Spatial autocorrelation of <i>T</i>. <i>trichiura</i> infection between and within households in Kyela.

<p>The red line shows Moran’s I of spatial autocorrelation for the raw data. The blue and green lines show the autocorrelation of deviance residuals for the models M1 and M2, respectively. The horizontal axis shows the distance bands between households.</p

Location of the EMINI study area.

<p>The study area consists of nine distinct study sites. Participating household are shown as red circles.</p

<i>T</i>. <i>trichiura</i> prevalence by age.

<p>The red line shows LOWESS-smoothed <i>T</i>. <i>trichiura</i> infection prevalence, grey bars indicate the number of participants in each age stratum.</p

Univariable association of different factors with <i>T</i>. <i>trichiura</i> infection in Kyela.

<p>Results of univariable Poisson regression models adjusted for household clustering using robust variance estimates (N = 912).</p

Multivariable association of different factors with <i>T</i>. <i>trichiura</i> infection in Kyela.

<p>Results of multivariable Poisson regression models adjusted for household clustering using robust variance estimates (N = 912). Multivariable results are only shown for those variables that were included into the respective model.</p

Characteristics of the study population and environmental conditions at their place of residence.

<p>Characteristics of the study population and environmental conditions at their place of residence.</p

Prevalence of <i>T</i>. <i>trichiura</i> infection in the nine EMINI study sites in Mbeya region, Tanzania (A) and details for Kyela site (B).

<p>Households with at least one infected person are represented by red Voronoi polygons, households without are shown in green. Subsite A and B in this text refer to the western and eastern part of Kyela, respectively.</p

CD27 and Mip-1β expression of CMV specific IFNγ producing CD8 T cell responses are not significantly influenced by HIV as well as tuberculosis co-infection in adults.

<p>Scatter-plots illustrating (A) the CD27 MFI fold-change to all CD8⁺ T cells and B) the proportion of cells producing Mip-1β of CMV specific IFNγ⁺-CD8⁺ T cell in patients under 10 or over 18 years old and according to HIV and TB infection status.</p

Frequencies and absolute counts of IFNγ⁺-CMVpp65–specific CD4⁺ or CD8⁺ T cell in children below 10 years of age and adults.

<p>Scatter-plots depicting A) the frequencies or absolute counts of CMV specific IFNγ⁺-CD4⁺ T cells s and C) frequencies of CMV specific IFNγ⁺-CD8⁺ T cells in the two-age groups and according to HIV status.</p

CD27 and Mip-1β expression of CMV specific IFNγ producing CD4 T cell responses are influenced by HIV as well as tuberculosis co-infection in adults.

<p>Scatter-plots illustrating (A) the CD27 MFI fold-change to all CD4⁺ T cells and B) the proportion of cells producing Mip-1β of CMV specific IFNγ⁺-CD4⁺ T cell in patients under 10 or over 18 years old and according to HIV and TB infection status.</p