16 research outputs found

    Additional file 1: Table S1. of The prevalence and impact of childhood sexual abuse on HIV-risk behaviors among men who have sex with men (MSM) in India

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    Unweighted characteristics by childhood sexual abuse1 (CSA) among 11,788 men who have sex with men in 12 Indian cities. Table S2. Number of HIV risk behaviors and experiences by childhood sexual abuse1 (CSA). Table S3. The relationship between CSA and HIV-related risk score. Table S4. The relationship between CSA and recent and lifetime HIV-related risk behaviors and experiences1. (DOCX 37 kb

    Immune reconstitution inflammatory syndrome in association with HIV/AIDS and tuberculosis: Views over hidden possibilities-0

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    <p><b>Copyright information:</b></p><p>Taken from "Immune reconstitution inflammatory syndrome in association with HIV/AIDS and tuberculosis: Views over hidden possibilities"</p><p>http://www.aidsrestherapy.com/content/4/1/29</p><p>AIDS Research and Therapy 2007;4():29-29.</p><p>Published online 30 Nov 2007</p><p>PMCID:PMC2216023.</p><p></p>ion. Initiation of HAART in the subject leads to abrupt restoration of CD4+ T-cells and almost any pathogen-specific immune response. IRIS developers have a high burden of LPS and proinflammatory cytokines produced against LPS could result in an exaggerated, nonspecific attack on latent mycobacterial antigens that are presented in the local lymph nodes leading to localized inflammation. We also hypothesize that subjects that do not develop IRIS could have developed either tolerance (anergy) to persistent LPS and tubercle antigens or could have normal FOXP3+ gene (not shown) and that those with defective FOXP3+ gene or enormous plasma LPS could be vulnerable to IRIS (as demonstrated by researchers that defective FOXP3+ gene is associated with increased risk for inflammatory conditions). (Bold lines indicate the availability of clinical/experimental evidence and dashed lines indicate the possible mechanism)

    Results of multiple logistic regression of the association between seropositivity to Chikungunya/Dengue/Both with household and individual factors.

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    <p>*Income categories are described in <a href="http://www.plosntds.org/article/info:doi/10.1371/journal.pntd.0003906#pntd.0003906.t002" target="_blank">table 2</a></p><p>Results of the best fitting model (based on AIC) for each outcome are shown. All models included a random effect for location.</p

    Estimates of the force of infection (λ, summed across all serotypes) and R<sub>0</sub> obtained from catalytic models fit to dengue age-specific seroprevalence data.

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    <p>*We fit models with increasing number of parameters. Thus, while model A assumes that the force of infection has been constant historically, models B and C allow for periods with different transmission intensity.</p><p><sup>†</sup> p-value of a likelihood ratio test comparing model B vs. model A.</p><p><sup>γ</sup>p-value of a likelihood ratio test comparing model C vs. model B.</p><p>Estimates of the force of infection (λ, summed across all serotypes) and R<sub>0</sub> obtained from catalytic models fit to dengue age-specific seroprevalence data.</p
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