The trend of hr-HPV types and HIV infection by age-group.

<p>The trend of hr-HPV types and HIV infection by age-group.</p

Risk factors associated with hr-HPV infection (N = 644).

<p>Risk factors associated with hr-HPV infection (N = 644).</p

Overall hr-HPV, HIV and hr-HPV/HIV co-infection prevalence by age groups and study site (Crude and weighted) (N = 644).

<p>Overall hr-HPV, HIV and hr-HPV/HIV co-infection prevalence by age groups and study site (Crude and weighted) (N = 644).</p

Age-specific prevalence of HPV and HIV among reproductive age women in Swaziland.

<p>Age-specific prevalence of HPV and HIV among reproductive age women in Swaziland.</p

Map showing the location, political regions of Swaziland and health facilities (study sites with a red star).

<p><b>Source:</b> Macmillan. Geography and map of Swaziland Matsapha2005 [accessed 2012 13]. Available from: <a href="http://geography.about.com/library/blcswaziland.htm" target="_blank">http://geography.about.com/library/blcswaziland.htm</a>.</p

The weighted prevalence and estimated population burden of hr-HPV infection among sexual reproductive women (15–49 years) in Swaziland (n = 644).

<p>The weighted prevalence and estimated population burden of hr-HPV infection among sexual reproductive women (15–49 years) in Swaziland (n = 644).</p

<i>CHRN</i> variants and propensity to smoke or chew tobacco.

?<p>derived from linear regression of log transformed number of cigarettes smoked per day as outcome, respective <i>CHRN</i> variant as explanatory.</p>*<p>derived from linear regression of log transformed number of chewing events per day as outcome, respective <i>CHRN</i> variant as explanatory.</p>$<p>Base model included age, sex, center and case-control status.</p><p>significant p- values are indicated in bold.</p

<i>CHRN</i> variants and risk of oral cancer.

<p>Panel A shows the association between rs16969968 and oral cancer; Panel B shows the association between rs578776 and oral cancer. Forest plot represents odds ratios derived from the log additive multivariate model adjusted for age, sex, level of education and center, as appropriate.</p

Association between <i>ADH</i> & <i>ALDH2</i> variants and risk of oral cancer.

*<p>Odds ratios were derived from log additive model adjusted for age, sex, tobacco exposures and center.</p><p>P indicates p-value for heterogeneity.</p

HPV39 variant tree topology and pairwise comparisons of individual complete genomes.

<p>The phylogenetic tree was constructed as described in <a href="http://www.plosone.org/article/info:doi/10.1371/journal.pone.0072565#pone-0072565-g002" target="_blank">Figure 2</a>. Distinct variant lineages and sublineages were determined as described in <a href="http://www.plosone.org/article/info:doi/10.1371/journal.pone.0072565#pone-0072565-g002" target="_blank">Figure 2</a>. The percent nucleotide sequence differences are shown in the panel to the right of the phylogenetic tree as described in <a href="http://www.plosone.org/article/info:doi/10.1371/journal.pone.0072565#pone-0072565-g002" target="_blank">Figure 2</a>.</p