658 research outputs found
Direct application of plasmid DNA containing type I interferon transgenes to vaginal mucosa inhibits HSV-2 mediated mortality
- Author
- A Wald
- BA Austin
- BK Kleinschmidt-DeMasters
- Bobbie Ann Austin
- BT Rouse
- C Celum
- Cassandra M. James
- CM Lawson
- DA Baker
- Daniel J. J. Carr
- DJ Carr
- DT Fleming
- EJ Bartlett
- G Raguin
- HH Lee
- I Seif
- IW Emonyi
- J Harland
- J Rajcani
- J Sambrook
- JJ Donnelly
- KA Kelley
- KW Liang
- L Corey
- L Corey
- LR Stanberry
- MB Parr
- MH Wolff
- P Harle
- P Harle
- P Harle
- P Harle
- Peter Härle
- PH Nass
- PH Tan
- R Wang
- RJ Mumper
- S Inoda
- S Lee
- S Noisakran
- S Noisakran
- S Noisakran
- S Noisakran
- SB Mostad
- SJ Noisakran
- SL Sacks
- T Tuting
- TH Tran
- TL Cherpes
- V Liu
- WL McClements
- WP Halford
- WS Yeow
- Publication venue
- Biological Procedures Online
- Publication date
- 01/01/2006
- Field of study
The application of naked DNA containing type I interferon (IFN) transgenes is a promising potential therapeutic approach for controlling chronic viral infections. Herein, we detail the application of this approach that has been extensively used to restrain ocular HSV-1 infection, for antagonizing vaginal HSV-2 infection. We show that application of IFN-α1, -α5, and –β transgenes to vaginal mouse lumen 24 hours prior to HSV-2 infection reduces HSV-2 mediated mortality by 2.5 to 3-fold. However, other type I IFN transgenes (IFN- α4, -α5, -α6, and –α9) are non effectual against HSV-2. We further show that the efficacy of IFN-α1 transgene treatment is independent of CD4+ T lymphocytes. However, in mice depleted of CD8+ T lymphocytes, the ability of IFN-α1 transgene treatment to antagonize HSV-2 was lost
Breast cancer chemoprevention: beyond tamoxifen
- Author
- A Buzdar
- AR Nelson
- AT Ferguson
- AT Koki
- B Bonanni
- B Fisher
- BJ van der Leede
- C Atkinson
- Carol J Fabian
- CJ Fabian
- CJ Fabian
- CJ Fabian
- CJ Fabian
- CK Osborne
- CL Harper-Wynne
- CW Boone
- DL Page
- DV Spicer
- E Daly
- EA Ariazi
- ED Bischoff
- EE Cameron
- FA Tavassoli
- GH Ripple
- GJ Kelloff
- HJ Thompson
- J Leveque
- JA Cauley
- JFR Robertson
- JM Dixon
- JM Nabholtz
- JR Pasqualini
- KC Chan
- Llombart-Cussac
- M Dowsett
- M Pollak
- M Widschwendter
- M Wrensch
- MB Sporn
- MH Gail
- MJ Duffy
- MN Gould
- P Roger
- PA Marks
- PH Brown
- R Day
- R Mehta
- RC Guzman
- RE Harris
- RJ Santen
- S Kato
- S Swami
- SA Fuqua
- SA Khan
- SE Hankinson
- SJ Nass
- T Vink-van Wijngaarden
- TA Fregene
- TJ Powles
- U Veronesi
- X Xu
- XC Xu
- Y Liu
- Publication venue
- BioMed Central
- Publication date
- 17/01/2001
- Field of study
A large number of new potential chemoprevention agents are available that target molecular abnormalities found in estrogen receptor (ER)-negative and/or ER-positive precancerous breast tissue and have side effect profiles that differ from tamoxifen. Classes of agents currently undergoing evaluation in clinical prevention trials or those for which testing is planned in the near future include new selective ER modulators, aromatase inactivators/inhibitors, gonadotrophin-releasing hormone agonists, monoterpenes, isoflavones, retinoids, rexinoids, vitamin D derivatives, and inhibitors of tyrosine kinase, cyclooxygenase-2, and polyamine synthesis. New clinical testing models will use morphological and molecular biomarkers to select candidates at highest short-term risk, to predict the response to a particular class of agent, and to assess the response in phase II prevention trials. If validated, morphological and molecular markers could eventually replace cancer incidence as an indicator of efficacy in future phase III trials
Determination of c-myc amplification and overexpression in breast cancer patients: evaluation of its prognostic value against c-erbB-2, cathepsin-D and clinicopathological characteristics using univariate and multivariate analysis
- Author
- A Feinberg
- A Losch
- A Scorilas
- A Scorilas
- A Scorilas
- AD Thor
- B Tormod
- B Van der Burg
- C Dati
- C Escot
- CG Miyada
- CK Osborne
- D Dubik
- DC Allred
- EL Kaplan
- EM Berns
- EMJJ Berns
- EMJJ Berns
- F Spyratos
- G Ferrandina
- G Gasparini
- GF Santos
- H Kreipe
- H Rochefort
- I Garcia
- JA Foekens
- K Alitalo
- KI Bland
- L Sistonen
- LD Read
- LP Pertschuk
- M Guerin
- M Roux-Dosseto
- OW Prall
- P Chomczynski
- PH Watson
- PS Thomas
- R Alarscon
- S Sjogren
- SJ Nass
- T Ben-Yosef
- T Maniatis
- T Pietilainen
- TC Suen
- U Lonn
- WL McGuire
- ZP Pavelic
- Publication venue
- Nature Publishing Group
- Publication date
- Field of study
C-myc and c-erbB-2 amplification and/or overexpression as well as total cathepsin-D (CD) concentration have been reported to be associated with poor prognosis in breast cancer. The prognostic significance, however, remains somewhat controversial, partly because of discrepancies among the different methodologies used. We determined the amplification and overexpression of c-myc oncogene in 152 breast cancer patients and examined its prognostic value in relation to c-erbB-2 amplification and overexpression, high concentration of CD (≥ 60 pmol mg–1 protein) and standard clinicopathological prognostic factors of the disease. High CD concentration, as well as c-myc amplification and overexpression, proved to be the best of the new variables examined for prediction of early relapse (ER; before 3 years). After multivariate analysis only CD remained significant, which suggests that the prognostic power of these variables is similar. Using univariate analysis we proved that c-myc amplification and overexpression were highly significant for disease-free survival (DFS) (P = 0.0016 and P = 0.0001 respectively) and overall survival (OS) (P < 0.0001 and P = 0.0095 respectively), although by multivariate analysis c-myc overexpression was statistically significant only for DFS (P = 0.0001) and c-myc amplification only for OS (P = 0.0006). With regard to c-erbB-2, only its overexpression appeared to be significant for DFS and OS, although after multivariate analysis its prognostic power was weaker (P = 0.030 and P = 0.024 respectively). c-myc amplification and overexpression exhibited a tendency for locoregional recurrence (LRR) (P = 0.0024 and P = 0.0075 respectively), however, their prognostic value was lower after multivariate analysis and only CD remained significant. © 1999 Cancer Research Campaig
Investigating Bacterial Sources of Toxicity as an Environmental Contributor to Dopaminergic Neurodegeneration
- Author
- A Priyadarshi
- AB Manning-Bog
- AH Schapira
- AL Deimain
- BY Zeng
- C Cook
- CM Tanner
- CM Tanner
- D Jones
- DA Loeffler
- David G. Standaert
- DP Barry
- EG Stringham
- EJ Ryu
- ER Sawin
- EV Mosharov
- F Sun
- FJ Díaz-Corrales
- FP Gailliot
- G Chapman
- GA Caldwell
- Guy A. Caldwell
- J Sulston
- JA Firestone
- Jafa Armagost
- Jeana E. Blalock
- JH Kordower
- JM Gorell
- Jue Chen
- Julie B. Olson
- JY Sze
- K Strange
- Kim A. Caldwell
- KM Lickteig
- KS McNaught
- KT Bush
- L Bedford
- L Carvelli
- M Calfon
- M Maduro
- Mark R. Cookson
- Michelle L. Tucci
- NB Nedelsky
- NF Bence
- PH Janssen
- Philip J. Webber
- Qingmin Ruan
- R Nass
- Robert H. Findlay
- RY Lee
- S Brenner
- S Cao
- S Costello
- S Kohbata
- S Sanyal
- S Tam
- SA Ogata
- SA Ogata
- Shermeen B. Memon
- SL McIntire
- Susan M. DeLeon
- T Decker
- TM Dawson
- Tyler W. Hodges
- UB Pandey
- V Praitis
- X Li
- Y Zhang
- YC Park
- Z Ling
- ZD Ling
- Publication venue
- Public Library of Science
- Publication date
- 01/10/2009
- Field of study
Parkinson disease (PD) involves progressive neurodegeneration, including loss of dopamine (DA) neurons from the substantia nigra. Select genes associated with rare familial forms of PD function in cellular pathways, such as the ubiquitin-proteasome system (UPS), involved in protein degradation. The misfolding and accumulation of proteins, such as α-synuclein, into inclusions termed Lewy Bodies represents a clinical hallmark of PD. Given the predominance of sporadic PD among patient populations, environmental toxins may induce the disease, although their nature is largely unknown. Thus, an unmet challenge surrounds the discovery of causal or contributory neurotoxic factors that could account for the prevalence of sporadic PD. Bacteria within the order Actinomycetales are renowned for their robust production of secondary metabolites and might represent unidentified sources of environmental exposures. Among these, the aerobic genera, Streptomyces, produce natural proteasome inhibitors that block protein degradation and may potentially damage DA neurons. Here we demonstrate that a metabolite produced by a common soil bacterium, S. venezuelae, caused DA neurodegeneration in the nematode, Caenorhabditis elegans, which increased as animals aged. This metabolite, which disrupts UPS function, caused gradual degeneration of all neuronal classes examined, however DA neurons were particularly vulnerable to exposure. The presence of DA exacerbated toxicity because neurodegeneration was attenuated in mutant nematodes depleted for tyrosine hydroxylase (TH), the rate-limiting enzyme in DA production. Strikingly, this factor caused dose-dependent death of human SH-SY5Y neuroblastoma cells, a dopaminergic line. Efforts to purify the toxic activity revealed that it is a highly stable, lipophilic, and chemically unique small molecule. Evidence of a robust neurotoxic factor that selectively impacts neuronal survival in a progressive yet moderate manner is consistent with the etiology of age-associated neurodegenerative diseases. Collectively, these data suggest the potential for exposures to the metabolites of specific common soil bacteria to possibly represent a contributory environmental component to PD
Environmental Enrichment Preceding Early Adulthood Methylphenidate Treatment Leads to Long Term Increase of Corticosterone and Testosterone in the Rat
- Author
- A Angold
- A Avital
- A Avital
- A Avital
- A Degroot
- AR Aron
- AR Lumia
- Avi Avital
- B Sahakian
- C Poulin
- C Smith
- CA Bolanos
- CA Bolanos
- CJ Teter
- DC Lagace
- E Bruel-Jungerman
- E Palsson
- E Waite
- EE Belz
- EJ Hermans
- EM Marco
- FK Graham
- G Laviola
- GB Britton
- GB Varty
- GM Bright
- H Abikoff
- H Bonilla-Jaime
- H Zhang
- I Branchi
- I van Bokhoven
- IC Weiss
- J Buitelaar
- J Hannestad
- J Sinzig
- J Veena
- JM Carre
- JM Rey
- JM Swanson
- JM Zito
- JR Sanchez-Martin
- JT Vreeburg
- JT Vreeburg
- K Kirby
- KA Frahm
- KE Bogle
- KE Scholes
- KJ Miller
- KK Soma
- KM Frick
- L Spear
- LP Spear
- LW Hawk Jr
- M Cui
- MA Mehta
- MB Forrester
- MG Leggio
- MH Teicher
- N Benaroya-Milshtein
- ND Volkow
- OB Amaral
- P Basu
- P Chapillon
- P Golubchik
- PH Mehta
- PS Jensen
- R Quinn
- RD Nass
- RJ Williams
- S Hart
- S King
- S Morley-Fletcher
- S Rose
- SA Ferguson
- Salman Zubedat
- SD Mague
- Shlomit Aga-Mizrachi
- SI Svetlov
- Talya Dolev
- Thomas Burne
- TL Schaefer
- TM Pham
- VR Anderson
- WA Carlezon Jr
- Y Ilin
- YP Tang
- Z Kohl
- Publication venue
- Public Library of Science
- Publication date
- 01/01/2011
- Field of study
Attention-deficit/hyperactivity disorder (ADD/ADHD) has been emerging as a world-wide psychiatric disorder. There appears to be an increasing rate of stimulant drug abuse, specifically methylphenidate (MPH) which is the most common treatment for ADHD, among individuals who do not meet the criteria for ADHD and particularly for cognitive enhancement among university students. However, the long term effects of exposure to MPH are unknown. Thus, in light of a developmental approach in humans, we aimed to test the effects of adolescence exposure to enriched environment (EE) followed by MPH administration during early adulthood, on reactions to stress in adulthood. Specifically, at approximate adolescence [post natal days (PND) 30–60] rats were reared in EE and were treated with MPH during early adulthood (PND 60–90). Adult (PND 90–92) rats were exposed to mild stress and starting at PND 110, the behavioral and endocrine effects of the combined drug and environmental conditions were assessed. Following adolescence EE, long term exposure to MPH led to decreased locomotor activity and increased sucrose preference. EE had a beneficial effect on PPI (attentive abilities), which was impaired by long term exposure to MPH. Finally, the interaction between EE and, exposure to MPH led to long-term elevated corticosterone and testosterone levels. In view of the marked increase in MPH consumption over the past decade, vigilance is crucial in order to prevent potential drug abuse and its long term detrimental consequences
Does the Design of a Robot Influence Its Animacy and Perceived Intelligence?
- Author
- Abdullah Al Mahmud
- B Scholl
- B Webb
- C Bartneck
- C Bartneck
- C Bartneck
- C Bartneck
- C Bartneck
- C Nass
- CF DiSalvo
- Christoph Bartneck
- D Kulic
- D Poulin-Dubois
- DH Rakison
- E Kubinyi
- F Heider
- GF Melson
- H Ishiguro
- H McGurk
- J Halloy
- JL Jipson
- KM Lee
- M Slater
- O Holland
- Omar Mubin
- Oxford University Press
- P Blythe
- P Kahn
- PD Tremoulet
- PH Kahn
- R Sparrow
- RM Warner
- RV Dawis
- S Kiesler
- S Milgram
- S Parise
- S Turkle
- SY Okita
- Takayuki Kanda
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- Field of study
Determination of the parton distribution functions of the proton using diverse ATLAS data from pp collisions at √s = 7, 8 and 13 TeV
- Author
- Aad G
- Abbott B
- Abbott DC
- Abeling K
- Abhayasinghe DK
- Abidi SH
- Aboulhorma A
- Abramowicz H
- Abreu H
- Abud A Abed
- Abulaiti Y
- Acharya BS
- Achkar B
- Adam L
- Adamczyk L
- Adamek L
- Addepalli SV
- Adelman J
- Adiguzel A
- Adorni S
- Adye T
- Affolder AA
- Afik Y
- Agapopoulou C
- Agaras MN
- Agarwala J
- Aggarwal A
- Agheorghiesei C
- Aguilar-Saavedra JA
- Agullo P Martinez
- Ahmad A
- Ahmadov F
- Ahmed WS
- Ahnen J Von
- Ai X
- Aielli G
- Aizenberg I
- Akbiyik M
- Akimov AV
- Alberghi GL
- Albert J
- Albicocco P
- Alderweireldt S
- Aleksa M
- Aleksandrov IN
- Alexa C
- Alexopoulos T
- Alfonsi A
- Alfonsi F
- Alhroob M
- Ali B
- Ali S
- Aliev M
- Alimonti G
- Allaire C
- Allbrooke BMM
- Allport PP
- Aloisio A
- Alonso F
- Alpigiani C
- Alves FL Lucio
- Alviggi MG
- Ambler A
- Ambroz L
- Amelung C
- Amidei D
- Amoroso S
- Amos KR
- Amrouche CS
- Ananiev V
- Anastopoulos C
- Andana RY González
- Andari N
- Andeen T
- Anders JK
- Andrean SY
- Andreazza A
- Angelidakis S
- Angerami A
- Anisenkov AV
- Annovi A
- Antel C
- Anthony MT
- Antipov E
- Antonelli M
- Antrim DJA
- Anulli F
- Aoki M
- Aparo MA
- Aranda C Padilla
- Aranzabal N
- Araujo M Verissimo De
- Araya S Tapia
- Arcangeletti C
- Arce ATH
- Arena E
- Arguin J-F
- Argyropoulos S
- Arling J-H
- Armbruster AJ
- Armstrong A
- Arnaez O
- Arnold H
- Artoni G
- Asada H
- Asai K
- Asai S
- Asbah NA
- Asimakopoulou EM
- Asmundis R De
- Asquith L
- Assahsah J
- Assamagan K
- Astalos R
- Astigarraga ME Pozo
- Atkin RJ
- Atkinson M
- Atlay NB
- Atmani H
- Atmasiddha PA
- Augsten K
- Auricchio S
- Austrup VA
- Avner G
- Avolio G
- Ayoub MK
- Azuelos G
- Babal D
- Bachacou H
- Bachas K
- Bachiu A
- Backman F
- Badea A
- Bagnaia P
- Bahilo JJ Lozano
- Bahmani M
- Bailey AJ
- Bailey VR
- Baines JT
- Bakalis C
- Baker OK
- Bakker PJ
- Bakos E
- Balaji S
- Balasubramanian R
- Baldin EM
- Balek P
- Ballabene E
- Balli F
- Baltes LM
- Balunas WK
- Balz J
- Banas E
- Bandieramonte M
- Bandyopadhyay A
- Bansal S
- Barak L
- Barberio EL
- Barberis D
- Barbero M
- Barbour G
- Barends KN
- Barillari T
- Barisits M-S
- Barkeloo J
- Barklow T
- Barnett RM
- Baron P
- Baroncelli A
- Barone G
- Barr AJ
- Barreiro F
- Barron U
- Barrue RF Coelho
- Barsov S
- Bartels F
- Bartoldus R
- Bartolini G
- Barton AE
- Bartos P
- Basalaev A
- Basan A
- Baselga M
- Bashta I
- Bassalat A
- Basso MJ
- Basson CR
- Bates RL
- Batlamous S
- Batley JR
- Batool B
- Battaglia M
- Bauce M
- Bauer F
- Bauer P
- Bayirli A
- Beacham JB
- Beau T
- Beauchemin PH
- Becherer F
- Bechtle P
- Beck HP
- Becker K
- Becot C
- Beddall AJ
- Bednyakov VA
- Bee CP
- Beemster LJ
- Beermann TA
- Begalli M
- Begel M
- Behera A
- Behr JK
- Beirer JF
- Beisiegel F
- Belfkir M
- Bella G
- Bella L Aperio
- Bellagamba L
- Bellerive A
- Bello FA Di
- Bellos P
- Beloborodov K
- Belotskiy K
- Belyaev NL
- Benchekroun D
- Benhammou Y
- Benjamin DP
- Benoit M
- Bensinger JR
- Bentvelsen S
- Beresford L
- Beretta M
- Berge D
- Berger N
- Bergmann B
- Bergsten LJ
- Beringer J
- Berlendis S
- Berlingen J Montejo
- Bernardi G
- Bernius C
- Bernlochner FU
- Berry T
- Berta P
- Bertram IA
- Bethke S
- Betti A
- Beurs M De
- Bevan AJ
- Bhatta S
- Bhattacharya DS
- Bhattarai P
- Bhopatkar VS
- Bi R
- Bi R
- Bianchi RM
- Biebel O
- Bielski R
- Biesuz NV
- Biglietti M
- Billoud TRV
- Bindi M
- Bingul A
- Bini C
- Biondi S
- Biondini A
- Birch-sykes CJ
- Bird GA
- Birman M
- Bisanz T
- Biswal JP
- Biswas D
- Bitadze A
- Bjørke K
- Bloch I
- Blocker C
- Blue A
- Blumenschein U
- Blumenthal J
- Bobbink GJ
- Bobrovnikov VS
- Boehler M
- Boeriu OE Vickey
- Bogavac D
- Bogdanchikov AG
- Bohm C
- Boisvert V
- Bokan P
- Bolanos G Rabanal
- Bold T
- Bomben M
- Bona M
- Boonekamp M
- Booth CD
- Borbély AG
- Borecka-Bielska HM
- Borgna LS
- Borissov G
- Bortoletto D
- Bosca S Rodriguez
- Boscherini D
- Bosman M
- Bostanabad M Ghasemi
- Bouaouda K
- Boudreau J
- Bouhova-Thacker EV
- Boumediene D
- Bouquet R
- Bourdarios C Adam
- Boveia A
- Boyd J
- Boye D
- Boyko IR
- Bracinik J
- Brahimi N
- Brandt G
- Brandt O
- Braren F
- Brau B
- Brau JE
- Brendlinger K
- Brener R
- Brenner L
- Brenner R
- Bressler S
- Bret M Cano
- Brickwedde B
- Britton D
- Britzger D
- Brock I
- Brooijmans G
- Brooks WK
- Brost E
- Bruncko D
- Bruni A
- Bruni G
- Bruschi M
- Bruscino N
- Bryngemark L
- Brüers B
- Buanes T
- Buat Q
- Buchholz P
- Buckley AG
- Budagov IA
- Bugge MK
- Bulekov O
- Bullard BA
- Burdin S
- Burgard CD
- Burger AM
- Burghgrave B
- Burr JTP
- Burton CD
- Burzynski JC
- Busch EL
- Bussey PJ
- Butler JM
- Buttar CM
- Butterworth JM
- Buttinger W
- Buzykaev AR
- Bylund O Bessidskaia
- Büscher V
- Cabras G
- Caforio D
- Cai H
- Cairo VMM
- Cakir I Turk
- Cakir O
- Calace N
- Calafiura P
- Calderini G
- Calfayan P
- Callea G
- Caloba LP
- Calvet D
- Calvet S
- Calvet TP
- Calvetti M
- Camarda S
- Camarri P
- Camelia E Alunno
- Camerlingo MT
- Cameron D
- Camincher C
- Campanelli M
- Camplani A
- Canale V
- Canesse A
- Cantero J
- Cao Y
- Capocasa F
- Capriles FG Diaz
- Capua M
- Carbone A
- Cardarelli R
- Cardenas JCJ
- Cardillo F
- Carducci G
- Carli T
- Carlino G
- Carlson BT
- Carlson EM
- Carlson T Ingebretsen
- Carminati L
- Carnesale M
- Caron S
- Carquin E
- Carratta G
- Carrá S
- Carter JWS
- Carter TM
- Carvalho AL Moreira De
- Casadei D
- Casado MP
- Casha AF
- Castiglia EG
- Castillo FL
- Castillo LR Flores
- Castro NF
- Castro S De
- Catinaccio A
- Catmore JR
- Cavaliere V
- Cavalli N
- Cavasinni V
- Celebi E
- Celli F
- Centonze MS
- Cerny K
- Cerqueira AS
- Cerri A
- Cerrito L
- Cerutti F
- Cervelli A
- Cetin SA
- Chadi Z
- Chakraborty D
- Chala M
- Chan J
- Chan WS
- Chan WY
- Chapman JD
- Chargeishvili B
- Charlton DG
- Charman TP
- Chatterjee M
- Chekanov S
- Chekulaev SV
- Chelkov GA
- Chen A
- Chen B
- Chen B
- Chen C
- Chen H
- Chen H
- Chen J
- Chen J
- Chen S
- Chen SJ
- Chen X
- Chen X
- Chen Y
- Cheng CL
- Cheng HC
- Cheplakov A
- Cheremushkina E
- Cherepanova E
- Cheu E
- Cheung K
- Chevalier L
- Chiarella V
- Chiarelli G
- Chiodini G
- Chisholm AS
- Chitan A
- Chiu YH
- Chizhov MV
- Choi K
- Chomont AR
- Chou Y
- Chow YS
- Chowdhury T
- Christopher LD
- Chu MC
- Chu X
- Chudoba J
- Chwastowski JJ
- Ciaccio A Di
- Ciaccio L Di
- Cieri D
- Ciesla KM
- Cindro V
- Ciocio A
- Cirotto F
- Citron ZH
- Citterio M
- Ciubotaru DA
- Ciungu BM
- Clark A
- Clark PJ
- Clawson SE
- Clement C
- Clissa L
- Coadou Y
- Cobal M
- Coccaro A
- Codina E Perez
- Coelli S
- Cohen H
- Coimbra AEC
- Cole B
- Collot J
- Columbie JM Clavijo
- Connell SH
- Connelly IA
- Conroy EI
- Conventi F
- Cooke HG
- Cooper-Sarkar AM
- Cormier F
- Cornell S Díez
- Corpe LD
- Corradi M
- Correia AM Henriques
- Corrigan EE
- Corriveau F
- Costa AM Mendes Jacques Da
- Costa MJ
- Costanza F
- Costanzo D
- Cote BM
- Coutinho Y Amaral
- Cowan G
- Cowley JW
- Cranmer K
- Crescioli F
- Cristinziani M
- Cristoforetti M
- Croft V
- Crosetti G
- Crépé-Renaudin S
- Cueto A
- Cui H
- Cui Z
- Cukierman AR
- Cunningham WR
- Curcio F
- Czodrowski P
- Czurylo MM
- da Costa J Barreiro Guimarães
- Daalen TR Van
- Dabrowski W
- Dado T
- Dahbi S
- Dai T
- Dallapiccola C
- Dam M
- Damazio D Oliveira
- Damp J
- Dandoy JR
- Daneri MF
- Danninger M
- Dao V
- Darbo G
- Darmora S
- Dattagupta A
- David C
- Davidek T
- Davis DR
- Davis-Purcell B
- Dawson I
- de Jong P
- de la Hoz S González
- de Renstrom PA Bruckman
- De K
- Dedovich DV
- Degens J
- Deiana AM
- Deliot F
- Delitzsch CM
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- Zhemchugov A
- Zheng Z
- Zhong D
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- Zhukov K
- Zhulanov V
- Zieminska D
- Zimine NI
- Zimmermann S
- Zinsser J
- Ziolkowski M
- Zoccoli A
- Zoch K
- Zorbas TG
- Zormpa O
- Zou W
- Zwalinski L
- Åkesson TPA
- Öncel ÖO
- Ženiš T
- Živković L
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2022
- Field of study
This paper presents an analysis at next-to-next-to-leading order in the theory of quantum chromodynamics for the determination of a new set of proton parton distribution functions using diverse measurements in pp collisions at \sqrt{s} = 7, 8 and 13 TeV, performed by the ATLAS experiment at the Large Hadron Collider, together with deep inelastic scattering data from ep collisions at the HERA collider. The ATLAS data sets considered are differential cross-section measurements of inclusive W^{±} and Z/gamma^{*} boson production, W^{±} and Z boson production in association with jets, t\bar{t} production, inclusive jet production and direct photon production. In the analysis, particular attention is paid to the correlation of systematic uncertainties within and between the various ATLAS data sets and to the impact of model, theoretical and parameterisation uncertainties. The resulting set of parton distribution functions is called ATLASpdf21
Modelling and computational improvements to the simulation of single vector-boson plus jet processes for the ATLAS experiment
- Author
- Aad G
- Abbott B
- Abbott DC
- Abeling K
- Abhayasinghe DK
- Abidi SH
- Aboulhorma A
- Abramowicz H
- Abreu H
- Abud A Abed
- Abulaiti Y
- Abusleme Hoffman AC
- Acharya BS
- Achkar B
- Adam L
- Adamczyk L
- Adamek L
- Addepalli SV
- Adelman J
- Adiguzel A
- Adorni S
- Adye T
- Affolder AA
- Afik Y
- Agapopoulou C
- Agaras MN
- Agarwala J
- Aggarwal A
- Agheorghiesei C
- Aguilar-Saavedra JA
- Ahmad A
- Ahmadov F
- Ahmed WS
- Ai X
- Aielli G
- Aizenberg I
- Akbiyik M
- Akesson TPA
- Akimov AV
- Al Khoury K
- Alberghi GL
- Albert J
- Albicocco P
- Alderweireldt S
- Aleksa M
- Aleksandrov IN
- Alexa C
- Alexopoulos T
- Alfonsi A
- Alfonsi F
- Alhroob M
- Ali B
- Ali S
- Aliev M
- Alimonti G
- Allaire C
- Allbrooke BMM
- Allport PP
- Aloisio A
- Alonso F
- Alpigiani C
- Alvarez Estevez M
- Alviggi MG
- Ambler A
- Ambroz L
- Amelung C
- Amidei D
- Amor Dos Santos SP
- Amoroso S
- Amos KR
- Amrouche CS
- Ananiev V
- Anastopoulos C
- Andari N
- Andeen T
- Anders JK
- Andrean SY
- Andreazza A
- Angelidakis S
- Angerami A
- Anisenkov AV
- Annovi A
- Antel C
- Anthony MT
- Antipov E
- Antonelli M
- Antrim DJA
- Anulli F
- Aoki M
- Aparisi Pozo JA
- Aparo MA
- Aranda C Padilla
- Aranzabal N
- Araya S Tapia
- Arcangeletti C
- Arce ATH
- Arena E
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- Argyropoulos S
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- Zhemchugov A
- Zheng Z
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- Zieminska D
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- Zimmermann S
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- Zivkovic L
- Zoccoli A
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- Zorbas TG
- Zormpa O
- Zou W
- Zwalinski L
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2022
- Field of study
This paper presents updated Monte Carlo configurations used to model the production of single electroweak vector bosons (W, Z/gamma*) in association with jets in proton-proton collisions for the ATLAS experiment at the Large Hadron Collider. Improvements pertaining to the electroweak input scheme, parton-shower splitting kernels and scale-setting scheme are shown for multi-jet merged configurations accurate to next-to-leading order in the strong and electroweak couplings. The computational resources required for these set-ups are assessed, and approximations are introduced resulting in a factor three reduction of the per-event CPU time without affecting the physics modelling performance. Continuous statistical enhancement techniques are introduced by ATLAS in order to populate low cross-section regions of phase space and are shown to match or exceed the generated effective luminosity. This, together with the lower per-event CPU time, results in a 50% reduction in the required computing resources compared to a legacy set-up previously used by the ATLAS collaboration. The set-ups described in this paper will be used for future ATLAS analyses and lay the foundation for the next generation of Monte Carlo predictions for single vector-boson plus jets production
Measurement of the energy asymmetry in t(t)over-barj production at 13 TeV with the ATLAS experiment and interpretation in the SMEFT framework
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- Zhao Z
- Zhemchugov A
- Zheng Z
- Zhong D
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- Publication venue
- 'Springer Fachmedien Wiesbaden GmbH'
- Publication date
- 28/04/2022
- Field of study
A measurement of the energy asymmetry in jet-associated top-quark pair production is presented using 139fb−1
139
fb
-
1
of data collected by the ATLAS detector at the Large Hadron Collider during pp collisions at s=13TeV
s
=
13
TeV
. The observable measures the different probability of top and antitop quarks to have the higher energy as a function of the jet scattering angle with respect to the beam axis. The energy asymmetry is measured in the semileptonic ttˉ
t
t
¯
decay channel, and the hadronically decaying top quark must have transverse momentum above 350GeV
350
GeV
. The results are corrected for detector effects to particle level in three bins of the scattering angle of the associated jet. The measurement agrees with the SM prediction at next-to-leading-order accuracy in quantum chromodynamics in all three bins. In the bin with the largest expected asymmetry, where the jet is emitted perpendicular to the beam, the energy asymmetry is measured to be −0.043±0.020
-
0.043
±
0.020
, in agreement with the SM prediction of −0.037±0.003
-
0.037
±
0.003
. Interpreting this result in the framework of the Standard Model effective field theory (SMEFT), it is shown that the energy asymmetry is sensitive to the top-quark chirality in four-quark operators and is therefore a valuable new observable in global SMEFT fits
Constraints on Higgs boson production with large transverse momentum using H \rightarrow b\bar{b} decays in the ATLAS detector
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- Aad G
- Abbott B
- Abbott DC
- Abed Abud A
- Abeling K
- Abhayasinghe DK
- Abidi SH
- Aboulhorma A
- Abramowicz H
- Abreu H
- Abulaiti Y
- Abusleme Hoffman AC
- Acharya BS
- Achkar B
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- Adamek L
- Addepalli SV
- Adelman J
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- Alberghi GL
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- Öncel ÖO
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- Publication venue
- 'American Physical Society (APS)'
- Publication date
- 01/05/2022
- Field of study
This paper reports constraints on Higgs boson production with transverse momentum above 1 TeV. The analyzed data from proton–proton collisions at a center-of-mass energy of 13 TeV were recorded with the ATLAS detector at the Large Hadron Collider from 2015 to 2018 and correspond to an integrated luminosity of 136fb^{-1}. Higgs bosons decaying into b\bar{b} are reconstructed as single large-radius jets recoiling against a hadronic system and are identified by the experimental signature of two b-hadron decays. The experimental techniques are validated in the same kinematic regime using the Z \rightarrow b\bar{b} process. The 95% confidence-level upper limit on the cross section for Higgs boson production with transverse momentum above 450 GeV is 115 fb, and above 1 TeV it is 9.6 fb. The Standard Model cross section predictions for a Higgs boson with a mass of 125 GeV in the same kinematic regions are 18.4 fb and 0.13 fb, respectively
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