Taking Care of a Dancing Body: An investigation of how anatomical knowledge effects a dancer's risk of injury and performance quality

My Senior Distinction project, Taking Care of a Dancing Body, investigates how the body can be trained to extend the career of a contemporary dancer. This study’s goal is to develop a movement series that prepares a dancer’s body for athletic movement encountered in choreography. The purpose of the research deals with injury prevention, as well as investigating the possible secondary benefit of improved performance quality. My research method involves comparing the theories and findings of related topics from various fields such as Pilates, Yoga, strength training, and contemporary dance techniques. I am also working with a group of five dancers from the Department of Dance to help me construct the series and observe their progress as these ideas are discussed and applied. So far, I have been exploring the importance of anatomical symmetry of musculature versus meticulous stretching. Strength and flexibility are essential to dancers but unfortunately, stretching before exercise is not always sufficient in injury prevention. The human body tends to compensate for unevenness in the body by increasing tension in muscles and joints. This can eventually lead to injury. My research has been concentrated on the complex anatomy of the spinal joints and surrounding muscles. I hypothesize that lower back pain and injury can be prevented through the awareness and correction of asymmetry in the surrounding anatomy, the strengthening of the agonistic and antagonistic muscle groups, as well as maintaining and improving joints' range of motion. In addition, I believe the mere knowledge and awareness of the bodily structures and how they all systematically coordinate to allow integrated movement can increase physicality in a dancer. As experience is beginning to outweigh youth in the modern dance field, it is of the utmost importance that dancers take care of their bodies. Maintaining a healthy spine is essential to the success of a dancer’s ability to keep dancing through years of stress on the body. With the information gathered through my study, I hope to bring awareness to dancers about the possible steps that can be taken in order to ensure a lifelong career in this unique performing art-form.No embarg

Internalizing Symptoms in Autistic Young Adults: Comparing the Cognitive and Physiological Components of Emotion Regulation

Autistic people experience co-occurring mental illness at a substantially greater rate compared to their neurotypical counterparts. Of these comorbid psychopathologies, internalizing disorders (anxiety and depression) are among the most prevalent. Emotion dysregulation has been identified as a contributing factor to this phenomenon and potential treatment target. The current study employed cognitive (e.g., use of suppression and use of cognitive reappraisal) and physiological (e.g., respiratory sinus arrhythmia (RSA) and heart period) measures to more holistically capture the multifaceted construct of emotion regulation compared to prior research. In a sample of autistic young adults (N = 63) ages 17-29 (M = 20.14), backward hierarchical regression revealed that cognitive reappraisal significantly predicted symptoms of both anxiety and depression whereas suppression significantly predicted symptoms of only anxiety. The physiological variables were not retained as significant predictors in either model. In sum, the present multi-method study underscores the cognitive component of emotion dysregulation in autistic young adults, particularly as it relates to internalizing symptomatology. These findings have clinical implications for the treatment of anxiety and depression in autistic adult populations. Possible explanations for the null physiological findings and future directions are discussed

Developing the next generation of diverse computer scientists: the need for enhanced, intersectional computing identity theory

This theoretical paper explores the need for enhanced, intersectional computing identity theory for the purpose of developing a diverse group of computer scientists for the future. Greater theoretical understanding of the identity formation process specifically for computing is needed in order to understand how students come to understand themselves as computer scientists. To ensure that the next generation of computer scientists is diverse, this paper presents a case for examining identity development intersectionally, understanding the ways in which women and underrepresented students may have difficulty identifying as computer scientists and be systematically oppressed in their pursuit of computer science careers. Through a review of the available scholarship, this paper suggests that creating greater theoretical understanding of the computing identity development process will inform the way in which educational stakeholders consider computer science practices and policies

Feasibility of an Opioid Sparing Discharge Protocol Following Laparoscopic Bariatric Surgery

BACKGROUND: Opioids are commonly prescribed after laparoscopic bariatric surgery but have untoward effects including dependence and diversion. Prior investigation revealed that over three-fourths of discharge opioids prescribed to our patients went unused. OBJECTIVES: To determine the feasibility of an opioid sparing discharge protocol following laparoscopic bariatric surgery. METHODS: A total of 212 opioid-naïve patients undergoing laparoscopic bariatric surgery were examined and divided into two groups; 106 prior to (Cohort A) and 106 after implementation of an opioid sparing discharge protocol (Cohort B). Opioids were converted to morphine milligram equivalents (MME) and post-operative consumption was examined. Data was described as mean ± standard deviation. RESULTS: No patients in Cohort B and 54.7% (58) in Cohort A received an opioid discharge prescription (37.5 MME). Of the 154 patients that remained, only 1.3% (2) received one after discharge. Cohort A took greater amounts of opioids than Cohort B after discharge (4.74 ± 11 vs. 0.21 ± 2 MME; p \u3c 0.001). During hospitalization, Cohort A took greater amounts of opioids (6.92 ± 11 vs. 2.74 ± 5 MME; p \u3c 0.001) but lower amounts of methocarbamol (759 ± 590 vs. 966 ± 585 mg; p = 0.011). No patient requested an opioid prescription refill or presented to the emergency room secondary to pain. CONCLUSION: Following laparoscopic bariatric surgery, an opioid sparing discharge protocol is feasible with \u3c 2% of patients receiving opioids after discharge and no increase in emergency room visits. Education regarding these protocols may impact the amount of opioids taken during hospitalization

Neurons of origin and fiber trajectory of amygdalofugal projections to the medial preoptic area in syrian hamsters

The amygdaloid neurons of origin and the trajectory of amygdaloid fibers to the medial preoptic area of the adult male Syrian hamster were identified by using horseradish peroxidase (HRP) histochemistry. After iontophoresis of HRP into the medial preoptic area, retrogradely labeled amygdaloid neurons were located in the dorsal and caudal parts of the medial amygdaloid nucleus and throughout the amygdalohippocampal area. No amygdaloid neurons were labeled after HRP applications confined to the most rostral portion of the medial preoptic area (anterior to the body of the anterior commissure). Following more caudal medial preoptic area injections (body of the anterior commissure to the suprachiasmatic nucleus) the distribution of retrogradely labeled cells in the medial amygdaloid nucleus and the amygdalohippocampal area revealed no topographic organization of the amygdalopreoptic connections. When amygdaloid neurons were labeled, the amygdalohippocampal area contained two to five times as many HRP-filled cells as the medial amygdaloid nucleus. Retrogradely transported HRP could be followed from the medial preoptic area to the amygdala through fibers in the dorsomedial quadrant of the stria terminalis. In addition, electrolytic lesions of the stria terminalis prior to iontophoresis of HRP into the medial preoptic area prevented retrograde transport to neurons in both the dorsocaudal medial amygdaloid nucleus and the amygdalohippocampal area. These results confirm earlier observations describing the location of autoradiographically labeled efferents from the medial amygdaloid nucleus to the medial preoptic area and provide new information about the restricted region within the medial amygdaloid nucleus from which these projections arise. They also suggest that, unlike the projections from the medial amygdaloid nucleus to the bed nucleus of the stria terminalis, the efferents to the medial preoptic area travel entirely in the stria terminalis.Peer Reviewedhttp://deepblue.lib.umich.edu/bitstream/2027.42/50043/1/902800106_ftp.pd

Vomeronasal and olfactory pathways to the amygdala controlling male hamster sexual behavior: Autoradiographic and behavioral analyses

Previous studies suggest that the rostral corticomedial amygdala (CMA), particularly the medial nucleus, is an important site where vomeronasal and olfactory stimuli critical to male hamster copulatory behavior are processed. To test the possibility that mating deficits seen after lesions of the rostrally-placed medial nucleus may be due to the interruption of chemosensory afferents to more caudal areas, we injected tritiated amino acids into the accessory and main olfactory bulbs of male hamsters in which we had first produced bilateral electrolytic lesions or sham lesions in either the rostral CMA or basolateral amygdala, and then observed mating behavior. Autoradiographic analysis of `vomeronasal' projections from the accessory olfactory bulb and `olfactory' projections from the main bulb, revealed that rostral CMA lesions which damaged the medial nucleus and extended to the ventral surface of the brain (ventral lesions) interrupted vomeronasal input to the more caudally-placed posteromedial cortical nucleus, but spared olfactory inputs to adjacent caidal areas of the amygdala and piriform lobe. In contrast, lesions which damaged a major portion of the medial nucleus but left its ventral surface intact (dorsal lesions) spared both vomeronasal and olfactory inputs to more caudal areas. Animals with both dorsal and ventral lesions failed to mate posteperatively, whereas animals bearing sham lesions of basolateral amygdaloid lesions, which, like dorsal lesions, spared caudally-directed chemosensory afferents, continued to mate normally. We conclude that mating deficits seen after rostral CMA lesions are due primarily to destruction of the medial nucleus.Peer Reviewedhttp://deepblue.lib.umich.edu/bitstream/2027.42/23980/1/0000229.pd

Evidence for a ventral non-strial pathway from the amygdala to the bed nucleus of the stria terminalis in the male golden hamster

Male hamsters in which the stria terminalis (ST) had been interrupted either by electrolytic lesions or knife cuts, or normal control males, received iontophoretic injections of horseradish peroxidase in either the bed nucleus of the stria terminalis (BNST) or the medial preoptic-anterior hypothalamic area (MPOAH). Comparison of intact and St-lesioned brains revealed the existence of a ventral non-striatal pathway, from cells in the medial amygdaloid nucleus (M) to the preoptic portion of the BNST but not to the MPOAH. Since bilateral lesions of M completely eliminate male hamster mating behavior, but ST lesions do not, we suggest that the ventral pathway to the BNST may be an important route by which M influences male copulatory behavior.Peer Reviewedhttp://deepblue.lib.umich.edu/bitstream/2027.42/25215/1/0000655.pd

Effect of Fetal Striatal and Astrocyte Transplants into Unilateral Excitotoxin-Lesioned Striatum

Studies have suggested that neurotrophic mechanisms may underlie transplant-induced functional recovery. Astrocytes have been reported to be a source of neurotrophic factors. The present study examined the possible role of cultured astrocytes in promoting recovery of apomorphine-induced rotation behavior in rats with unilateral kainic acid (KA) lesions of the striatum. Five weeks after the lesions, one group of rats received fetal striatal tissue (E17) transplants, another group received transplants of cultured astrocyte suspension, and the remaining rats received sham transplants and served as controls. Apomorphine-induced rotation behavior was tested 4 weeks after the KA lesions, and 5 and 10 weeks following the transplantation. The KA-induced rotation behavior was reduced by the striatal transplants but not by the cultured astrocyte transplants 5 and 10 weeks following the transplantation. Histochemicai analysis indicated that the striatal transplants had survived and grown and contained neurons and glia with similar morphology to those in the host brain. Immunocytochemical analysis of the astrocyte transplant sites revealed heavy glial fibrillary acidic protein and OX-42 staining in the transplant areas, suggesting that the transplanted astrocytes may have survived in the host brain. Although fetal striatal transplants can ameliorate apomorphine-induced rotation behavior, transplants of astrocytes alone may not be sufficient to reverse the functional deficits produced by KA lesions

Increase in West Nile Neuroinvasive Disease after Hurricane Katrina

After Hurricane Katrina, the number of reported cases of West Nile neuroinvasive disease (WNND) sharply increased in the hurricane-affected regions of Louisiana and Mississippi. In 2006, a >2-fold increase in WNND incidence was observed in the hurricane-affected areas than in previous years