O(αs) O(\alpha_s) corrections to polarized top decay into a charged Higgs t(↑)→H++b t(\uparrow) \to H^+ + b

We calculate the $O(\alpha_s)$ radiative corrections to polarized top quark decay into a charged Higgs boson and a massive bottom quark in two variants of the two-Higgs-doublet model. The radiative corrections to the polarization asymmetry of the decay may become as large as $25$. We provide analytical formulae for the unpolarized and polarized rates for $m_b \neq 0$ and for $m_b = 0$. For $m_b = 0$ our closed-form expressions for the unpolarized and polarized rates become rather compact.Comment: 24 pages, 5 figures in the text, content modified, comments added, appendices added, references updated, replaced with published versio

Polarized top decay into polarized W: t(up)->W(up) + b at O(alpha_s)

We consider the decay of a polarized top quark into a polarized W-boson plus a bottom quark, followed by the decay of the W-boson into a pair of leptons or quarks. The polar angle distribution of the top spin relative to the W-momentum and the polar angle distribution of the lepton (or quark) in the W-rest frame is governed by three polarized and three unpolarized rate functions which are related to the double density matrix elements of the decay $t \to W^+ + b$. We obtain analytical expressions for the $O(\alpha_s)$ radiative corrections to the three polarized and three unpolarized rate functions. We also provide a comprehensive discussion of the dependence of the longitudinal, transverse and normal polarization of the top quarks produced at $e^+e^-$-colliders on beam polarization parameters.Comment: 13 pages, 3 postscript figures added, file standard.sty included, material on normal polarization added, some misprints correcte

Eosinophils: Biological properties and role in health and disease

Eosinophils are pleiotropic multifunctional leukocytes involved in initiation and propagation of diverse inflammatory responses, as well as modulators of innate and adaptive immunity. In this review, the biology of eosinophils is summarized, focusing on transcriptional regulation of eosinophil differentiation, characterization of the growing properties of eosinophil granule proteins, surface proteins and pleiotropic mediators, and molecular mechanisms of eosinophil degranulation. New views on the role of eosinophils in homeostatic function are examined, including developmental biology and innate and adaptive immunity (as well as their interaction with mast cells and T cells) and their proposed role in disease processes including infections, asthma, and gastrointestinal disorders. Finally, strategies for targeted therapeutic intervention in eosinophil-mediated mucosal diseases are conceptualized