Generalized Chiral Kinetic Equations

We derive the generalized chiral kinetic equations which are applicable to the fermions with arbitrary mass. We show how the dynamical magnetic-moment distribution function could lead to spin polarization and electric charge separation. We also show how the electric/magnetic moment distribution and pseudoscalar distribution could be induced by vorticity and acceleration in global equilibrium.Comment: 6 pages, no figure

Study on turbidity current head going through the changing width section

AbstractBased on former research on the turbidity current, and learning lessons from the study on turbidity current,11 flume experiments has been operated with combined factors on different sediment concentration and different width. From the surveyed data turbidity current head going through the changing width section have been analyzed. Taken use of mathematical statistics method, local resistance coefficient of turbidity current head has been acquired on the changing width section

catena-Poly[silver(I)-μ-acridine-9-carboxyl­ato-κ3 N:O,O′]

In the title coordination polymer, [Ag(C14H8NO2)]n, the AgI cation is coordinated by two O atoms and one N atom from two symmetry-related acridine-9-carboxyl­ate ligands in a distorted trigonal-planar geometry. The metal atoms are connected by the ligands to form chains running parallel to the b axis. π–π stacking inter­actions [centroid-to-centroid distances 3.757 (2)–3.820 (2) Å] and weak Ag⋯O inter­actions further link the chains to form a layer network parallel to the ab plane. The AgI cation is disordered over two positions, with refined site-occupancy factors of 0.73 (3):0.27 (3)

Manipulation of magnetic nanoparticle retention and hemodynamic consequences in microcirculation: assessment by laser speckle imaging

Magnetic nanoparticles (MNPs) have been proposed for targeted or embolization therapeutics. How MNP retention occurs in circulation may critically determine local hemodynamics, tissue distribution of MNPs, and the therapeutic effects. We attempted to establish a microcirculation model to study the magnetic capture of MNPs in small vessels and to determine the factors affecting MNP retention. Two-dimensional hemodynamic changes in response to magnet-induced MNP retention in the microvessels of the cremaster muscle in vivo were observed in a real-time manner using a laser speckle imaging technique. Changes in tissue perfusion of the cremaster muscle appeared to be closely correlated with the location of the magnet placement underneath the muscle in response to intra-arterial administration of dextran-coated MNPs. Magnet-related retention was observed along the edge of the magnet, as corroborated by the results of histology analysis and microcomputed tomography. In these preparations, tissue iron content almost doubled, as revealed by inductively coupled plasma optical emission spectroscopy. In addition, MNP retention was associated with reduced downstream flow in a dose-dependent manner. Dissipation of MNPs (5 mg/kg) occurred shortly after removal of the magnet, which was associated with significant recovery of tissue flow. However, MNP dissipation did not easily occur after administration of a higher MNP dose (10 mg/kg) or prolonged exposure to the magnetic field. An ultrasound after removal of the magnet may induce the partial dispersion of MNPs and thus partially improve hemodynamics. In conclusion, our results revealed the important correlation of local MNP retention and hemodynamic changes in microcirculation, which can be crucial in the application of MNPs for effective targeted therapeutics

The G Protein Coupled Receptor 3 Is Involved in cAMP and cGMP Signaling and Maintenance of Meiotic Arrest in Porcine Oocytes

The arrest of meiotic prophase in mammalian oocytes within fully grown follicles is dependent on cyclic adenosine monophosphate (cAMP) regulation. A large part of cAMP is produced by the Gs-linked G-protein-coupled receptor (GPR) pathway. In the present study, we examined whether GPR3 is involved in the maintenance of meiotic arrest in porcine oocytes. Expression and distribution of GPR3 were examined by western blot and immunofluorescence microscopy, respectively. The results showed that GPR3 was expressed at various stages during porcine oocyte maturation. At the germinal vesicle (GV) stage, GPR3 displayed a maximal expression level, and its expression remained stable from pro-metaphase I (MI) to metaphase II (MII). Immunofluorescence staining showed that GPR3 was mainly distributed at the nuclear envelope during the GV stage and localized to the plasma membrane at pro-MI, MI and MII stages. RNA interference (RNAi) was used to knock down the GPR3 expression within oocytes. Injection of small interfering double-stranded RNA (siRNA) targeting GPR3 stimulated meiotic resumption of oocytes. On the other hand, overexpression of GPR3 inhibited meiotic maturation of porcine oocytes, which was caused by increase of cGMP and cAMP levels and inhibition of cyclin B accumulation. Furthermore, incubation of porcine oocytes with the GPR3 ligand sphingosylphosphorylcholine (SPC) inhibited oocyte maturation. We propose that GPR3 is required for maintenance of meiotic arrest in porcine oocytes through pathways involved in the regulation of cAMP and cGMP

Methoxo[N′-(3-meth­oxy-2-oxidobenzyl­idene)benzohydrazidato]oxidovanadium(V)

In the title complex, [V(C15H12N2O4)(CH3O)O], the VV ion exhibits a distorted square-pyramidal coordination geometry; three donor atoms from a hydrazone ligand and one O atom of the deprotonated methanol define the coordination basal plane. The VV ion is displaced by 0.464 (1) Å from the basal plane towards the axial oxide O atom. Intra­molecular O—H⋯N hydrogen bonding occurs. Inter­molecular C—H⋯O hydrogen bonding is also observed in the crystal structure