The impact of husbands' involvement in goal‐setting training on women's empowerment: First evidence from an intervention among female microfinance borrowers in Sri Lanka

Offering women access to microcredit and business training is a prominent approach to stimulate women's empowerment. Whereas men seem to profit from business training, women do not. We adjusted a goal‐setting training session on the basis of women's needs in collaboration with a women organization in Sri Lanka. We invited female microfinance borrowers and their husbands to the training as both parties should be involved to change existing gender roles with respect to their income‐generating activity. We investigated the impact of the training on goal‐setting skills, self‐esteem, and the couples' interaction in a subsequent task. In two field experiments, female borrowers and their husbands (nstudy1 = 68; nstudy2 = 76) were randomly assigned to one of three conditions: (a) goal‐setting training and setting goals as couple, (b) goal‐setting training and setting goals individually, or (c) no training (control condition). Participation in the training increased women's SMART (specific, measurable, achievable, realistic, time bound) goal‐setting skills. We coded couples' interactions in a subsequent decision‐making task to assess signs of women's empowerment. Descriptively, we found some initial evidence of increased women's empowerment in the interaction (Study 2). We critically discuss results and how gendered power imbalances may need to be addressed to stimulate social change towards gender equity

RNA-Binding Protein Khd1 and Ccr4 Deadenylase Play Overlapping Roles in the Cell Wall Integrity Pathway in Saccharomyces cerevisiae▿

The Saccharomyces cerevisiae RNA-binding protein Khd1/Hek2 associates with hundreds of potential mRNA targets preferentially, including the mRNAs encoding proteins localized to the cell wall and plasma membrane. We have previously revealed that Khd1 positively regulates expression of MTL1 mRNA encoding a membrane sensor in the cell wall integrity (CWI) pathway. However, a khd1Δ mutation has no detectable phenotype on cell wall synthesis. Here we show that the khd1Δ mutation causes a severe cell lysis when combined with the deletion of the CCR4 gene encoding a cytoplasmic deadenylase. We identified the ROM2 mRNA, encoding a guanine nucleotide exchange factor (GEF) for Rho1, as a target for Khd1 and Ccr4. The ROM2 mRNA level was decreased in the khd1Δ ccr4Δ mutant, and ROM2 overexpression suppressed the cell lysis of the khd1Δ ccr4Δ mutant. We also found that Ccr4 negatively regulates expression of the LRG1 mRNA encoding a GTPase-activating protein (GAP) for Rho1. The LRG1 mRNA level was increased in the ccr4Δ and khd1Δ ccr4Δ mutants, and deletion of LRG1 suppressed the cell lysis of the khd1Δ ccr4Δ mutant. Our results presented here suggest that Khd1 and Ccr4 modulate a signal from Rho1 in the CWI pathway by regulating the expression of RhoGEF and RhoGAP