23 research outputs found
Visual Rationalizations in Deep Reinforcement Learning for Atari Games
Due to the capability of deep learning to perform well in high dimensional
problems, deep reinforcement learning agents perform well in challenging tasks
such as Atari 2600 games. However, clearly explaining why a certain action is
taken by the agent can be as important as the decision itself. Deep
reinforcement learning models, as other deep learning models, tend to be opaque
in their decision-making process. In this work, we propose to make deep
reinforcement learning more transparent by visualizing the evidence on which
the agent bases its decision. In this work, we emphasize the importance of
producing a justification for an observed action, which could be applied to a
black-box decision agent.Comment: presented as oral talk at BNAIC 201
How to successfully select and implement electronic health records (EHR) in small ambulatory practice settings
Sickness behaviour pushed too far – the basis of the syndrome seen in severe protozoal, bacterial and viral diseases and post-trauma
Certain distinctive components of the severe systemic inflammatory syndrome are now well-recognized to be common to malaria, sepsis, viral infections, and post-trauma illness. While their connection with cytokines has been appreciated for some time, the constellation of changes that comprise the syndrome has simply been accepted as an empirical observation, with no theory to explain why they should coexist. New data on the effects of the main pro-inflammatory cytokines on the genetic control of sickness behaviour can be extended to provide a rationale for why this syndrome contains many of its accustomed components, such as reversible encephalopathy, gene silencing, dyserythropoiesis, seizures, coagulopathy, hypoalbuminaemia and hypertriglyceridaemia. It is thus proposed that the pattern of pathology that comprises much of the systemic inflammatory syndrome occurs when one of the usually advantageous roles of pro-inflammatory cytokines – generating sickness behaviour by moderately repressing genes (Dbp, Tef, Hlf, Per1, Per2 and Per3, and the nuclear receptor Rev-erbα) that control circadian rhythm – becomes excessive. Although reversible encephalopathy and gene silencing are severe events with potentially fatal consequences, they can be viewed as having survival advantages through lowering energy demand. In contrast, dyserythropoiesis, seizures, coagulopathy, hypoalbuminaemia and hypertriglyceridaemia may best be viewed as unfortunate consequences of extreme repression of these same genetic controls when the pro-inflammatory cytokines that cause sickness behaviour are produced excessively. As well as casting a new light on the previously unrationalized coexistence of these aspects of systemic inflammatory diseases, this concept is consistent with the case for a primary role for inflammatory cytokines in their pathogenesis across this range of diseases
International R&D rivalry and industrial strategy without government commitment
Available from British Library Document Supply Centre- DSC:3597.9512(CEPR-DP--1199) / BLDSC - British Library Document Supply CentreSIGLEGBUnited Kingdo
Grounding Visual Explanations
Existing models which generate textual explanations enforce task relevance
through a discriminative term loss function, but such mechanisms only weakly
constrain mentioned object parts to actually be present in the image. In this
paper, a new model is proposed for generating explanations by utilizing
localized grounding of constituent phrases in generated explanations to ensure
image relevance. Specifically, we introduce a phrase-critic model to refine
(re-score/re-rank) generated candidate explanations and employ a
relative-attribute inspired ranking loss using "flipped" phrases as negative
examples for training. At test time, our phrase-critic model takes an image and
a candidate explanation as input and outputs a score indicating how well the
candidate explanation is grounded in the image.Comment: Presented at NIPS 2017 Symposium on Interpretable Machine Learnin