265 research outputs found
Multi-vortex solution in the Sutherland model
We consider the large- Sutherland model in the Hamiltonian
collective-field approach based on the expansion. The Bogomol'nyi limit
appears and the corresponding solutions are given by static-soliton
configurations. They exist only for \l<1, i.e. for the negative coupling
constant of the Sutherland interaction. We determine their creation energies
and show that they are unaffected by higher-order corrections. For \l=1, the
Sutherland model reduces to the free one-plaquette Kogut-Susskind model.Comment: Latex, using ioplppt.sty, 11 page
Solitons and fractional statistics
Solitons in the continuum limit of the Calogero model are derived and shown
to correspond to one-particle excitations. The statistical mechanics of
exclusion statistics particles is then formulated in terms of a priori
probabilities and a path integral is thereoff constructed. (Talk delivered at
the Trieste April 1995 Conference on statistical mechanics and QFT and at the
Oslo August 1995 Worskhop on low-dimensional systems.)Comment: 7 pages, Latex, no figures; References correcte
Progressive collapse analysis of a bulk carrier using IACS prescribed and NLFEM derived load - end shortening curves
Density Correlation Functions in Calogero Sutherland Models
Using arguments from two dimensional Yang-Mills theory and the collective
coordinate formulation of the Calogero-Sutherland model, we conjecture the
dynamical density correlation function for coupling and , where is
an integer. We present overwhelming evidence that the conjecture is indeed
correct.Comment: 12 pages phyzzx, CERN-TH/94.7243 One reference change
METABOLIC ISSUES IN PSYCHOTIC DISORDERS WITH THE FOCUS ON FIRST-EPISODE PATIENTS: A REVIEW
Before the onset of the illness, future schizophrenia patients do not weigh more comparing to their peers. However, during the
later course of the illness, obesity is twice as prevalent as in general public, afflicting the half of schizophrenia patient population.
There is a list of potential factors that contribute to this, including lifestyle, dietary habits, unsatisfactory monitoring of physical
health etc, but nowadays side effects of antipsychotic medication become the most prominent concern when weight gain and
metabolic issues in psychosis are addressed.
The fact is that second generation antipsychotics (SGA) are associated with weight gain and metabolic syndrome, but that might
be the case with the first generation antipsychotics (FGA) too. Besides, obesity might be evident in patients before any exposure to
medications, and all that bring lot of dilemmas into the field. This paper critically reviews available data on metabolic problems in
patients with psychotic disorders, raging from genetic to molecular and environmental factors, and highlights the necessity of
screening for the early signs of metabolic disturbances, as well as of multidisciplinary assessment of psychiatric and medical
conditions from the first psychotic episode
Multielectron spectroscopy: Auger decays of the argon 2p hole
All the different Auger decay paths of Argon 2p holes have been characterized using a time of flight spectrometer of the magnetic bottle type. All electrons (the photoelectron and up to three Auger electrons) are detected in coincidence and resolved in energy. Double Auger decay is shown to proceed either through a direct process or by intense cascade paths, implying highly excited autoionizing Ar2+ states, which are identified as Ar2+ 3sâ2 correlation satellites. Triple Auger decay is also observed and estimated to account for 0.2% only of all Auger decay
Dynamics of electron emission in double photoionization processes near the Krypton 3d threshold
Two electron emission following photoabsorption near the Kr 3d threshold is investigated both experimentally and theoretically. On the experimental side, electron/electron coincidences using a magnetic bottle time of flight spectrometer allow us to observe the complete Double Photo Ionisation (DPI) continua of selected Kr2+ final states, and to see how these continua are affected by resonant processes in the vicinity of the Kr 3d threshold. The analysis is based on a quantum mechanical approach that takes into account the contribution of three different processes: A) Auger decay of the inner 3d vacancy with the associated post collision interaction (PCI) effects, B) capture of slow photoelectrons into discrete states followed by valence multiplet decay (VMD) of the excited ionic states and C) valence shell DPI. The dominant process for each Kr2+(4p-2) final state is the photoionization of the inner shell followed by Auger decay of the 3d vacancies. Moreover, for the 4p2(3P) and 4p-2(1D) final ionic states an important contribution comes from the processes of slow photoelectron capture followed by VMD as well as from double ionization of the outer shell involving also VMD
The emerging role of the FKBP5 gene polymorphisms in vulnerability-stress model of schizophrenia: further evidence from a Serbian population
Increased reactivity to stress is observed in patients with schizophrenia spectrum disorders and their healthy siblings in comparison with the general population. Additionally, higher levels of neuroticism, as a proposed psychological measure of stress sensitivity, increase the risk of schizophrenia. HPA axis dysregulation is one of the possible mechanisms related to the vulnerabilityâstress model of schizophrenia, and recent studies revealed a possible role of the functional genetic variants of FK506-binding protein 51 (FKBP5) gene which modulate activity of HPA axis. The purpose of the present study was to investigate impact of FKBP5 on schizophrenia in Serbian patients and to explore relationship between genetic variants and neuroticism by using the caseâsiblingâcontrol design. In 158 subjects, we measured psychotic experiences, childhood trauma and neuroticism. Nine single-nucleotide polymorphisms (rs9295158, rs3800373, rs9740080, rs737054, rs6926133, rs9380529, rs9394314, rs2766533 and rs12200498) were genotyped. The genetic influence was modeled using logistic regression, and the relationship between genetic variants and neuroticism was assessed by linear mixed model. Our results revealed genetic main effect of FKBP5 risk alleles (A allele of rs9296158 and T allele of rs3800373) and AGTC âriskâ haplotype combination (rs9296158, rs3800373, rs9470080 and rs737054, respectively) on schizophrenia, particularly when childhood trauma was set as a confounding factor. We confirmed strong relationship between neuroticism and psychotic experiences in patients and siblings and further showed relationship between higher levels of neuroticism and FKBP5 risk variants suggesting potential link between biological and psychosocial risk factors. Our data support previous findings that trauma exposure shapes FKBP5 impact on schizophreni
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