Adapting Visual Question Answering Models for Enhancing Multimodal Community Q&A Platforms

Question categorization and expert retrieval methods have been crucial for information organization and accessibility in community question & answering (CQA) platforms. Research in this area, however, has dealt with only the text modality. With the increasing multimodal nature of web content, we focus on extending these methods for CQA questions accompanied by images. Specifically, we leverage the success of representation learning for text and images in the visual question answering (VQA) domain, and adapt the underlying concept and architecture for automated category classification and expert retrieval on image-based questions posted on Yahoo! Chiebukuro, the Japanese counterpart of Yahoo! Answers. To the best of our knowledge, this is the first work to tackle the multimodality challenge in CQA, and to adapt VQA models for tasks on a more ecologically valid source of visual questions. Our analysis of the differences between visual QA and community QA data drives our proposal of novel augmentations of an attention method tailored for CQA, and use of auxiliary tasks for learning better grounding features. Our final model markedly outperforms the text-only and VQA model baselines for both tasks of classification and expert retrieval on real-world multimodal CQA data.Comment: Submitted for review at CIKM 201

Degeneracy Relations in QCD and the Equivalence of Two Systematic All-Orders Methods for Setting the Renormalization Scale

The Principle of Maximum Conformality (PMC) eliminates QCD renormalization scale-setting uncertainties using fundamental renormalization group methods. The resulting scale-fixed pQCD predictions are independent of the choice of renormalization scheme and show rapid convergence. The coefficients of the scale-fixed couplings are identical to the corresponding conformal series with zero $\beta$-function. Two all-orders methods for systematically implementing the PMC-scale setting procedure for existing high order calculations are discussed in this article. One implementation is based on the PMC-BLM correspondence \mbox{(PMC-I)}; the other, more recent, method \mbox{(PMC-II)} uses the ${\cal R}_\delta$-scheme, a systematic generalization of the minimal subtraction renormalization scheme. Both approaches satisfy all of the principles of the renormalization group and lead to scale-fixed and scheme-independent predictions at each finite order. In this work, we show that PMC-I and PMC-II scale-setting methods are in practice equivalent to each other. We illustrate this equivalence for the four-loop calculations of the annihilation ratio $R_{e^+ e^-}$ and the Higgs partial width $\Gamma(H\to b\bar{b})$. Both methods lead to the same resummed (`conformal') series up to all orders. The small scale differences between the two approaches are reduced as additional renormalization group $\{\beta_i\}$-terms in the pQCD expansion are taken into account. We also show that {\it special degeneracy relations}, which underly the equivalence of the two PMC approaches and the resulting conformal features of the pQCD series, are in fact general properties of non-Abelian gauge theory.Comment: 7 pages, 1 figur

The Quark/Antiquark Asymmetry of the Nucleon Sea

Although the distributions of sea quarks and antiquarks generated by leading-twist QCD evolution through gluon splitting $g \rightarrow \bar q q$ are necessarily CP symmetric, the distributions of nonvalence quarks and antiquarks which are intrinsic to the nucleon's bound state wavefunction need not be identical. In this paper we investigate the sea quark/antiquark asymmetries in the nucleon wavefunction which are generated by a light-cone model of energetically-favored meson-baryon fluctuations. The model predicts striking quark/antiquark asymmetries in the momentum and helicity distributions for the down and strange contributions to the proton structure function: the intrinsic $d$ and $s$ quarks in the proton sea are predicted to be negatively polarized, whereas the intrinsic $\bar d$ and $\bar s$ antiquarks give zero contributions to the proton spin. Such a picture is supported by experimental phenomena related to the proton spin problem and the violation of the Ellis-Jaffe sum rule. The light-cone meson-baryon fluctuation model also suggests a structured momentum distribution asymmetry for strange quarks and antiquarks which could be relevant to an outstanding conflict between two different determinations of the strange quark sea in the nucleon. The model predicts an excess of intrinsic $d \bar d$ pairs over $u \bar u$ pairs, as supported by the Gottfried sum rule violation. We also predict that the intrinsic charm and anticharm helicity and momentum distributions are not identical.Comment: LaTex 18 pages, 4 figures. To obtain a copy, send e-mail to [email protected]

Dynamic critical phenomena in the AdS/CFT duality

In critical phenomena, singular behaviors arise not only for thermodynamic quantities but also for transport coefficients. We study this dynamic critical phenomenon in the AdS/CFT duality. We consider black holes with a single R-charge in various dimensions and compute the R-charge diffusion in the linear perturbations. In this case, the black holes belong to model B according to the classification of Hohenberg and Halperin.Comment: 17 pages, ReVTeX4; v2: added references and discussio

Hypothalamic actions of neuromedin U.

The central nervous system and gut peptide neuromedin U (NMU) inhibits feeding after intracerebroventricular injection. This study explored the hypothalamic actions of NMU on feeding and the hypothalamo-pituitary-adrenal axis. Intraparaventricular nucleus (intra-PVN) NMU dose-dependently inhibited food intake, with a minimum effective dose of 0.1 nmol and a robust effect at 0.3 nmol. Feeding inhibition was mapped by NMU injection into eight hypothalamic areas. NMU (0.3 nmol) inhibited food intake in the PVN (0-1 h, 59 ± 6.9% of the control value; P < 0.001) and arcuate nucleus (0-1 h, 76 ± 10.4% of the control value; P < 0.05). Intra-PVN NMU markedly increased grooming and locomotor behavior and dose-dependently increased plasma ACTH (0.3 nmol NMU, 24.8 ± 1.9 pg/ml; saline, 11.4 ± 1.0; P < 0.001) and corticosterone (0.3 nmol NMU, 275.4 ± 40.5 ng/ml; saline, 129.4 ± 25.0; P < 0.01). Using hypothalamic explants in vitro, NMU stimulated CRH (100 nM NMU, 5.9 ± 0.95 pmol/explant; basal, 3.8 ± 0.39; P < 0.01) and arginine vasopressin release (100 nM NMU, 124.5 ± 21.8 fmol/explant; basal, 74.5 ± 7.6; P < 0.01). Leptin stimulated NMU release (141.9 ± 20.4 fmol/explant; basal, 92.9 ± 9.4; P < 0.01). Thus, we describe a novel role for NMU in the PVN to stimulate the hypothalamo-pituitary-adrenal axis and locomotor and grooming behavior and to inhibit feeding

Light-Cone Representation of the Spin and Orbital Angular Momentum of Relativistic Composite Systems

The matrix elements of local operators such as the electromagnetic current, the energy momentum tensor, angular momentum, and the moments of structure functions have exact representations in terms of light-cone Fock state wavefunctions of bound states such as hadrons. We illustrate all of these properties by giving explicit light-cone wavefunctions for the two-particle Fock state of the electron in QED, thus connecting the Schwinger anomalous magnetic moment to the spin and orbital momentum carried by its Fock state constituents. We also compute the QED one-loop radiative corrections for the form factors for the graviton coupling to the electron and photon. Although the underlying model is derived from elementary QED perturbative couplings, it in fact can be used to simulate much more general bound state systems by applying spectral integration over the constituent masses while preserving all of the Lorentz properties, giving explicit realization of the spin sum rules and other local matrix elements. The role of orbital angular momentum in understanding the "spin crisis" problem for relativistic systems is clarified. We also prove that the anomalous gravitomagnetic moment B(0) vanishes for any composite system. This property is shown to follow directly from the Lorentz boost properties of the light-cone Fock representation and holds separately for each Fock state component. We show how the QED perturbative structure can be used to model bound state systems while preserving all Lorentz properties. We thus obtain a theoretical laboratory to test the consistency of formulae which have been proposed to probe the spin structure of hadrons.Comment: Version to be published in Nuclear Physics B. Includes illustrations of graviton-lepton form factors at one loop in QE

Wavefunction-Independent Relations between the Nucleon Axial-Coupling g_A and the Nucleon Magnetic Moments

We calculate the proton's magnetic moment $\mu_p$ and its axial-vector coupling $g_A$ as a function of its Dirac radius $R_1$ using a relativisitic three-quark model formulated on the light-cone. The relationship between $\mu_p$ and $g_A$ is found to be independent of the assumed form of the light-cone wavefunction. At the physical radius $R_1=0.76$ fm, one obtains the experimental values for both $\mu_p$ and $g_A$, and the helicity carried by the valence $u$ and $d$ quarks are each reduced by a factor $\simeq 0.75$ relative to their non-relativistic values. At large proton radius, $\mu_p$ and $g_A$ are given by the usual non-relativistic formulae. At small radius, $\mu_p$ becomes equal to the Dirac moment, as demanded by the Drell-Hearn-Gerasimov sum rule. In addition, as $R_1 \to 0,$ the constituent quark helicities become completely disoriented and $g_A \to 0$.Comment: 17 pages, RevTeX, 4 uuencoded figures, SLAC-PUB-643

trans-Complementation of an NS2 Defect in a Late Step in Hepatitis C Virus (HCV) Particle Assembly and Maturation

Recent studies using cell culture infection systems that recapitulate the entire life cycle of hepatitis C virus (HCV) indicate that several nonstructural viral proteins, including NS2, NS3, and NS5A, are involved in the process of viral assembly and release. Other recent work suggests that Ser-168 of NS2 is a target of CK2 kinase–mediated phosphorylation, and that this controls the stability of the genotype 1a NS2 protein. Here, we show that Ser-168 is a critical determinant in the production of infectious virus particles. Substitution of Ser-168 with Ala (or Gly) ablated production of infectious virus by cells transfected with a chimeric viral RNA (HJ3-5) containing core-NS2 sequences from the genotype 1a H77 virus within the background of genotype 2a JFH1 virus. An S168A substitution also impaired production of virus by cells transfected with JFH1 RNA. This mutation did not alter polyprotein processing or genome replication. This defect in virus production could be rescued by expression of wt NS2 in trans from an alphavirus replicon. The trans-complementing activities of NS2 from genotypes 1a and 2a demonstrated strong preferences for rescue of the homologous genotype. Importantly, the S168A mutation did not alter the association of core or NS5A proteins with host cell lipid droplets, nor prevent the assembly of core into particles with sedimentation and buoyant density properties similar to infectious virus, indicating that NS2 acts subsequent to the involvement of core, NS5A, and NS3 in particle assembly. Second-site mutations in NS2 as well as in NS5A can rescue the defect in virus production imposed by the S168G mutation. In aggregate, these results indicate that NS2 functions in trans, in a late-post assembly maturation step, perhaps in concert with NS5A, to confer infectivity to the HCV particle