85 research outputs found

    Two-Dimensional Bosonization from Variable Shifts in the Path Integral

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    A method to perform bosonization of a fermionic theory in (1+1) dimensions in a path integral framework is developed. The method relies exclusively on the path integral property of allowing variable shifts, and does not depend on the explicit form of Greens functions. Two examples, the Schwinger model and the massless Thirring model, are worked out.Comment: 4 page

    Fermion Condensates of massless QED2QED_2 at Finite Density in non-trivial Topological Sectors

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    Vacuum expectation values of products of local bilinears ψˉψ\bar\psi\psi are computed in massless QED2QED_2 at finite density. It is shown that chiral condensates exhibit an oscillatory inhomogeneous behaviour depending on the chemical potential. The use of a path-integral approach clarifies the connection of this phenomenon with the topological structure of the theory.Comment: 16 pages, no figures, To be published in Phys.Rev.

    Smooth Bosonization II: The Massive Case

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    The (1+1)-dimensional bosonization relations for fermionic mass terms are derived by choosing a specific gauge in an enlarged gauge-invariant theory containing both fermionic and bosonic fields. The fermionic part of the generating functional subject to the gauge constraint can be cast into the form of a strongly coupled Schwinger model, which can be solved exactly. The resulting bosonic theory coupled to the scalar sources then exhibits directly the bosonic counterparts of the fermionic scalar and pseudoscalar mass densities.Comment: 8 pages, Latex, CERN-TH-6563/9

    Numerical study of the critical behavior of the Ashkin-Teller model at a line defect

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    We consider the Ashkin-Teller model on the square lattice, which is represented by two Ising models (σ\sigma and τ\tau) having a four-spin coupling of strength, Ï”\epsilon, between them. We introduce an asymmetric defect line in the system along which the couplings in the σ\sigma Ising model are modified. In the Hamiltonian version of the model we study the scaling behavior of the critical magnetization at the defect, both for σ\sigma and for τ\tau spins by density matrix renormalization. For Ï”>0\epsilon>0 we observe identical scaling for σ\sigma and τ\tau spins, whereas for Ï”<0\epsilon<0 one model becomes locally ordered and the other locally disordered. This is different of the critical behavior of the uncoupled model (Ï”=0\epsilon=0) and is in contradiction with the results of recent field-theoretical calculations.Comment: 6 pages, 4 figure

    A gauge invariant and string independent fermion correlator in the Schwinger model

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    We introduce a gauge invariant and string independent two-point fermion correlator which is analyzed in the context of the Schwinger model (QED_2). We also derive an effective infrared worldline action for this correlator, thus enabling the computation of its infrared behavior. Finally, we briefly discuss possible perspectives for the string independent correlator in the QED_3 effective models for the normal state of HTc superconductors.Comment: 14 pages, LaTe

    Multiflavor Correlation Functions in non-Abelian Gauge Theories at Finite Density in two dimensions

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    We compute vacuum expectation values of products of fermion bilinears for two-dimensional Quantum Chromodynamics at finite flavored fermion densities. We introduce the chemical potential as an external charge distribution within the path-integral approach and carefully analyse the contribution of different topological sectors to fermion correlators. We show the existence of chiral condensates exhibiting an oscillatory inhomogeneous behavior as a function of a chemical potential matrix. This result is exact and goes in the same direction as the behavior found in QCD_4 within the large N approximation.Comment: 28 pages Latex (3 pages added and other minor changes) to appear in Phys.Rev.

    Role of mitochondrial raft-like microdomains in the regulation of cell apoptosis

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    Lipid rafts are envisaged as lateral assemblies of specific lipids and proteins that dissociate and associate rapidly and form functional clusters in cell membranes. These structural platforms are not confined to the plasma membrane; indeed lipid microdomains are similarly formed at subcellular organelles, which include endoplasmic reticulum, Golgi and mitochondria, named raft-like microdomains. In addition, some components of raft-like microdomains are present within ER-mitochondria associated membranes. This review is focused on the role of mitochondrial raft-like microdomains in the regulation of cell apoptosis, since these microdomains may represent preferential sites where key reactions take place, regulating mitochondria hyperpolarization, fission-associated changes, megapore formation and release of apoptogenic factors. These structural platforms appear to modulate cytoplasmic pathways switching cell fate towards cell survival or death. Main insights on this issue derive from some pathological conditions in which alterations of microdomains structure or function can lead to severe alterations of cell activity and life span. In the light of the role played by raft-like microdomains to integrate apoptotic signals and in regulating mitochondrial dynamics, it is conceivable that these membrane structures may play a role in the mitochondrial alterations observed in some of the most common human neurodegenerative diseases, such as Amyotrophic lateral sclerosis, Huntington's chorea and prion-related diseases. These findings introduce an additional task for identifying new molecular target(s) of pharmacological agents in these pathologies

    Production of D∗+(2010)D^{*+}(2010) mesons by high energy neutrinos from the Tevatron

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    Charged vector D∗+(2010)D^{*+}(2010) meson production is studied in a high energy neutrino bubble chamber experiment with mean neutrino energy of 141 GeV. The D∗+D^{*+} are produced in (5.6±1.8)%(5.6 \pm 1.8)\% of the neutrino charged current interactions, indicating a steep increase of cross section with energy. The mean fractional hadronic energy of the D∗+D^{*+} meson is 0.55±0.060.55 \pm 0.06

    A membrane-inserted structural model of the yeast mitofusin Fzo1

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    Mitofusins are large transmembrane GTPases of the dynamin-related protein family, and are required for the tethering and fusion of mitochondrial outer membranes. Their full-length structures remain unknown, which is a limiting factor in the study of outer membrane fusion. We investigated the structure and dynamics of the yeast mitofusin Fzo1 through a hybrid computational and experimental approach, combining molecular modelling and all-atom molecular dynamics simulations in a lipid bilayer with site-directed mutagenesis and in vivo functional assays. The predicted architecture of Fzo1 improves upon the current domain annotation, with a precise description of the helical spans linked by flexible hinges, which are likely of functional significance. In vivo site-directed mutagenesis validates salient aspects of this model, notably, the long-distance contacts and residues participating in hinges. GDP is predicted to interact with Fzo1 through the G1 and G4 motifs of the GTPase domain. The model reveals structural determinants critical for protein function, including regions that may be involved in GTPase domain-dependent rearrangements
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