8 research outputs found
Endocrine disruptor levels in specimens (mg/L).
<p>Endocrine disruptor levels in specimens (mg/L).</p
Frequency distribution of MEHP levels in cord blood and meconium.
<p> The frequency distribution of MEHP levels in cord blood and meconium, illustrating that MEHP levels in meconium were much higher than those in cord blood.</p
Fetal-maternal ratios of EDs.
<p>The ratio of MEHP, OP, 4-NP BDE17-154, BDE183-209 and ∑PBDEs levels in the fetal and maternal circulation, showing that their levels were approximately 20% lower in cord blood samples than those in maternal blood samples.</p
Regression analysis between non-POPs levels in maternal and cord blood.
<p> Regression analysis was used to analyze the relationship between these EDs levels in maternal and cord blood. (A) The relationship between MEHP levels in maternal and cord blood. The regression coefficient was 0.807, meaning that about 80% MEHP could cross the placenta; (B) The relationship between OP levels in maternal and cord blood. The regression coefficient was 0.846, meaning that about 85% OP could go through the placenta; (C) The relationship between 4-NP levels in maternal and cord blood. The regression coefficient was 0.862, meaning that more than 86% 4-NP could cross the placenta.</p
Correlation between MEHP levels in cord blood and in meconium.
<p>MEHP was detected in 135 of 201 mother-newborn pairs, presenting a good correlation in cord blood and meconium.</p
Non-POPs levels in meconium specimens.
<p>The concentrations of MEHP, OP and 4-NP in meconium specimens.</p
PBDE levels in maternal blood and cord blood specimens (ng/g lipid).
<p>GM = geometric mean.</p>*<p>p<0.05.</p
Non-POPs levels in all specimens.
<p>The concentrations of MEHP, OP and 4-NP in maternal blood, cord blood and meconium specimens. And their levels in maternal blood were significantly higher than those in cord blood (p<0.0001).</p