973 research outputs found
Pemahaman Identitas dan Toleransi Keberagaman Budaya Mahasiswa Sastra Inggris UAI melalui Puisi Multikultural Kesusasteraan Inggris: sebuah Kajian Multikulturalisme
- Penelitian ini dilakukan untuk melihat respon mahasiswa Sastra Inggris UAI dalam memahami konflik keberagaman dan pemahaman identitas budaya terkait dengan ideologi multikulturalisme melalui puisi multikultural kesusasteraan Inggris. Penelitian ini merupakan penelitian kualitatif dengan menggunakan metode reader-response (respon pembaca) dengan subjek penelitian yaitu mahasiswa Sastra Inggris UAI yang mengambil mata kuliah Pengkajian Puisi sebanyak 26 orang. Dengan menggunakan teori Identitas oleh Stuart Hall sebagai kajian multikulturalisme dan teori intrinsik puisi, penelitian ini diharapkan dapat memberikan gambaran jelas mengenai respon mahasiswa Sastra Inggris terhadap teks-teks sastra bertema multikultural dan juga gambaran jelas sejauh mana pemahaman lintas budaya mereka. Pada akhirnya hasil ini secara garis besar menunjukkan bahwa pemahaman keberagaman, kepedulian sosial dan toleransi di kalangan mahasiswa sastra Inggris UAI belum baik karena sebanyak 53.86% responden terlihat mengalami kesulitan untuk memahami dan menginterpretasikan isi puisi serta tidak bisa mengaitkan tema puisi dengan kondisi keberagaman di kehidupan mereka sehari-hari. Kata Kunci – Multikulturalisme, Identitas Budaya, Identitas Budaya, Tanggapan Pembac
Periods for flat algebraic connections
In previous work, we established a duality between the algebraic de Rham
cohomology of a flat algebraic connection on a smooth quasi-projective surface
over the complex numbers and the rapid decay homology of the dual connection
relying on a conjecture by C. Sabbah, which has been proved recently by T.
Mochizuki for algebraic connections in any dimension. In the present article,
we verify that Mochizuki's results allow to generalize these duality results to
arbitrary dimensions also
Analytic continuation of residue currents
Let be a complex manifold and f\colon X\to \C^p a holomorphic mapping
defining a complete intersection. We prove that the iterated Mellin transform
of the residue integral associated to has an analytic continuation to a
neighborhood of the origin in \C^p
Verdier specialization via weak factorization
Let X in V be a closed embedding, with V - X nonsingular. We define a
constructible function on X, agreeing with Verdier's specialization of the
constant function 1 when X is the zero-locus of a function on V. Our definition
is given in terms of an embedded resolution of X; the independence on the
choice of resolution is obtained as a consequence of the weak factorization
theorem of Abramovich et al. The main property of the specialization function
is a compatibility with the specialization of the Chern class of the complement
V-X. With the definition adopted here, this is an easy consequence of standard
intersection theory. It recovers Verdier's result when X is the zero-locus of a
function on V. Our definition has a straightforward counterpart in a motivic
group. The specialization function and the corresponding Chern class and
motivic aspect all have natural `monodromy' decompositions, for for any X in V
as above. The definition also yields an expression for Kai Behrend's
constructible function when applied to (the singularity subscheme of) the
zero-locus of a function on V.Comment: Minor revision. To appear in Arkiv f\"or Matemati
Cosmic ray short burst observed with the Global Muon Detector Network (GMDN) on June 22, 2015
We analyze the short cosmic ray intensity increase ("cosmic ray burst": CRB)
on June 22, 2015 utilizing a global network of muon detectors and derive the
global anisotropy of cosmic ray intensity and the density (i.e. the
omnidirectional intensity) with 10-minute time resolution. We find that the CRB
was caused by a local density maximum and an enhanced anisotropy of cosmic rays
both of which appeared in association with Earth's crossing of the heliospheric
current sheet (HCS). This enhanced anisotropy was normal to the HCS and
consistent with a diamagnetic drift arising from the spatial gradient of cosmic
ray density, which indicates that cosmic rays were drifting along the HCS from
the north of Earth. We also find a significant anisotropy along the HCS,
lasting a few hours after the HCS crossing, indicating that cosmic rays
penetrated into the inner heliosphere along the HCS. Based on the latest
geomagnetic field model, we quantitatively evaluate the reduction of the
geomagnetic cut-off rigidity and the variation of the asymptotic viewing
direction of cosmic rays due to a major geomagnetic storm which occurred during
the CRB and conclude that the CRB is not caused by the geomagnetic storm, but
by a rapid change in the cosmic ray anisotropy and density outside the
magnetosphere.Comment: accepted for the publication in the Astrophysical Journa
Singularities and Topology of Meromorphic Functions
We present several aspects of the "topology of meromorphic functions", which
we conceive as a general theory which includes the topology of holomorphic
functions, the topology of pencils on quasi-projective spaces and the topology
of polynomial functions.Comment: 21 pages, 1 figur
Retinoic acid inducible gene I Activates innate antiviral response against human parainfluenza virus type 3
Human parainfluenza virus type 3 (HPIV3) is a respiratory paramyxovirus that infects lung epithelial cells to cause high morbidity among infants and children. To date, no effective vaccine or antiviral therapy exists for HPIV3 and therefore, it is important to study innate immune antiviral response induced by this virus in infected cells. Type-I interferons (IFN, interferon-α/β) and tumor necrosis factor-α (TNFα activated by NFκB) are potent antiviral cytokines that play an important role during innate immune antiviral response. A wide-spectrum of viruses utilizes pattern recognition receptors (PRRs) like toll-like receptors (TLRs) and RLH (RIG like helicases) receptors such as RIGI (retinoic acid inducible gene -I) and Mda5 to induce innate antiviral response. Previously it was shown that both TNFα and IFNβ are produced from HPIV3 infected cells. However, the mechanism by which infected cells activated innate response following HPIV3 infection was not known. In the current study, we demonstrated that RIGI serves as a PRR in HPIV3 infected cells to induce innate antiviral response by expressing IFNβ (via activation of interferon regulatory factor-3 or IRF3) and TNFα (via activation of NF-κB)
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