A dsorption studies of malachite green dye on mesoporous silica synthesized in 1-octyl-3-methylimidazolium chloride ionic liquid

In this work, one long-chain ionic liquid (IL), 1-octyl-3-methylimidazolium chloride, was used as a template for the preparation of mesoporous silica via a modified sol-gel route. The morphology of the obtained material was characterized by scanning electron microscopy (SEM). The various vibrational modes of different functional groups in the mesoporous silica materials were revealed by Fourier transform infrared (FTIR) spectroscopy. N2 adsorption-desorption isotherms measurement was used to characterize the pore diameter and BET surface area. The adsorption capacity of malachite green dye from aqueous solution has been performed using the mesoporous silica synthesized in ionic liquid. The adsorption equilibrium of dye on mesoporous silica was found to be at 150 min. The maximum malachite green dye adsorption was found to be 97 %

Pvr expression regulators in equilibrium signal control and maintenance of Drosophila blood progenitors.

Blood progenitors within the lymph gland, a larval organ that supports hematopoiesis in Drosophila melanogaster, are maintained by integrating signals emanating from niche-like cells and those from differentiating blood cells. We term the signal from differentiating cells the 'equilibrium signal' in order to distinguish it from the 'niche signal'. Earlier we showed that equilibrium signaling utilizes Pvr (the Drosophila PDGF/VEGF receptor), STAT92E, and adenosine deaminase-related growth factor A (ADGF-A) (Mondal et al., 2011). Little is known about how this signal initiates during hematopoietic development. To identify new genes involved in lymph gland blood progenitor maintenance, particularly those involved in equilibrium signaling, we performed a genetic screen that identified bip1 (bric à brac interacting protein 1) and Nucleoporin 98 (Nup98) as additional regulators of the equilibrium signal. We show that the products of these genes along with the Bip1-interacting protein RpS8 (Ribosomal protein S8) are required for the proper expression of Pvr