33 research outputs found

    Ariel - Volume 2 Number 6

    Get PDF
    Editors Richard J. Bonanno Robin A. Edwards Associate Editors Steven Ager Stephen Flynn Shep Dickman Tom Williams Lay-out Editor Eugenia Miller Contributing Editors Michael J. Blecker W. Cherry Light James J. Nocon Lynne Porter Editors Emeritus Delvyn C. Case, Jr. Paul M. Fernhof

    Antibody Duration after infection From Sars-Cov-2 in the Texas Coronavirus antibody Response Survey

    Get PDF
    Understanding the duration of antibodies to the severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) virus that causes COVID-19 is important to controlling the current pandemic. Participants from the Texas Coronavirus Antibody Response Survey (Texas CARES) with at least 1 nucleocapsid protein antibody test were selected for a longitudinal analysis of antibody duration. A linear mixed model was fit to data from participants (n = 4553) with 1 to 3 antibody tests over 11 months (1 October 2020 to 16 September 2021), and models fit showed that expected antibody response after COVID-19 infection robustly increases for 100 days postinfection, and predicts individuals may remain antibody positive from natural infection beyond 500 days depending on age, body mass index, smoking or vaping use, and disease severity (hospitalized or not; symptomatic or not)

    Environmentalism, pre-environmentalism, and public policy

    Full text link
    In the last decade, thousands of new grassroots groups have formed to oppose environmental pollution on the basis that it endangers their health. These groups have revitalized the environmental movement and enlarged its membership well beyond the middle class. Scientists, however, have been unable to corroborate these groups' claims that exposure to pollutants has caused their diseases. For policy analysts this situation appears to pose a choice between democracy and science. It needn't. Instead of evaluating the grassroots groups from the perspective of science, it is possible to evaluate science from the perspective of environmentalism. This paper argues that environmental epidemiology reflects ‘pre-environmentalist’ assumptions about nature and that new ideas about nature advanced by the environmental movement could change the way scientists collect and interpret data.Peer Reviewedhttp://deepblue.lib.umich.edu/bitstream/2027.42/45449/1/11077_2005_Article_BF01006494.pd

    Differences in Inmate-Inmate and Inmate-Staff Altercations: Examples from a Medium Security Prison

    No full text
    Inmate violence is a major concern for correctional organizations. Most research on violence lump together inmate-inmate and inmate-staff violence and attempt to understand them from a single perspective. This article posits that inmate-inmate and inmate-staff violence are different phenomenon. Data from a medium security prison is used to understand the relationship between inmate-inmate and inmate-staff violence and other variables. Inmate-inmate altercations are related to structural and interpersonal variables. Inmate-staff altercations are related to the extent to which inmates are involved in social relationship with other inmates and see the correctional staff as a physical threat to them. variables. Inmate-staff altercations are related to the extent to which inmates are involved in social relationship with other inmates and see the correctional staff as a physical threat to them. variables. Inmate-staff altercations are related to the extent to which inmates are involved in social relationship with other inmates and see the correctional staff as a physical threat to them

    Lipoprotein cholesterol, apolipoprotein A-I and B and lipoprotein (a) abnormalities in men with premature coronary artery disease

    Get PDF
    AbstractThe prevalence of abnormalities of lipoprotein cholesterol and apolipoproteins A-I and B and lipoprotein (a) [Lp(a)] was determined in 321 men (mean age 50 ± 7 years) with angiographically documented coronary artery disease and compared with that in 901 control subjects from the Framingham Offspring Study (mean age 49 ± 6 years) who were clinically free of coronary artery disease. After correction for sampling in hospital, beta-adrenergic medication use and effects of diet, patients had significantly higher cholesterol levels (224 ± 53 vs. 214 ± 36 mg/dl), triglycerides (189 ± 95 vs. 141 +- 104 mg/dl), low density lipoprotein (LDL) cholesterol (156 ± 51 vs. 138 ± 33 mg/dl), apolipoprotein B (131 ± 37 vs. 108 ± 33 mg/dl) and Lp(a) levels (19.9 ± 19 vs. 14.9 ± 17.5 mg/dl). They also had significantly lower high density lipoprotein (HDL) cholesterol (36 ± 11 vs. 45 ± 12 mg/dl) and apolipoprotein A-I levels (114 ± 26 vs. 136 ± 32 mg/dl) (all p < 0.005).On the basis of Lipid Research Clinic 90th percentile values for triglycerides and LDL cholesterol and 10th percentile values for HDL cholesterol, the most frequent dyslipidemias were low HDL cholesterol alone (19.3% vs. 4.4%), elevated LDL cholesterol (12.1% vs. 9%), hypertriglyceridemia with low HDL cholesterol (9.7% vs. 4.2%), hypertriglyceridemia and elevated LDL cholesterol with low HDL cholesterol (3.4% vs. 0.2%) and Lp(a) excess (15.8% vs. 10%) in patients versus control subjects, respectively (p < 0.05). Stepwise discriminant analysis indicates that smoking, hypertension, decreased apolipoprotein A-I, increased apolipoprotein B, increased Lp(a) and diabetes are all significant (p < 0.05) factors in descending order of importance in distinguishing patients with coronary artery disease from normal control subjects.Not applying a correction for beta-adrenergic blocking agents, sampling bias and diet effects leads to a serious underestimation of the prevalence of LDL abnormalities and an overestimation of HDL abnormalities in patients with coronary artery disease. However, 35% of patients had a total cholesterol level <200 mg/dl after correction; of those patients, 73% had an HDL cholesterol level <35 mg/dl

    Settler-Colonialism, Memoricide and Indigenous Toponymic Memory: The Appropriation of Palestinian Place Names by the Israeli State

    Get PDF
    Cartography, place-naming and state-sponsored explorations were central to the modern European conquest of the earth, empire building and settler-colonisation projects. Scholars often assume that place names provide clues to the historical and cultural heritage of places and regions. This article uses social memory theory to analyse the cultural politics of place-naming in Israel. Drawing on Maurice Halbwachs’ study of the construction of social memory by the Latin Crusaders and Christian medieval pilgrims, the article shows Zionists’ toponymic strategies in Palestine, their superimposition of Biblical and Talmudic toponyms was designed to erase the indigenous Palestinian and Arabo-Islamic heritage of the land. In the pre-Nakba period Zionist toponymic schemes utilised nineteenth century Western explorations of Biblical ‘names’ and ‘places’ and appropriated Palestinian toponyms. Following the ethnic cleansing of Palestine in 1948, the Israeli state, now in control of 78 percent of the land, accelerated its toponymic project and pursued methods whose main features were memoricide and erasure. Continuing into the post-1967 occupation, these colonial methods threaten the destruction of the diverse historical cultural heritage of the land
    corecore