6 research outputs found
Asymmetric Bethe-Salpeter equation for pairing and condensation
The Martin-Schwinger hierarchy of correlations are reexamined and the
three-particle correlations are investigated under various partial summations.
Besides the known approximations of screened, ladder and maximally crossed
diagrams the pair-pair correlations are considered. It is shown that the
recently proposed asymmetric Bethe-Salpeter equation to avoid unphysical
repeated collisions is derived as a result of the hierarchical dependencies of
correlations. Exceeding the parquet approximation we show that an asymmetry
appears in the selfconsistent propagators. This form is superior over the
symmetric selfconsistent one since it provides the Nambu-Gorkov equations and
gap equation for fermions and the Beliaev equations for bosons while from the
symmetric form no gap equation results. The selfenergy diagrams which account
for the subtraction of unphysical repeated collisions are derived from the
pair-pair correlation in the three-particle Greenfunction. It is suggested to
distinguish between two types of selfconsistency, the channel-dressed
propagators and the completely dressed propagators, with the help of which the
asymmetric expansion completes the Ward identity and is -derivable.Comment: 12 pages. 26 figure
Stability of condensate in superconductors
According to the BCS theory the superconducting condensate develops in a
single quantum mode and no Cooper pairs out of the condensate are assumed. Here
we discuss a mechanism by which the successful mode inhibits condensation in
neighboring modes and suppresses a creation of noncondensed Cooper pairs. It is
shown that condensed and noncondensed Cooper pairs are separated by an energy
gap which is smaller than the superconducting gap but large enough to prevent
nucleation in all other modes and to eliminate effects of noncondensed Cooper
pairs on properties of superconductors. Our result thus justifies basic
assumptions of the BCS theory and confirms that the BCS condensate is stable
with respect to two-particle excitations
The Modulation of Pain by Circadian and Sleep-Dependent Processes: A Review of the Experimental Evidence
AbstractThis proceedings paper is the first in a series of three papers developing mathematical models for the complex relationship between pain and the sleep-wake cycle. Here, we briefly review what is known about the relationship between pain and the sleep-wake cycle in humans and laboratory rodents in an effort to identify constraints for the models. While it is well accepted that sleep behavior is regulated by a daily (circadian) timekeeping system and homeostatic sleep drive, the joint modulation of these two primary biological processes on pain sensitivity has not been considered. Under experimental conditions, pain sensitivity varies across the 24 h day, with highest sensitivity occurring during the evening in humans. Pain sensitivity is also modulated by sleep behavior, with pain sensitivity increasing in response to the build up of homeostatic sleep pressure following sleep deprivation or sleep disruption. To explore the interaction between these two biological processes using modeling, we first compare the magnitude of their effects across a variety of experimental pain studies in humans. To do this comparison, we normalize the results from experimental pain studies relative to the range of physiologicallymeaningful stimulation levels. Following this normalization, we find that the estimated impact of the daily rhythm and of sleep deprivation on experimental pain measurements is surprisingly consistent across different pain modalities. We also review evidence documenting the impact of circadian rhythms and sleep deprivation on the neural circuitry in the spinal cord underlying pain sensation. The characterization of sleep-dependent and circadian influences on pain sensitivity in this review paper is used to develop and constrain the mathematical models introduced in the two companion articles.</jats:p