The generation of helical magnetic field in a viable scenario of Inflationary Magnetogenesis

We study the generation of helical magnetic fields in a model of inflationary magnetogenesis which is free from the strong coupling and back-reaction problems. To generate helical magnetic fields, we add an $f^2 \tilde{F}^{\mu\nu} F_{\mu\nu}$ term to the lagrangian of Ratra model. The strong coupling and back-reaction problems are avoided if we take a particular behaviour of coupling function $f$, in which $f$ increases during inflation and decreases post inflation to reheating. The generated magnetic field is fully helical and has a blue spectrum, $d\rho_B/d\ln k \propto k^4$. This spectrum is obtained when coupling function $f\propto a^2$ during inflation. The scale of reheating in our model has to be lower than $4000$ GeV to avoid back-reaction post inflation. The generated magnetic field spectrum satisfies the $\gamma$-ray bound for all the possible scales of reheating. The comoving magnetic field strength and its correlation length are $\sim 4 \times 10^{-11}$ G and $70$ kpc respectively, if reheating takes place at 100 GeV. For reheating at the QCD scales of $150$ MeV, the field strength increases to $\sim$ nano gauss, with coherence scale of $0.6$ Mpc.Comment: 11 pages, Submitted to PR

Radiating spherical collapse with heat flow

We present here a simple model of radiative gravitational collapse with radial heat flux which describes qualitatively the stages close to the formation of a superdense cold star. Starting with a static general solution for a cold star, the model can generate solutions for the earlier evolutionary stages. The temporal evolution of the model is specified by solving the junction conditions appropriate for radiating gravitational collapse.Comment: 13 pages, including 3 figures, submitted to IJMP-

Exosomes regulate neurogenesis and circuit assembly.

Exosomes are thought to be released by all cells in the body and to be involved in intercellular communication. We tested whether neural exosomes can regulate the development of neural circuits. We show that exosome treatment increases proliferation in developing neural cultures and in vivo in dentate gyrus of P4 mouse brain. We compared the protein cargo and signaling bioactivity of exosomes released by hiPSC-derived neural cultures lacking MECP2, a model of the neurodevelopmental disorder Rett syndrome, with exosomes released by isogenic rescue control neural cultures. Quantitative proteomic analysis indicates that control exosomes contain multiple functional signaling networks known to be important for neuronal circuit development. Treating MECP2-knockdown human primary neural cultures with control exosomes rescues deficits in neuronal proliferation, differentiation, synaptogenesis, and synchronized firing, whereas exosomes from MECP2-deficient hiPSC neural cultures lack this capability. These data indicate that exosomes carry signaling information required to regulate neural circuit development