(Lattice) Propagators and Extraction of Spectral Densities

In this proceeding, we explain a few steps for an alternative extraction of the spectral density of a two-point function (propagator) based on a discrete set of data points. We present a so-called Tikhonov regularization of this particular inverse problem. We test it on 2 cases: lattice 0++} glueball data and mock gluon data.Comment: 8 pages, 4 figures. Proceedings of Xth Quark Confinement and the Hadron Spectrum, October 8-12, 2012, TUM Campus Garching, Munich, German

The lattice gluon propagator in renormalizable ξ\xi gauges

We study the SU(3) gluon propagator in renormalizable $R_\xi$ gauges implemented on a symmetric lattice with a total volume of (3.25 fm)$^4$ for values of the guage fixing parameter up to $\xi=0.5$. As expected, the longitudinal gluon dressing function stays constant at its tree-level value $\xi$. Similar to the Landau gauge, the transverse $R_\xi$ gauge gluon propagator saturates at a non-vanishing value in the deep infrared for all values of $\xi$ studied. We compare with very recent continuum studies and perform a simple analysis of the found saturation with a dynamically generated effective gluon mass.Comment: 6 pages, 4 figure

Analysis of the Disturbances Caused by Intraocular Forced Convection Mechanism Failure

In this chapter, we show the refractive error treatment result of a patient, the first author, who restarted in 2000, after a 4-year break, at the study start. According to previous publications, the treatment consists of rehydration and elimination of agglutinated, dehydrated and deposited metabolic residues in the cornea, the trabecular meshwork, the crystalline lens and the retina, as a consequence of the failure in the mechanism of intraocular mass transfer by forced convection. However, the forced movement of the metabolic mass to rehydrate one region can cause dehydration in another region. Therefore, the patient developed posterior and capsular cataract in their respective eyes, right and left. This dehydration, during the treatment, increases the difficulties for the success of the treatment. The first part is a chronological record of the most important components of the treatment. Then, the research method and the material used are discussed. The main symptoms and signs are analyzed and correlated with the failure of the mass transfer process and the accumulation of metabolic residues. The anatomy of binocular vision is analyzed as a part of the forced convection mechanism, and in conclusion, the report shows the main oculomotor functions, topographic mapping of corneas over an interval of 17 months

Finding the right answer: an information retrieval approach supporting knowledge sharing

Knowledge Management can be defined as the effective strategies to get the right piece of knowledge to the right person in the right time. Having the main purpose of providing users with information items of their interest, recommender systems seem to be quite valuable for organizational knowledge management environments. Here we present KARe (Knowledgeable Agent for Recommendations), a multiagent recommender system that supports users sharing knowledge in a peer-to-peer environment. Central to this work is the assumption that social interaction is essential for the creation and dissemination of new knowledge. Supporting social interaction, KARe allows users to share knowledge through questions and answers. This paper describes KARe�s agent-oriented architecture and presents its recommendation algorithm

Intraocular Forced Convection Mechanism Defect as Probable Cause of Normal-Tension Glaucoma

This paper describes several pathologies associated with pathological movements that can cause physical effort on the optic nerve and damage to vision. The accumulation of intraocular metabolic residues increases ocular globe mass and can change its position in the orbit, as well as increase the cornea and crystalline, accommodation resistance, in addition to being able to increase the aqueous humor output resistance. A series of discreet pathologies may result in optic nerve impairment: cyclotorsion and saccadic movement, position in the orbit, and increased intraocular pressure. The cyclotorsion movements can be stimulated by the superior visual field restriction, due to the metabolic residue accumulation in the light transmission regions of this visual field, preventing correct fusion of the images