Ritual, Myth and Tragedy: Origins of Theatre in Dionysian Rites

In the deep, dark forests and in the lush green valleys, worshippers of Dionysus celebrated the eternal cycles of death and rebirth, symbolized in the sacred mask of the wild god. Drunk and intoxicated, wearing the mask of Dionysus, the actor is at once the shaman and the priest. Channeling the presence of the fearsome divinity, he drinks the sacred wine and eats the raw flesh of his prey. In this eternal moment, he becomes one with the god and the beast residing inside of him. Within Ancient Greek culture, the sacred rites of Dionysus have been appropriated and transformed to theatre performances. The shaman became the actor, the participants became the audience, the sacred altar became the stage. From myth as a ritual performance emerged the theatre of tragedy, in which the undying spirit of Dionysus, majestic and terrifying, speaks to us even today.

Ritual, Myth and Tragedy: Origins of Theatre in Dionysian Rites

In the deep, dark forests and in the lush green valleys, worshippers of Dionysus celebrated the eternal cycles of death and rebirth, symbolized in the sacred mask of the wild god. Drunk and intoxicated, wearing the mask of Dionysus, the actor is at once the shaman and the priest. Channeling the presence of the fearsome divinity, he drinks the sacred wine and eats the raw flesh of his prey. In this eternal moment, he becomes one with the god and the beast residing inside of him. Within Ancient Greek culture, the sacred rites of Dionysus have been appropriated and transformed to theatre performances. The shaman became the actor, the participants became the audience, the sacred altar became the stage. From myth as a ritual performance emerged the theatre of tragedy, in which the undying spirit of Dionysus, majestic and terrifying, speaks to us even today

An investigation of the relationship between free-viewing perceptual asymmetries for vertical and horizontal stimuli

Two experiments examine the relationship between free-viewing vertical and horizontal perceptual biases. In Experiment 1, normal participants (n=24) made forced-choice luminance judgments on two mirror-reversed luminance gradients (the 'grayscales' task). The stimuli were presented in vertical, horizontal and oblique ( +/- 45degrees) orientations. Leftward and upward biases were observed in the horizontal and vertical conditions, respectively. In the oblique conditions, leftward and upward biases combined to produce a strong shift of attention away from the lower/right space toward the upper/left. Regression analyses revealed that the oblique biases were the combined product of the vertical and horizontal biases. A lack of correlation between the vertical and horizontal biases, however, suggests they reflect the operation of independent cognitive/neural mechanisms. In Experiment 2, the same stimuli were given to right-hemisphere-lesioned patients with spatial neglect (n = 4). Rightward and upward biases were observed for horizontal and vertical stimuli, respectively. The biases combined to produce a strong shift of attention away from the lower/left space toward the upper/right. While our research demonstrates that vertical and horizontal attentional biases are additive, it also appears that they reflect the operation of independent cognitive/neural mechanisms. Potential applications of these findings to the remediation of spatial neglect are discussed. (C) 2004 Elsevier B.V. All rights reserved

Prism adaptation and spatial attention: A study of visual search in normals and patients with unilateral neglect

Visuomotor adaptation to a prism-induced lateral displacement of the visual field induces mild perceptual biases in healthy individuals and improves symptoms of unilateral neglect. The present study employed a speeded visual search task to test the hypothesis that prism adaptation induces an adaptive redistribution of selective spatial attention. In Experiment 1, 32 neurologically healthy, right-handed participants were adapted to a 15degrees prism-induced lateral (left or right) displacement of the visual field. Spatial attention was measured by search time and error-rate in unique-feature ("preattentive") and feature-absent ("serial") visual search tasks, before and after prism adaptation. The single target appeared at different locations within arrays of 12, 24 or 48 items. Contrary to the attentional hypothesis, the pattern of search performance across the display remained unchanged following prism adaptation. In Experiment 2, we tested four patients with unilateral right hemisphere damage on the visual search tasks, before and after adaptation to 15degrees rightward-displacing prisms. All four patients showed a pathological gradient of spatial attention toward the ipsilesional side prior to adaptation. Consistent with the results from Experiment 1, the gradient in search performance shown by the patients did not change following prism adaptation. Taken together, these findings suggest that the perceptual aftereffects-in normals and amelioration of unilateral neglect following prism adaptation are not mediated by an adaptive redistribution of spatial attention

Abnormal fMRI adaptation to unfamiliar faces in a case of developmental prosopamnesia

In rare cases, damage to the temporal lobe causes a selective impairment in the ability to learn new faces, a condition known as prosopamnesia [1]. Here we present the case of an individual with prosopamnesia in the absence of any acquired structural lesion. "C" shows intact processing of simple and complex non-face objects, but her ability to learn new faces is severely impaired. We used a neural marker of perceptual learning known as repetition suppression to examine functioning within C's fusiform face area (FFA), a region of cortex involved in face perception [2]. For comparison, we examined repetition suppression in the scene-selective parahippocampal place area (PPA) [3]. As expected, normal controls showed significant region-specific attenuation of neural activity across repetitions of each stimulus class. C also showed normal attenuation within the PPA to familiar and unfamiliar scenes, and within the FFA to familiar faces. Critically, however, she failed to show any adaptive change within the FFA for repeated unfamiliar faces, despite a face-specific blood-oxygen-dependent response (BOLD) response in her FFA during viewing of face stimuli. Our findings suggest that in developmental prosopamnesia, the FFA cannot maintain stable representations of new faces for subsequent recall or recognition

Impaired working memory for location but not for colour or shape in visual neglect: A comparison of parietal and non-parietal lesions

Patients with spatial neglect due to right hemisphere pathology may show 'revisiting' behaviour during visual search and cancellation tasks, such that previously encountered targets are treated as if they are new discoveries. Revisiting behaviour is particularly evident when no visible trace is left to inform patients that a particular target has already been detected (Husain et al., 2001; Wojciulik et al., 2001), implying that spatial working memory may be impaired in neglect. To test whether working memory for location is selectively impaired relative to memory for colour and shape, we compared performances of right hemisphere neglect patients with parietal (n = 4) and non-parietal (n = 4) lesions on a change detection task. Patients were presented with a matrix containing four objects in different positions, and required to detect a change in the location, colour or shape of one of the objects following presentation of a brief visual mask. Parietal patients were selectively impaired in detecting location changes, regardless of the horizontal position of the object in the matrix, relative to colour and shape changes. This deficit of spatial working memory was not apparent for neglect patients with lesions that spared the parietal cortex. We conclude that the human parietal cortex is crucially involved in the updating and maintenance of spatial representations across saccades, and that neglect arising from parietal damage causes impairment in these re-mapping mechanisms