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Remote transfer of Gaussian quantum discord
Quantum discord quantifies quantum correlation between quantum systems, which
has potential application in quantum information processing. In this paper, we
propose a scheme realizing the remote transfer of Gaussian quantum discord, in
which another quantum discordant state or an Einstein-Podolsky-Rosen entangled
state serves as ancillary state. The calculation shows that two independent
optical modes that without direct interaction become quantum correlated after
the transfer. The output Gaussian quantum discord can be higher than the
initial Gaussian quantum discord when optimal gain of the classical channel and
the ancillary state are chosen. The physical reason for this result comes from
the fact that the quantum discord of an asymmetric Gaussian quantum discordant
state can be higher than that of a symmetric one. The presented scheme has
potential application in quantum information network
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The roles of endoglin gene in cerebrovascular diseases.
Endoglin (ENG, also known as CD105) is a transforming growth factor β (TGFβ) associated receptor and is required for both vasculogenesis and angiogenesis. Angiogenesis is important in the development of cerebral vasculature and in the pathogenesis of cerebral vascular diseases. ENG is an essential component of the endothelial nitric oxide synthase activation complex. Animal studies showed that ENG deficiency impairs stroke recovery. ENG deficiency also impairs the regulation of vascular tone, which contributes to the pathogenesis of brain arteriovenous malformation (bAVM) and vasospasm. In human, functional haploinsufficiency of ENG gene causes type I hereditary hemorrhagic telangiectasia (HHT1), an autosomal dominant disorder. Compared to normal population, HHT1 patients have a higher prevalence of AVM in multiple organs including the brain. Vessels in bAVM are fragile and tend to rupture, causing hemorrhagic stroke. High prevalence of pulmonary AVM in HHT1 patients are associated with a higher incidence of paradoxical embolism in the cerebral circulation causing ischemic brain injury. Therefore, HHT1 patients are at risk for both hemorrhagic and ischemic stroke. This review summarizes the possible mechanism of ENG in the pathogenesis of cerebrovascular diseases in experimental animal models and in patients
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