Human Papilloma Virus-Type 16 (HPV-16) & Human Herpes Virus-Type 8 (HHV-8) Infections Were Found to be Co-Existing Major Cancer-Contributing Factors. Individualized, Safe, Effective Treatment of Hopelessly Advanced Cancer Patients with Metastasis by Combining 4 Methods of Effective Treatment: 1) Optimal Dose of Vitamin D3 Using its 10 Unique Beneficial Effects, 2) Selective Drug Uptake Enhancement Method, 3) Stimulation of Newly Discovered Thymus Gland Representation Areas on the Back of Each Hand, & 4) Identification & Removal of Co-Existing Cancer-Contributing Factors

The article presents a study that described how human papillomavirus type 16 (HPV-16) and human herpes virus-type 8 infections are non-invasively detected and how to reduce the infections by the use of optimal dose of vitamin D3 to safe range. A method of determining individualized optimal dose of vitamin D3 is presented. The significance of co-existing the infections with single-cell parasite toxoplasma gondii is discussed. Example of the eggs with HPV-16 infection is shown

Murine leukemia virus envelope protein in transgenic-mouse serum blocks infection in vitro.

Transgenic mice bearing a murine retroviral envelope transgene (Fv4) have Fv4 gp70env (SU) in their serum in amounts sufficient to block infection by ecotropic virus in vitro. Fv4 Env in serum is derived largely but not exclusively from hematopoietic cells. Tail cells from Fv4 mice and cell lines transduced with the Fv4 env transgene synthesize both components of the envelope protein (gp70 SU and p15E TM) but secrete the gp70 moiety, in the absence of retroviral particles. Blocking of the ecotropic viral receptor by secreted gp70 SU may contribute to resistance to retroviral infection in these mice