Cost-effectiveness of Insulin Degludec versus Insulin Glargine U100 in Patients with Type 1 and Type 2 Diabetes Mellitus in Serbia

<p></p><p><b>Article full text</b></p> <p><br></p> <p>The full text of this article can be found here<b>. </b><a href="https://link.springer.com/article/10.1007/s13300-018-0426-0">https://link.springer.com/article/10.1007/s13300-018-0426-0</a></p><p></p><p></p><p> </p><p><br></p> <p><b>Provide enhanced content for this article</b></p> <p><br></p> <p>If you are an author of this publication and would like to provide additional enhanced content for your article then please contact <a href="http://www.medengine.com/Redeem/”mailto:[email protected]”"><b>[email protected]</b></a>.</p> <p><br></p> <p>The journal offers a range of additional features designed to increase visibility and readership. All features will be thoroughly peer reviewed to ensure the content is of the highest scientific standard and all features are marked as ‘peer reviewed’ to ensure readers are aware that the content has been reviewed to the same level as the articles they are being presented alongside. Moreover, all sponsorship and disclosure information is included to provide complete transparency and adherence to good publication practices. This ensures that however the content is reached the reader has a full understanding of its origin. No fees are charged for hosting additional open access content.</p> <p><br></p> <p>Other enhanced features include, but are not limited to:</p> <p><br></p> <p>• Slide decks</p> <p>• Videos and animations</p> <p>• Audio abstracts</p> <p>• Audio slides</p><br><p></p

Obesity and Diabetes: Pathophysiology of Obesity-Induced Hyperglycemia and Insulin Resistance

Insulin resistance along with type 2 diabetes (T2D) is some of the complications of obesity. Insulin resistance occurs due to the increased discharge of fatty acids, lipids, and other advancing factors, by the adipose tissue, leading to a myriad of complications. Several other substances namely glycerol, enzymes, inflammatory factors, and hormones are also involved in the overall mechanism. At the point when resistance towards insulin occurs, along with abnormally functioning β-cells, the overall burden of the inability to control elevated glucose levels in the blood ensues. Anomalies in β-cell are thus more critical in the development of T2D and its related symptoms. This information is encouraging investigation of the molecular and hereditary basis of the ailment and new ways to deal with its management and avoidance