The Arabic version of the Stroop Test and its equivalency to the English version

The Stroop test is one of the most popular tests frequently used to assess the function of the frontal lobe in neurological and psychiatric patient populations. Performance on the Stroop test is very sensitive to lesions of the frontal lobes and is commonly used in clinical settings. In 1999, we decided to find out if the Stroop test will be as reliable after translation to the Arabic language as the original English version. We completed the work in Riyadh Military Hospital in 2000. A sample of 10 Saudi adult healthy individuals participated in this study. Their mean age was 31.9 and their mean years of education were 17.3. All subjects performed the Arabic and the English versions of Stroop test. Performance showed no differences between the English and the Arabic versions of the Stroop test. Equivalency data between the two versions of Stroop test are provided. The availability of this data will help us to provide a normative data for the Saudi Committee of Health Education, and in carrying out research on frontal lobe function

Saudi normative data for the Wisconsin Card Sorting Test, Stroop Test, Test of Non-verbal Intelligence-3, Picture Completion and Vocabulary (subtest of the Wechsler Adult Intelligence Scale-Revised).

There are 2 aims for this study: first, to collect normative data for the Wisconsin Card Sorting Test (WCST), Stroop test, Test of Non-verbal Intelligence (TONI-3), Picture Completion (PC) and Vocabulary (VOC) sub-test of the Wechsler Adult Intelligence Scale-Revised for use in a Saudi Arabian culture, and second, to use the normative data provided to generate the regression equations. To collect the normative data and generate the regression equations, 198 healthy individuals were selected to provide a representative distribution for age, gender, years of education, and socioeconomic class. The WCST, Stroop test, TONI-3, PC, and VOC were administrated to the healthy individuals. This study was carried out at the Department of Clinical Neurosciences, Riyadh Military Hospital, Riyadh, Kingdom of Saudi Arabia from January 2000 to July 2002. Normative data were obtained for all tests, and tables were constructed to interpret scores for different age groups. Regression equations to predict performance on the 3 tests of frontal function from scores on tests of fluid (TONI-3) and premorbid intelligence were generated from the data from the healthy individuals. The data collected in this study provide normative tables for 3 tests of frontal lobe function and for tests of general intellectual ability for use in Saudi Arabia. The data also provide a method to estimate preinjury ability without the use of verbally based tests

A review of the scorpion fauna of Saudi Arabia

The scorpions of Saudi Arabia were surveyed in the major regions of Jazan, Al-Medina, Al-Baha, Hail, and Riyadh, in addition to nine provinces surveyed more superficially. Jazan (1,440 specimens) had 10 buthids and two scorpionid species and subspecies; Al-Medina (867) had seven buthid and two scorpionid species and subspecies, one of which, the scorpionid Scorpio maurus (palmatus?), needs further confirmation of identity. The Al-Baha region (2421 specimens) contained five buthids and two scorpionid species and subspecies; Hail (1,921) had eight buthid and two scorpionid species and subspecies - the most common subspecies here was Scorpio maurus kruglovi. Androctonus crassicauda and Leiurus quinquestriatus were only found in Hail and Al-Baha; Androctonus bicolor was newly recorded in Hail and Riyadh. Riyadh (4,164 specimens) had nine buthid, one scorpionid and at least two hemiscorpiid species and subspecies. The Saudi fauna was found to comprise at least 28 species and subspecies of the families Buthidae, Scorpionidae and Hemiscorpiidae.Keywords: Buthidae, Scorpionidae, Diplocentrida

Normative data for the two equivalent forms of the Arabic verbal fluency test

Performance on verbal fluency (VF) test is sensitive to lesions on the frontal lobes. We earlier developed two equivalent forms of formal VF in the Arabic language for use with healthy Saudi individuals

Establishment of the military neurosurgeons committee within the world federation of neurosurgical societies

In 2009, during the World Congress of Neurological Surgery in Boston, Massachusetts, the World Federation of Neurosurgical Societies (WFNS) Executive Committee decided to establish a Military Neurosurgeons Committee. A separate scientific session on military neurosurgery was held at the next WFNS Interim Meeting in September 2011 in Brazil. A further separate session on military neurosurgery will take place at the next WFNS Meeting in Seoul, South Korea. © 2013 Elsevier Inc. All rights reserved.SCOPUS: re.jinfo:eu-repo/semantics/publishe

Protection by 2-Deoxy-D-glucose against b,b8-Iminodipropionitrile-Induced Neurobehavioral Toxicity in Mice

This investigation was undertaken to study the effect of 2-deoxy-D-glucose (2-DG) on b,b8-iminodipropionitrile (IDPN)-induced neurobehavioral toxicity in mice. Animals were divided into five groups of nine animals each. One of the groups served as control and received vehicle only, whereas the remaining four groups were treated with IDPN (250 mg/kg, ip) daily for 11 days. 2-DG was injected intraperitoneally in the doses of 0 (vehicle only), 100, 300, and 600 mg/kg daily 30 min before IDPN administration. The animals were observed for dyskinetic behavior including vertical (retrocollis) and horizontal (laterocollis) head movements and circling. Twenty-four hours after the last dose of IDPN, the animals were sacrificed by decapitation and striata were isolated from the brain for the analysis of serotonin (5-HT). Our results showed that 2-DG significantly and dose dependently attenuated the incidence and severity of IDPN-induced neurobehavioral toxicity. Administration of 2-DG also protected mice against IDPN-induced increase in striatal 5-HT levels. Further studies are warranted to investigate the neuroprotective mechanism of 2-DG against IDPN-induced neurotoxicityAcademic Pres

Effect of acrylamide on neurological recovery following spinal cord injury in rats

Acrylamide (ACR) is a cumulative neurotoxin which causes axonal degeneration in animals and man. Industrial workers exposed to ACR have been reported to suffer from a variety of central and peripheral neuropathological symptoms including numbness of hands and feet, skin peeling and muscular weakness of legs. These reports suggest that the body burden of ACR may be a risk factor in recovery patterns following neurotrauma. The present study was designed to assess the effect of ACR on neurological recovery following spinal cord injury (SCI) in rats. Male Sprague-Dawley rats weighing 200-230 g were anaesthetised with chloral hydrate and laminectomy was performed at T 7-8 level leaving the dura intact. A compression plate (2.2 x 5.0 mm) loaded with a weight of 35 g was placed on the exposed cord for 5 minutes. Animals were divided into seven groups of eight rats each. The animals in Group 1 served as control whereas rats in Group 2 underwent laminectomy alone (sham). The rats in Group 3 to 6 were subjected to SCI as mentioned above. Animals in Groups 4, 5 and 6 also received ACR in the doses of 10 mg/kg, 20 mg/kg and 40 mg/kg, i.p., respectively in addition to SCI, whereas the rats in Group 7 received ACR alone at a dose of 40 mg/kg body weight. The first dose of ACR was given 30 minutes before SCI, followed by daily administration of drug for 7 days. Post traumatic neurological recovery was recorded daily for 10 days using a modified Tarlov score, inclined plane test and sensory and vocal score. Electrophysiological changes were assessed using somatosensory and corticomotor evoked potentials. The animals were sacrificed at different time intervals and the injured site of the spinal cord was analysed for lipid hydroperoxides (LPH), conjugated dienes (CD) and glutathione (GSH). Neuropathological changes in the spinal cord were assessed using light microscopy. The rats exposed to compression injury alone showed a maximum neurological deficit at 24 hr and then a gradual recovery was observed over a period of 10 days. The rats treated with ACR along with SCI showed poor or no recovery over a period of 10 days. Our electrophysiological and histopathological studies also confirmed that concomitant exposure to ACR produces a significant deleterious effect on the recovery from SCI. SCI induced increase in oxidative stress (increase in LPH and CD and decrease in GSH) is also exacerbated by ACR suggesting a role of free radicals. The results of this study suggest that increased body burden of ACR may retard the recovery from neurotrauma or even lead to permanent disability