9 research outputs found

    Molecular and phenotypic characterization of <i>S. hominis</i> blood isolates.

    No full text
    1<p>Neg: biofilm non-producer; Glu: media with 1% glucose; NaCl: media with 3% NaCl.</p>2<p>FOX: cefoxitin test; R: resistant; S: susceptible.</p>3<p>Pos: <i>mecA</i> gene present, Neg: <i>mecA</i> absent.</p>4<p>Neg: not amplified.</p>5<p>SCCmec type III was assigned for <i>S. aureus</i> according to <a href="http://www.sccmec.org/Pages/SCC_TypesEN.html" target="_blank">http://www.sccmec.org/Pages/SCC_TypesEN.html</a>.</p><p>Neg: not amplified, UT: untyped; UT1, UT2 and UT3 types are assigned in this study.</p>6<p>PEN: penicillin; AMP: ampicillin; AUG: amoxicillin-clavulanic acid; ERY: erythromycin; TMP: trimethoprim; CIP: ciprofloxacin; TET: tetracycline; CHL: chloramphenicol; GEN: gentamicin; RIF: rifampin; NIT: nitrofurantoin; CTX: cefotaxime; DAP: daptomycin.</p>*<p>Non-susceptible.</p><p>All isolates were susceptible to vancomycin and linezolid.</p

    Findings of patients seeking care for acute respiratory infections at the Hospital Civil de Guadalajara during the (H1N1) pandemic 2009—Mexico <sub>◊</sub>.

    No full text
    <p>¥p = 0.01 when comparing patients who tested positive for seasonal influenza A with those who tested positive for pandemic (H1N1) 2009.</p><p>*p≤0.009 when comparing patients who tested positive for pandemic (H1N1) 2009 to those who tested negative.</p><p>‡Includes all hospitalized cases regardless of influenza RT-PCR test results.</p><p>∞Includes all hospitalized cases and ambulatory patients treated with oseltamivir who tested positive for influenza A.</p><p>◊Insufficient data available from patients discharged from triage without oseltamivir to include in table.</p
    corecore