Quanto tempo leva para se colocar um tênis? Um ritual obsessivo-compulsivo

Submitted by Rodrigo Senorans ([email protected]) on 2015-04-22T13:01:30Z No. of bitstreams: 1 How long does it take for putting on sneakers.pdf: 585668 bytes, checksum: 9bd79f6899a881fbad03a544f1363050 (MD5)Approved for entry into archive by Anderson Silva ([email protected]) on 2015-04-29T16:41:53Z (GMT) No. of bitstreams: 1 How long does it take for putting on sneakers.pdf: 585668 bytes, checksum: 9bd79f6899a881fbad03a544f1363050 (MD5)Approved for entry into archive by Anderson Silva ([email protected]) on 2015-05-04T16:33:59Z (GMT) No. of bitstreams: 1 How long does it take for putting on sneakers.pdf: 585668 bytes, checksum: 9bd79f6899a881fbad03a544f1363050 (MD5)Made available in DSpace on 2015-05-05T18:19:30Z (GMT). No. of bitstreams: 1 How long does it take for putting on sneakers.pdf: 585668 bytes, checksum: 9bd79f6899a881fbad03a544f1363050 (MD5) Previous issue date: 2014Universidade Federal do Rio de Janeiro. Hospital Universitário Clementino Fraga Filho. Departamento de Neurologia. Rio de Janeiro, RJ, Brasil.Universidade Federal do Rio de Janeiro. Hospital Universitário Clementino Fraga Filho. Departamento de Neurologia. Rio de Janeiro, RJ, Brasil.Fundação Oswaldo Cruz. Instituto Nacional de Infectologia Evandro Chagas. Laboratório de Pesquisa Clínica em Neuroinfecções. Rio de Janeiro, RJ, Brasil / Universidade Federal do Rio de Janeiro. Rio de Janeiro, RJ, Brasil

Post-parotidectomy facial nerve function: comparison between original and modified Sunnybrook Facial Grading Systems

Background: Facial nerve dysfunction is the principal postoperative complication related to parotidectomy

Clinical and Immunological Insights on Severe, Adverse Neurotropic and Viscerotropic Disease following 17D Yellow Fever Vaccination▿

Yellow fever (YF) vaccines (17D-204 and 17DD) are well tolerated and cause very low rates of severe adverse events (YEL-SAE), such as serious allergic reactions, neurotropic adverse diseases (YEL-AND), and viscerotropic diseases (YEL-AVD). Viral and host factors have been postulated to explain the basis of YEL-SAE. However, the mechanisms underlying the occurrence of YEL-SAE remain unknown. The present report provides a detailed immunological analysis of a 23-year-old female patient. The patient developed a suspected case of severe YEL-AVD with encephalitis, as well as with pancreatitis and myositis, following receipt of a 17D-204 YF vaccination. The patient exhibited a decreased level of expression of Fc-γR in monocytes (CD16, CD32, and CD64), along with increased levels of NK T cells (an increased CD3+ CD16+/− CD56+/−/CD3+ ratio), activated T cells (CD4+ and CD8+ cells), and B lymphocytes. Enhanced levels of plasmatic cytokines (interleukin-6 [IL-6], IL-17, IL-4, IL-5, and IL-10) as well as an exacerbated ex vivo intracytoplasmic cytokine pattern, mainly observed within NK cells (gamma interferon positive [IFN-γ+], tumor necrosis factor alpha positive [TNF-α+], and IL-4 positive [IL-4+]), CD8+ T cells (IL-4+ and IL-5+), and B lymphocytes (TNF-α+, IL-4+, and IL-10+). The analysis of CD4+ T cells revealed a complex profile that consisted of an increased frequency of IL-12+ and IFN-γ+ cells and a decreased percentage of TNF-α+, IL-4+, and IL-5+ cells. Depressed cytokine synthesis was observed in monocytes (TNF-α+) following the provision of antigenic stimuli in vitro. These results support the hypothesis that a strong adaptive response and abnormalities in the innate immune system may be involved in the establishment of YEL-AND and YEL-AVD