Deformation mechanism and process optimization of mechanical clinching joining technology used for steel and aluminum alloy sheets

Abstract. In this paper we construct several tools for manipulating pools of biases in the analysis of RC4. Then, we show that optimized strategies can break WEP based on 4000 packets by assuming that the first bytes of plaintext are known for each packet. We describe similar attacks for WPA. Firstly, we describe a distinguisher for WPA of complexity 2 43 and advantage 0.5 which uses 2 40 packets. Then, based on several partial temporary key recovery attacks, we recover the full 128-bit temporary key by using 2 38 packets. It works within a complexity of 2 96. So far, this is the best attack against WPA. We believe that our analysis brings further insights on the security of RC4.

Discovery and Exploitation of New Biases in RC4

Abstract. In this paper, we present several weaknesses in the stream cipher RC4. First, we present a technique to automatically reveal linear correlations in the PRGA of RC4. With this method, 48 new exploitable correlations have been discovered. Then we bind these new biases in the PRGA with known KSA weaknesses to provide practical key recovery attacks. Henceforth, we apply a similar technique on RC4 as a black box, i.e. the secret key words as input and the keystream words as output. Our objective is to exhaustively find linear correlations between these elements. Thanks to this technique, 9 new exploitable correlations have been revealed. Finally, we exploit these weaknesses on RC4 to some practical examples, such as the WEP protocol. We show that these correlations lead to a key recovery attack on WEP with only 9800 encrypted packets (less than 20 seconds), instead of 24200 for the best previous attack.

Alterações morfológicas de tecido laminar do casco e parâmetros clínicos e laboratoriais de equinos com síndrome cólica letal Morphologic alterations of the hoof lamellar tissue, and clinic and laboratorial analyses of horses with lethal colic syndrome

As afecções gastrintestinais dos cavalos são agravadas por complicações como a laminite, cuja etiopatogenia está relacionada à degradação da membrana basal do tecido laminar por metaloproteinases (MMPs). A ativação das MMPs pode ocorrer devido à liberação local de citocinas inflamatórias ou enzimas provenientes de leucócitos infiltrados no tecido laminar. O objetivo deste trabalho foi avaliar as alterações morfológicas do tecido laminar de equinos com síndrome cólica letal e sua provável associação com parâmetros clínicos e laboratoriais. Observou-se intensa destruição da arquitetura laminar, principalmente nos animais com alterações físicas e laboratoriais mais acentuadas, como tempo de preenchimento capilar prolongado (TPC), membranas mucosas congestas, taquicardia, hemoconcentração e redução nas contagens de plaquetas e leucócitos. Os resultados sinalizam o provável momento do desenvolvimento de lesões do tecido laminar em equinos com síndrome cólica, no qual é possível adotar medidas preventivas contra a laminite.<br>The gastrointestinal diseases of horses are aggravated by complications such as laminitis. The laminitis etiopathogeny are connected with lamellar basement membrane degradation by matrix metalloproteinases (MMPs). Inflammatory cytokines and leukocytes enzymes can active MMPs. The object of this study was to evaluate morphological changes on lamellar tissue of horses with colic syndrome and its association with clinical and laboratorial parameters. It was observed intensive destruction of lamellar architecture, mainly on animals with severe physical and laboratorial alterations, such as delayed capillary refill time, congested mucous membrane, tachycardia, hemoconcentration and low count of platelet and leukocytes. The results sign to the most likely moment of development of lamellar tissue injuries in horses with colic syndrome, which can be adopted preventive measures against laminitis