4 research outputs found

    Number of patients randomly assigned to the antibiotic group with different concentrations of microorganisms in the endotracheal aspirate at different time points

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    Five patients had polymicrobial ventilator-associated tracheobronchitis (VAT).<p><b>Copyright information:</b></p><p>Taken from "Antimicrobial treatment for ventilator-associated tracheobronchitis: a randomized, controlled, multicenter study"</p><p>http://ccforum.com/content/12/3/R62</p><p>Critical Care 2008;12(3):R62-R62.</p><p>Published online 2 May 2008</p><p>PMCID:PMC2481443.</p><p></p

    Kaplan-Meier survival curves for patients randomly assigned to the antibiotic and control groups

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    The dashed line represents the cumulative survival for patients randomly assigned to the antibiotic group, the solid line represents the cumulative survival for patients randomly assigned to the no antibiotic group, and + represents censored patients. = 0.047 by the log rank test. ICU, intensive care unit.<p><b>Copyright information:</b></p><p>Taken from "Antimicrobial treatment for ventilator-associated tracheobronchitis: a randomized, controlled, multicenter study"</p><p>http://ccforum.com/content/12/3/R62</p><p>Critical Care 2008;12(3):R62-R62.</p><p>Published online 2 May 2008</p><p>PMCID:PMC2481443.</p><p></p

    MOESM2 of Respiratory changes of the inferior vena cava diameter predict fluid responsiveness in spontaneously breathing patients with cardiac arrhythmias

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    Additional file 2: Table S1. Respiratory variables in responders and nonresponders before and after volume expansion. Table S2. Volume expansion-induced changes in hemodynamic variables in responders and nonresponders. Table S3. Baseline characteristics of the patients (VE-related change in VTIao ≥ 15% to define responders). Table S4. Hemodynamic variables before and after volume expansion in responders and nonresponders (VE-related change in VTIao ≥ 15% to define responders). Table S5. Accuracy of the inferior vena cava variables for predicting response to volume expansion (VE-related change in VTIao ≥ 15% to define responders)

    MOESM1 of Respiratory changes of the inferior vena cava diameter predict fluid responsiveness in spontaneously breathing patients with cardiac arrhythmias

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    Additional file 1: Figure S1. A, Receiver operating characteristics (ROC) curve of the collapsibility index (cIVC-st) and the inspiratory diameter (iIVC-st) of the inferior vena cava during a standardized inspiratory maneuver before volume expansion (VE) to discriminate responders from nonresponders to VE in the overall population. B, ROC curve of the collapsibility index (cIVC-sp) and the inspiratory diameter (iIVC-sp) of the inferior vena cava during unstandardized spontaneous breathing before VE to discriminate responders from nonresponders to VE in the overall population. Figure S2. A, Linear correlation between the collapsibility index of the inferior vena cava under standardized breathing (cIVC-st) before volume expansion (VE) and VE-induced change in the velocity time integral of aortic blood flow (VTIao). B, Linear correlation between the inspiratory diameter of the inferior vena cava under standardized breathing (iIVC-st) before VE and VE-induced change in VTIao. Figure S3. Scatterplot of individual values before volume expansion (VE) for the collapsibility index (cIVC-sp), minimum-inspiratory diameter (iIVC-sp), and the end-expiratory diameter of the inferior vena cava (eIVC-sp) under unstandardized spontaneous breathing in responders and nonresponders to VE
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