkov instability in continuously stratified fluids

Theory and calculations are presented for the evolution of Richtmyer–Meshkov instability in two-dimensional continuously stratified fluid layers. The initial acceleration and subsequent instability of the fluid layer are induced by means of an impulsive pressure distribution. The subsequent dynamics of the fluid layer are then calculated numerically using the incompressible equations of motion. Initial conditions representing single-scale perturbations and multiple-scale random perturbations are considered. It is found that the growth rates for Richtmyer–Meshkov instability of stratified fluid layers are substantially lower than those predicted by Richtmyer for a sharp fluid interface with an equivalent jump in density. A frozen field approximation for the early-time dynamics of the instability is proposed, and shown to approximate the initial behavior of the layer over a time equivalent to the traversal of several layer thicknesses. It is observed that the nonlinear development of the instability results in the formation of plumes of penetrating fluid. Late in the process, the initial momentum deposited by the impulse is primarily used in the internal mixing of the layer rather than in the overall growth of the stratified layer. At intermediate times, some evidence for the existence of scaling behavior in the width of the mixing layer of the instability is observed for the multiple-scale random perturbations, but not for the single-scale perturbations. The time variation of the layer thickness differs from the scaling derived using ideas of self-similarity due to Barenblatt [Non-Linear Dynamics and Turbulence, edited by G. I. Barenblatt, G. Ioos, and D. D. Joseph (Pitman, Boston, 1983), p. 48] even at low Atwood ratio, presumably because of the inhomogeneity and anisotropy due to the excitation of vortical plumes

Mixing and non-mixing local minima of the entropy contrast for blind source separation

In this paper, both non-mixing and mixing local minima of the entropy are analyzed from the viewpoint of blind source separation (BSS); they correspond respectively to acceptable and spurious solutions of the BSS problem. The contribution of this work is twofold. First, a Taylor development is used to show that the \textit{exact} output entropy cost function has a non-mixing minimum when this output is proportional to \textit{any} of the non-Gaussian sources, and not only when the output is proportional to the lowest entropic source. Second, in order to prove that mixing entropy minima exist when the source densities are strongly multimodal, an entropy approximator is proposed. The latter has the major advantage that an error bound can be provided. Even if this approximator (and the associated bound) is used here in the BSS context, it can be applied for estimating the entropy of any random variable with multimodal density.Comment: 11 pages, 6 figures, To appear in IEEE Transactions on Information Theor

Neural Relax

We present an algorithm for data preprocessing of an associative memory inspired to an electrostatic problem that turns out to have intimate relations with information maximization

Distinct subpopulations of enteric neuronal progenitors defined by time of development, sympathoadrenal lineage markers and Mash-1-dependence

Enteric and sympathetic neurons have previously been proposed to be lineally related. We present independent lines of evidence that suggest that enteric neurons arise from at least two lineages, only one of which expresses markers in common with sympathoadrenal cells. In the rat, sympathoadrenal markers are expressed, in the same order as in sympathetic neurons, by a subset of enteric neuronal precursors, which also transiently express tyrosine hydroxylase. If this precursor pool is eliminated in vitro by complement-mediated lysis, enteric neurons continue to develop; however, none of these are serotonergic. In the mouse, the Mash-1−/− mutation, which eliminates sympathetic neurons, also prevents the development of enteric serotonergic neurons. Other enteric neuronal populations, however, including those that contain calcitonin gene related peptide are present. Enteric tyrosine hydroxylase-containing cells co-express Mash-1 and are eliminated by the Mash-1−/− mutation, consistent with the idea that in the mouse, as in the rat, these precursors generate serotonergic neurons. Serotonergic neurons are generated early in development, while calcitonin gene related peptide-containing enteric neurons are generated much later. These data suggest that enteric neurons are derived from at least two progenitor lineages. One transiently expresses sympathoadrenal markers, is Mash-1-dependent, and generates early-born enteric neurons, some of which are serotonergic. The other is Mash-1-independent, does not express sympathoadrenal markers, and generates late-born enteric neurons, some of which contain calcitonin gene related peptide

The Distribution of Dengue Virus Serotype in Quang Nam Province (Vietnam) during the Outbreak in 2018

Objectives: Quang Nam province in the Centre of Vietnam has faced an outbreak of dengue hemorrhagic fever (DHF) in 2018. Although DHF is a recurrent disease in this area, no epidemiological and microbiological reports on dengue virus serotypes have been conducted mainly due to lack of facilities for such a kind of advanced surveillance. The aim of this study was to detect different dengue virus serotypes in patients’ blood samples. Design and Methods: Suspected cases living in Quang Nam province (Vietnam) and presenting clinical and hematological signs of dengue hemorrhagic fever were included in the study. The screening was performed, and the results were compared by using two methodologies: RT real-time PCR (RT-rPCR) and the Dengue NS1 rapid test. Results: From December 2018 to February 2019, looking both at RT-rPCR [+] and NS1 [+] methodologies, a total of 488 patients were screened and 336 were positive for dengue virus detection (74 children and 262 adults); 273 of these patients (81.3%) underwent viral serotype identification as follows: 12.82% (35/273) D1 serotype, 17.95% (49/273) D2, 0.37% (1/273) D3, 68.50 (187/283) D4, and 0.37% (1/273) D2+D4 serotypes. The RT-rPCR outcomes showed higher sensitivity during the first three days of infection compared to NS1 (92.3% vs. 89.7%). The NS1 increased sensitivity after the first 3 days whilst the RT-rPCR decreased. Conclusions: Advanced surveillance with dengue virus serotypes identification, if performed routinely, may help to predict and prevent further DHF epidemics based on the exposure of the different serotypes during different periods that lead to the intensification of disease severity as a consequence of antibody-dependent enhancement (ADE)

ηc\eta_c mixing effects on charmonium and BB meson decays

We include the $\eta_c$ meson into the $\eta$-$\eta'$-$G$ mixing formalism constructed in our previous work, where $G$ represents the pseudoscalar gluball. The mixing angles in this tetramixing matrix are constrained by theoretical and experimental implications from relevant hadronic processes. Especially, the angle between $\eta_c$ and $G$ is found to be about $11^\circ$ from the measured decay widths of the $\eta_c$ meson. The pseudoscalar glueball mass $m_G$, the pseudoscalar densities $m_{qq,ss,cc}$ and the U(1) anomaly matrix elements associated with the mixed states are solved from the anomalous Ward identities. The solution $m_G\approx 1.4$ GeV obtained from the $\eta$-$\eta'$-$G$ mixing is confirmed, while $m_{qq}$ grows to above the pion mass, and thus increases perturbative QCD predictions for the branching ratios $Br(B\to\eta'K)$. We then analyze the $\eta_c$-mixing effects on charmonium magnetic dipole transitions, and on the $B\to\eta^{(\prime)}K_S$ branching ratios and CP asymmetries, which further improve the consistency between theoretical predictions and data. A predominant observation is that the $\eta_c$ mixing enhances the perturbative QCD predictions for $Br(B\to\eta'K)$ by 18%, but does not alter those for $Br(B\to\eta K)$. The puzzle due to the large $Br(B\to\eta'K)$ data is then resolved.Comment: 12 pages, version to appear in PR

Topological invariants of plane curve singularities: Polar quotients and Lojasiewicz gradient exponents

In this paper, we study polar quotients and Łojasiewicz exponents of plane curve singularities, which are not necessarily reduced. We first show that, for complex plane curve singularities, the set of polar quotients is a topological invariant. We next prove that the Łojasiewicz gradient exponent can be computed in terms of the polar quotients, and so it is also a topological invariant. For real plane curve singularities, we also give a formula computing the Łojasiewicz gradient exponent via real polar branches. As an application, we give effective estimates of the Łojasiewicz exponents in the gradient and classical inequalities of polynomials in two (real or complex) variables