Efficient 2‑Aryl Benzothiazole Formation from Aryl Ketones and 2‑Aminobenzenethiols under Metal-Free Conditions

2-Aryl benzothiazole formation from aryl ketones and 2-aminobenzenethiols under metal- and I₂-free conditions was described. Various 2-aryl benzothiazoles were selectively obtained in good yields using molecular oxygen as oxidant. DMSO played an important role in this transformation. Functional groups such as methyl, methoxy, fluoro, chloro, bromo and nitro groups were tolerated under the optimized reaction conditions

Additional file 1: of Cross-linked hyaluronan gel inhibits the growth and metastasis of ovarian carcinoma

Figure S1. The expression of EGFR in A2780 and SKVO3 cells. The celluar lysates were subjected to Western blotting with antibody against EGFR. Expression of β-actin was used at the same time as loading control. (TIFF 676 kb

Nonfullerene Acceptors with Enhanced Solubility and Ordered Packing for High-Efficiency Polymer Solar Cells

The performance of polymer solar cells (PSCs) is commonly improved using additives or annealing treatment. However, these processes are accompanied by disadvantages, including poor reproducibility and stability. Herein, a molecular design strategy is proposed to obtain additive- and annealing-free PSCs. <b>IDTOT2F</b> containing two alkoxyl side chains at the central unit of the nonfullerene acceptor <b>IDTT2F</b> was developed. This molecular design results in excellent solubility in solutions, ordered molecular packing in films, slightly elevated energy levels, and a higher film absorption coefficient. Compared with its counterpart <b>IDTT2F</b>, its improved solubility provides an active layer with better morphology, its ordered molecular packing enhances the charge mobility in blend films, and its slightly elevated energy level furnishes a higher open-circuit voltage of devices. As a result, <b>IDTOT2F</b>-based devices display a maximum power conversion efficiency of 12.79%, which is one of the highest values reported for a PSC fabricated without any extra treatment

Additional file 2: of Long non-coding RNA UFC1 promotes gastric cancer progression by regulating miR-498/Lin28b

Figure S1. UFC1 knockdown inhibits gastric cancer cell proliferation, migration and invasion. Figure S2. UFC1 overexpression enhances gastric cancer cell proliferation, migration and invasion. Figure S3. Bioinformatic prediction of UFC1-binding miRNAs and target genes of miR-498. Figure S4. Relative expression levels of miR-498 and Lin-28b in gastric cancer cells and gastric cancer tissues. Figure S5. UFC1 overexpression antagonizes miR-498-medited inhibition of gastric cancer cell proliferation, migration and invasion. Figure S6. Lin28b knockdown inhibits gastric cancer cell proliferation, migration and invasion. Figure S7. Lin28b overexpression promotes gastric cancer cell proliferation, migration and invasion. Figure S8. UFC1 promotes gastric cancer cell proliferation, migration and invasion via the upregulation of Lin28b. (DOCX 19 kb